Powel H Brown

发表

Inducible overexpression of c-Jun in MCF7 cells causes resistance to vinblastine via inhibition of drug-induced apoptosis and senescence at a step subsequent to mitotic arrest.

P. Brown, T. Chambers, Lingling Duan, 2007, Biochemical pharmacology.

Blockade of AP-1 Potentiates Endocrine Therapy and Overcomes Resistance

Mathieu Lupien, Rinath Jeselsohn, Susan G Hilsenbeck, 2016, Molecular Cancer Research.

AP-1 blockade in breast cancer cells causes cell cycle arrest by suppressing G1 cyclin expression and reducing cyclin-dependent kinase activity

Qiang Shen, P. Brown, Chunhua Lu, 2004, Oncogene.

Identification of conserved gene expression features between murine mammary carcinoma models and human breast tumors

Zhiyuan Hu, Charles M Perou, Jason I. Herschkowitz, 2007, Genome Biology.

cJun overexpression in MCF-7 breast cancer cells produces a tumorigenic, invasive and hormone resistant phenotype

P. Brown, A. Sabichi, M. Birrer, 1999, Oncogene.

Preventive therapy for breast cancer: a consensus statement.

Jack Cuzick, Anthony Howell, J. Cuzick, 2011, The Lancet. Oncology.

Comprehensive Genomic Analysis Identifies Novel Subtypes and Targets of Triple-Negative Breast Cancer

Gordon B Mills, Kyle R Covington, Susan G Hilsenbeck, 2014, Clinical Cancer Research.

Global gene expression analysis of estrogen receptor transcription factor cross talk in breast cancer: identification of estrogen-induced/activator protein-1-dependent genes.

S. Hilsenbeck, P. Brown, D. DeNardo, 2005, Molecular endocrinology.

Prevention and early detection of prostate cancer.

Timothy J Wilt, Dragan Ilic, Jack Cuzick, 2014, The Lancet. Oncology.

The AP-1 transcription factor regulates postnatal mammary gland development.

Qiang Shen, Susan G Hilsenbeck, S. Hilsenbeck, 2006, Developmental biology.