C Napolitano

Evidence for a Cardiac Ion Channel Mutation Underlying Drug‐Induced QT Prolongation and Life‐Threatening Arrhythmias

S. Priori, L. Bianchi, P. Schwartz, 2000, Journal of cardiovascular electrophysiology.

Gene-specific differences in the circadian variation of ventricular repolarization in the long QT syndrome: a key to sudden death during sleep?

W Haverkamp, S. Priori, P. Schwartz, 2000, Italian heart journal : official journal of the Italian Federation of Cardiology.

CaMKII inhibition rectifies arrhythmic phenotype in a patient-specific model of catecholaminergic polymorphic ventricular tachycardia

M Miragoli, S. Priori, G. Condorelli, 2013, Cell Death and Disease.

Long QT syndrome patients with mutations of the SCN5A and HERG genes have differential responses to Na+ channel blockade and to increases in heart rate. Implications for gene-specific therapy.

S. Priori, P. Schwartz, T. Colatsky, 1995, Circulation.

Torsade de pointes. Mechanisms and management.

P. Schwartz, C. Napolitano, C Napolitano, 1994, Drugs.

Mechanisms of I(Ks) suppression in LQT1 mutants.

S. Priori, L. Bianchi, P. Schwartz, 2000, American journal of physiology. Heart and circulatory physiology.

Inherited Brugada and Long QT-3 Syndrome Mutations of a Single Residue of the Cardiac Sodium Channel Confer Distinct Channel and Clinical Phenotypes*

S. Priori, P. Vardas, M. Memmi, 2001, The Journal of Biological Chemistry.

Evaluation of the spatial aspects of T-wave complexity in the long-QT syndrome.

S. Priori, P. Schwartz, C. Napolitano, 1997, Circulation.

Gene-specific response of dynamic ventricular repolarization to sympathetic stimulation in LQT1, LQT2 and LQT3 forms of congenital long QT syndrome.

S. Priori, K. Sunagawa, J. Towbin, 2002, European heart journal.

Low penetrance in the long-QT syndrome: clinical impact.

S. Priori, P. Schwartz, C. Napolitano, 1999, Circulation.