T. Cocks

发表

Release and properties of endothelium‐derived relaxing factor (EDRF) from endothelial cells in culture

Julie H. Campbell, J. Campbell, J. Angus, 1985, Journal of cellular physiology.

Enzymatic activation of endothelial protease‐activated receptors is dependent on artery diameter in human and porcine isolated coronary arteries

T. Cocks, J. Hamilton, J. Tatoulis, 2002, British journal of pharmacology.

Protease‐Activated Receptor (PAR) 1 but Not PAR2 or PAR4 Mediates Endothelium‐Dependent Relaxation to Thrombin and Trypsin in Human Pulmonary Arteries

T. Cocks, J. Hamilton, A. Frauman, 2001, Journal of cardiovascular pharmacology.

Nitrergic relaxation of the mouse gastric fundus is mediated by cyclic GMP‐dependent and ryanodine‐sensitive mechanisms

T. Cocks, S. Selemidis, 2000, British journal of pharmacology.

Endothelium‐dependent and ‐independent responses to protease‐activated receptor‐2 (PAR‐2) activation in mouse isolated renal arteries

T. Cocks, J. Moffatt, T. Cocks, 1998, British journal of pharmacology.

5‐Hydroxytryptamine (5‐HT) mediates potent relaxation in the sheep isolated pulmonary vein via activation of 5‐HT4 receptors

T. Cocks, T. Cocks, P. Arnold, 1992, British journal of pharmacology.

Vascular Actions of Serotonin in Large and Small Arteries are Amplified by Loss of Endothelium, Atheroma and Hypertension

J. Angus, C. Wright, T. Cocks, 1989 .

Lipopolysaccharide Induces Epithelium- and Prostaglandin E2-Dependent Relaxation of Mouse Isolated Trachea through Activation of Cyclooxygenase (COX)-1 and COX-2

T. Cocks, Rowan W Balzary, 2006, Journal of Pharmacology and Experimental Therapeutics.

Vasodilatation by acetylcholine is endothelium‐dependent: a study by sonomicrometry in canine femoral artery in vivo.

J. Angus, G. Campbell, T. Cocks, 1983, The Journal of physiology.

Endothelium-dependent hyperpolarization: a role in the control of vascular tone.

C. Garland, T. Cocks, F. Plane, 1995, Trends in pharmacological sciences.

Increased Expression of Protease-Activated Receptor-2 (PAR2) and PAR4 in Human Coronary Artery by Inflammatory Stimuli Unveils Endothelium-Dependent Relaxations to PAR2 and PAR4 Agonists

T. Cocks, J. Hamilton, A. Frauman, 2001, Circulation research.

Pharmacologically Distinct Intracellular Calcium Pools Regulate Tonic and Oscillatory Responses in Porcine Thoracic Duct

T. Cocks, J. Moffatt, 2004, Journal of cardiovascular pharmacology.

Endothelium-dependent hyperpolarization as a remote anti-atherogenic mechanism.

T. Cocks, S. Selemidis, 2002, Trends in pharmacological sciences.

Balloon Catheter Injury to Rabbit Carotid Artery: II. Selective Increase in Reactivity to Some Vasoconstrictor Drugs

G. Campbell, T. Cocks, John A. Manderson, 1989, Arteriosclerosis.

Apamin‐ and nitric oxide‐sensitive biphasic non‐adrenergic non‐cholinergic inhibitory junction potentials in the rat anococcygeus muscle

T. Cocks, J. Ziogas, S. Selemidis, 1998, The Journal of physiology.

Endothelium-dependent relaxation of coronary arteries by noradrenaline and serotonin

J. Angus, T. Cocks, 1983, Nature.

Reactivity of endothelin-1 on human and canine large veins compared with large arteries in vitro.

J. Angus, T. Cocks, K. Sudhir, 1989, European journal of pharmacology.

Effects of neuronal polypeptides on intestinal smooth muscle; a comparison with non-adrenergic, non-cholinergic nerve stimulation and ATP.

G. Burnstock, T. Cocks, 1979, European journal of pharmacology.

Endothelium-dependent relaxation to the B1 kinin receptor agonist des-Arg9-bradykinin in human coronary arteries.

T. Cocks, G. Drummond, 1995, British journal of pharmacology.

SPECIAL REPORT Endothelium‐dependent relaxation to the B1 kinin receptor agonist des‐Arg ‐bradykinin in human coronary arteries

T. Cocks, G. Drummond, 1995 .

Apamin‐sensitive, non‐nitric oxide (NO) endothelium‐dependent relaxations to bradykinin in the bovine isolated coronary artery: no role for cytochrome P450 and K+

T. Cocks, G. Drummond, S. Selemidis, 2000, British journal of pharmacology.

Evidence for differential roles of nitric oxide (NO) and hyperpolarization in endothelium‐dependent relaxation of pig isolated coronary artery

T. Cocks, E.V. Kilpatrick, 1994, British journal of pharmacology.

Non-adrenergic, non-cholinergic (purinergic?) inhibitory innervation of the rabbit rectococcygeus muscle.

G. Burnstock, T. Cocks, R. Crowe, 1979, European journal of pharmacology.

EVIDENCE FOR PURINERGIC INNERVATION OF THE ANOCOCCYGEUS MUSCLE

G. Burnstock, T. Cocks, R. Crowe, 1978, British journal of pharmacology.

Evidence that contractions of isolated arteries by L-NMMA and NOLA are not due to inhibition of basal EDRF release

J. Angus, T. Cocks, 1991 .

The α adrenoceptors on endothelial cells

J. Angus, T. Cocks, K. Satoh, 1986 .

Alpha 2-adrenoceptors and endothelium-dependent relaxation in canine large arteries.

J. Angus, T. Cocks, K. Satoh, 1986, British journal of pharmacology.

The alpha adrenoceptors on endothelial cells.

J. Angus, T. Cocks, K. Satoh, 1986, Federation proceedings.

Protease‐activated receptor‐2 turnover stimulated independently of receptor activation in porcine coronary endothelial cells

T. Cocks, J. Hamilton, J. Chow, 1999, British journal of pharmacology.

Evidence for mediation by endothelium‐derived hyperpolarizing factor of relaxation to bradykinin in the bovine isolated coronary artery independently of voltage‐operated Ca2+ channels

T. Cocks, G. Drummond, T. Cocks, 1996, British journal of pharmacology.

Changes in Vascular Reactivity following Endothelial Denudation

G. Campbell, T. Cocks, J. Manderson, 1990 .

ENDOTHELIN IS BLOOD VESSEL SELECTIVE: STUDIES ON A VARIETY OF HUMAN AND DOG VESSELS IN VITRO AND ON REGIONAL BLOOD FLOW IN THE CONSCIOUS RABBIT

J. Angus, T. Cocks, M. Dib, 1989, Clinical and experimental pharmacology & physiology.

Endothelial Cells in Culture and Production of Endothelium-Derived Relaxing Factor

Julie H. Campbell, J. Angus, G. Campbell, 1988 .

The half‐life of endothelium‐derived relaxing factor released from bovine aortic endothelial cells in culture.

J. Angus, T. Cocks, T. Cocks, 1987, The Journal of physiology.

Vasodilatation and the discovery of endothelium‐derived relaxing factor

J. Angus, T. Cocks, 1987, The Medical journal of Australia.

Protease-activated receptor-2 activating peptide SLIGRL inhibits bacterial lipopolysaccharide-induced recruitment of polymorphonuclear leukocytes into the airways of mice.

K. Jeffrey, T. Cocks, J. Moffatt, 2002, American journal of respiratory cell and molecular biology.

Smooth muscle mediates circumferential conduction of hyperpolarization and relaxation to focal endothelial cell activation in large coronary arteries

T. Cocks, S. Selemidis, 2007, Naunyn-Schmiedeberg's Archives of Pharmacology.

Pharmacological Analysis of 5-HT Receptors in Human Small Coronary Arteries

J. Angus, T. Cocks, M. Ross-Smith, 1993 .

THE ACETYLCHOLINE PARADOX: A CONSTRICTOR OF HUMAN SMALL CORONARY ARTERIES EVEN IN THE PRESENCE OF ENDOTHELIUM

J. Angus, T. Cocks, G. Mcpherson, 1991, Clinical and experimental pharmacology & physiology.

Glibenclamide is a competitive antagonist of the thromboxane A2 receptor in dog coronary artery in vitro

J. Angus, T. Cocks, S. King, 1990, British journal of pharmacology.

Protease-activated receptor-2 regulates trypsin expression in the brain and protects against seizures and epileptogenesis

T. O'Brien, T. Cocks, R. Lohman, 2008, Neurobiology of Disease.

Initial support for the hypothesis that PAR2 is involved in the immune response to Nippostrongylus brasiliensis in mice

R. Gasser, T. Cocks, M. Devlin, 2007, Parasitology Research.

Amiloride analogues cause endothelium‐dependent relaxation in the canine coronary artery in vitro: possible role of Na+/Ca2+ exchange

P. Little, J. Angus, T. Cocks, 1988, British journal of pharmacology.

Role of the epithelium and acetylcholine in mediating the contraction to 5‐hydroxytryptamine in the mouse isolated trachea

C. Page, T. Cocks, J. Moffatt, 2004, British journal of pharmacology.

Comparison of relaxation responses of vascular and non-vascular smooth muscle to endothelium-derived relaxing factor (EDRF), acidified sodium nitrite (NO) and sodium nitroprusside

J. Angus, T. Cocks, 1990, Naunyn-Schmiedeberg's Archives of Pharmacology.

Oxyhaemoglobin increases the production of endothelin-1 by endothelial cells in culture.

J. Angus, T. Cocks, R. L. Woods, 1991, European journal of pharmacology.

ENDOTHELIUM‐DEPENDENT RELAXATION IS UNALTERED BY HYPERTENSION, CHOLESTEROL OR INTIMAL THICKENING

J. Angus, G. Campbell, C. Wright, 1986, Clinical and experimental pharmacology & physiology.

Effect of protease‐activated receptor (PAR)‐1, −2 and −4‐activating peptides, thrombin and trypsin in rat isolated airways

T. Cocks, J. Moffatt, J. Chow, 2000, British journal of pharmacology.

Evidence that prostaglandin is responsible for the 'rebound contraction' following stimulation of non-adrenergic, non-cholinergic ('purinergic') inhibitory nerves.

G. Burnstock, T. Cocks, B. Paddle, 1975, European journal of pharmacology.

Hepta and octapeptide agonists of protease‐activated receptor 2

D. Fairlie, B. Pfeiffer, B. Flanagan, 2007, Journal of peptide science : an official publication of the European Peptide Society.

Interactions between receptors that increase cytosolic calcium and cyclic AMP in guinea‐pig liver cells

D. Jenkinson, T. Cocks, K. Koller, 1984, British journal of pharmacology.

Coronary circulation and 5-hydroxytryptamine

J. Angus, C. Wright, T. Cocks, 1990 .

Direct evidence for ATP release from non-adrenergic, non-cholinergic ("purinergic") nerves in the guinea-pig taenia coli and bladder.

G. Burnstock, T. Cocks, L. Kasakov, 1978, European journal of pharmacology.

PURINERGIC INNERVATION OF THE GUINEA‐PIG URINARY BLADDER

G. Burnstock, T. Cocks, L. Kasakov, 1978, British journal of pharmacology.

EFFECTS OF ADENOSINE 5′‐TRIPHOSPHATE (ATP) AND β‐γ‐METHYLENE ATP ON THE RAT URINARY BLADDER

G. Burnstock, T. Cocks, C. Brown, 1979 .

A comparison between ATP and bradykinin as possible mediators of the responses of smooth muscle to non-adrenergic non-cholinergic nerves.

G. Burnstock, T. Cocks, J. Ferrero, 1980, European journal of pharmacology.

Apamin blocks certain neurotransmitter-induced increases in potassium permeability

D. Jenkinson, T. Cocks, G. Burgess, 1979, Nature.

B2 kinin receptor activation is the predominant mechanism by which trypsin mediates endothelium-dependent relaxation in bovine coronary arteries

T. Cocks, G. Drummond, S. Selemidis, 2008, Naunyn-Schmiedeberg's Archives of Pharmacology.

A regulatory role for protease-activated receptor-2 in motivational learning in rats

N. Jones, T. Cocks, R. Lohman, 2009, Neurobiology of Learning and Memory.

α2‐Adrenoceptors and endothelium‐dependent relaxation in canine large arteries

J. Angus, T. Cocks, K. Satoh, 1986, British journal of pharmacology.

Comparison of contractile responses to 5‐hydroxytryptamine and sumatriptan in human isolated coronary artery: synergy with the thromboxane A2‐receptor agonist, U46619

J. Angus, T. Cocks, D. Pruneau, 1993, British journal of pharmacology.

Protease-Activated Receptor-2 Peptides Activate Neurokinin-1 Receptors in the Mouse Isolated Trachea

D. Fairlie, T. Cocks, J. Moffatt, 2006, Journal of Pharmacology and Experimental Therapeutics.

Endothelium-derived relaxing factor.

J. Angus, T. Cocks, 1989, Pharmacology & therapeutics.

Role of endothelium in vascular responses to norepinephrine, serotonin and acetylcholine.

J. Angus, T. Cocks, 1984, Bibliotheca cardiologica.

The mechanism of the blockade by trifluoperazine of some actions of phenylephrine on liver and smooth muscle.

D. Jenkinson, P. Dilger, T. Cocks, 1981, Biochemical pharmacology.

Endothelium-dependent vasodilator mechanisms

T. Cocks, 1996 .

Evidence that both nitric oxide (NO) and a non‐NO hyperpolarizing factor elicit NANC nerve‐mediated relaxation in the rat isolated anococcygeus

T. Cocks, S. Selemidis, 1997, British journal of pharmacology.

Endothelium-Dependent Responses in Large Arteries and in the Microcirculation

J. Angus, G. Campbell, C. Wright, 1988 .

Nature and role of endothelium derived relaxing factor (EDRF)

Julie H. Campbell, J. Angus, G. Campbell, 1986 .

Thimerosal blocks stimulated but not basal release of endothelium‐derived relaxing factor (EDRF) in dog isolated coronary artery

P. Crack, T. Cocks, 1992, British journal of pharmacology.

Heterogeneous mechanisms of endothelium‐dependent relaxation for thrombin and peptide activators of protease‐activated receptor‐1 in porcine isolated coronary artery

T. Cocks, J. Hamilton, T. Cocks, 2000, British journal of pharmacology.

Pharmacological reactivity of human epicardial coronary arteries: phasic and tonic responses to vasoconstrictor agents differentiated by nifedipine

T. Cocks, A. Stork, Thomas M. Cocks, 1994, British journal of pharmacology.

Development of a large fibromuscular intimal thickening does not impair endothelium-dependent relaxation in the rabbit carotid artery.

J. Angus, P. Mosse, G. Campbell, 1987, Blood vessels.

Pharmacological reactivity of human epicardial coronary arteries: characterization of relaxation responses to endothelium‐derived relaxing factor

T. Cocks, A. Stork, Thomas M. Cocks, 1994, British journal of pharmacology.

A protective role for protease-activated receptors in the airways

M. Carr, G. Anderson, T. Cocks, 1999, Nature.

Activation of protease-activated receptor-2 (PAR-2) elicits nitric oxide-dependent dilatation of the basilar artery in vivo.

C. Sobey, T. Cocks, T. Cocks, 1998, Stroke.

Increased Expression of Protease-Activated Receptor-2 (PAR2) and PAR4 in Human Coronary Artery by Inflammatory Stimuli Unveils Endothelium-Dependent Relaxations to PAR2 and PAR4 Agonists

T. Cocks, J. Hamilton, A. Frauman, 2001, Circulation research.

Protease-activated receptors mediate apamin-sensitive relaxation of mouse and guinea pig gastrointestinal smooth muscle.

T. Cocks, S. Selemidis, V. Sozzi, 1999, Gastroenterology.