S. Breit

发表

Impact of pre‐analytical handling on bone marrow mRNA gene expression

Matthias Nees, Christian Hagemeier, A. Kulozik, 2004, British journal of haematology.

Cartilage-like gene expression in differentiated human stem cell spheroids: a comparison of bone marrow-derived and adipose tissue-derived stromal cells.

Hans Hauner, Anja Winter, Karin Benz, 2003, Arthritis and rheumatism.

Molecular analysis of expansion, differentiation, and growth factor treatment of human chondrocytes identifies differentiation markers and growth-related genes.

W. Richter, M. Lukoschek, S. Breit, 2002, Biochemical and biophysical research communications.

A strict requirement of interleukin‐4 for interleukin‐4 induction in antigen‐stimulated human memory T cells

A. Levine, W. Sebald, C. Heusser, 1996, European journal of immunology.

Exon-junction complex components specify distinct routes of nonsense-mediated mRNA decay with differential cofactor requirements.

M. Hentze, N. Gehring, G. Neu-Yilik, 2005, Molecular cell.

Profiling and functional annotation of mRNA gene expression in pediatric rhabdomyosarcoma and Ewing's sarcoma

Yvonne Braun, D. Wai, Y. Braun, 2004, International journal of cancer.

Enhanced expression of the human chitinase 3-like 2 gene (YKL-39) but not chitinase 3-like 1 gene (YKL-40) in osteoarthritic cartilage.

W. Richter, S. Breusch, S. Breit, 2002, Biochemical and biophysical research communications.

Downregulation of Notch signaling by γ-secretase inhibition can abrogate chemotherapy-induced apoptosis in T-ALL cell lines

M. Muckenthaler, A. Kulozik, S. Breit, 2009, Annals of Hematology.

Activating NOTCH1 mutations predict favorable early treatment response and long-term outcome in childhood precursor T-cell lymphoblastic leukemia.

M. Muckenthaler, M. Schrappe, W. Ludwig, 2006, Blood.

Activating NOTCH1 Mutations Predict Favorable Early Treatment Response and Long Term Outcome in Child- hood Precursor T-Cell Lymphoblastic Leukemia Short Title: NOTCH1mutations predict outcome in pediatric T-ALL

M. Muckenthaler, M. Schrappe, W. Ludwig, 2006 .

High-resolution genomic profiling of childhood T-ALL reveals frequent copy-number alterations affecting the TGF-beta and PI3K-AKT pathways and deletions at 6q15-16.1 as a genomic marker for unfavorable early treatment response.

M. Muckenthaler, P. Lichter, S. Pfister, 2009, Blood.

The favorable effect of activating NOTCH1 receptor mutations on long-term outcome in T-ALL patients treated on the ALL–BFM 2000 protocol can be separated from FBXW7 loss of function

M. Muckenthaler, M. Schrappe, W. Ludwig, 2010, Leukemia.

The abundance of RNPS1, a protein component of the exon junction complex, can determine the variability in efficiency of the Nonsense Mediated Decay pathway

M. Hentze, A. Kulozik, S. Breit, 2007, Nucleic acids research.

The uORF-containing thrombopoietin mRNA escapes nonsense-mediated decay (NMD)

M. Hentze, G. Neu-Yilik, A. Kulozik, 2006, Nucleic acids research.

Changing serine-485 to alanine in the opossum parathyroid hormone (PTH)/PTH-related peptide receptor enhances PTH stimulation of phospholipase C in a stably transfected human kidney cell line: a useful model for PTH-analog screening?

R. Ziegler, S. Breit, H. Wickert, 2001, Bone.

The Favorable Effect of Activating NOTCH1 Receptor Mutations On Long Term Outcome in T-ALL Is Treatment Related and Can Be Separated From NOTCH Pathway Activation by FBXW7 Loss of Function.

M. Muckenthaler, M. Schrappe, W. Ludwig, 2009 .

Suppression of MYC by high expression of NMYC in human neuroblastoma cells

M. Schwab, S. Breit, S. Breit, 1989, Journal of neuroscience research.

Non-syntenic amplification of MDM2 and MYCN in human neuroblastoma.

M. Schwab, L. Amler, M. Oren, 1995, Oncogene.