A. Uittenbogaard

发表

Caspase-3 Mediates the Pathogenic Effect of Yersinia pestis YopM in Liver of C57BL/6 Mice and Contributes to YopM's Function in Spleen

D. Cohen, Zhan Ye, Amanda A. Gorman, 2014, PloS one.

Toward a molecular pathogenic pathway for Yersinia pestis YopM

J. Ting, D. Cohen, Zhan Ye, 2012, Front. Cell. Inf. Microbio..

Temperature-dependence of yadBC phenotypes in Yersinia pestis.

B. J. Hinnebusch, Amanda A. Gorman, A. Uittenbogaard, 2014, Microbiology.

Distinct CCR2+ Gr1+ Cells Control Growth of the Yersinia pestis ΔyopM Mutant in Liver and Spleen during Systemic Plague

J. Ambati, D. Cohen, Zhan Ye, 2010, Infection and Immunity.

HDL-associated estradiol stimulates endothelial NO synthase and vasodilation in an SR-BI-dependent manner.

Xiang-An Li, T. Kelly, A. Uittenbogaard, 2003, The Journal of clinical investigation.

Caveolin-1 negatively regulates SR-BI mediated selective uptake of high-density lipoprotein-derived cholesteryl ester.

D. R. van der Westhuyzen, A. Uittenbogaard, S. Matveev, 2001, European journal of biochemistry.

Activated Cardiac Adenosine A1 Receptors Translocate Out of Caveolae*

P. Narayan, R. Lasley, A. Uittenbogaard, 2000, The Journal of Biological Chemistry.

High Density Lipoprotein Prevents Oxidized Low Density Lipoprotein-induced Inhibition of Endothelial Nitric-oxide Synthase Localization and Activation in Caveolae*

I. Yuhanna, P. Shaul, A. Uittenbogaard, 2000, The Journal of Biological Chemistry.

Characterization of a Cytosolic Heat-shock Protein-Caveolin Chaperone Complex

Y. Ying, A. Uittenbogaard, E. Smart, 1998, The Journal of Biological Chemistry.

Hypercholesterolemia Promotes a CD36-dependent and Endothelial Nitric-oxide Synthase-mediated Vascular Dysfunction*

M. Febbraio, A. Uittenbogaard, T. Guerin, 2002, The Journal of Biological Chemistry.

Targeting Pathogenic Lafo ra Bodies in Lafora Disease Using an Antibody-Enzyme Fusion Graphical

Matthew S. Gentry, A. Depaoli-Roach, P. Roach, 2019 .

High Density Lipoprotein Binding to Scavenger Receptor, Class B, Type I Activates Endothelial Nitric-oxide Synthase in a Ceramide-dependent Manner*

M. Nikolova-Karakashian, Xiang-An Li, A. Uittenbogaard, 2002, The Journal of Biological Chemistry.

Palmitoylation of Caveolin-1 Is Required for Cholesterol Binding, Chaperone Complex Formation, and Rapid Transport of Cholesterol to Caveolae*

A. Uittenbogaard, E. Smart, Annette M. Uittenbogaard, 2000, The Journal of Biological Chemistry.

Polyglucosan body structure in Lafora disease.

Matthew S. Gentry, J. Putaux, Mitchell A. Sullivan, 2020, Carbohydrate polymers.

Targeting pathogenic Lafora bodies in Lafora disease using an antibody-enzyme fusion

Matthew S. Gentry, A. Depaoli-Roach, P. Roach, 2019, bioRxiv.

Iron Toxicity in the Retina Requires Alu RNA and the NLRP3 Inflammasome.

Youn Seon Han, B. Gelfand, W. McDonald, 2015, Cell reports.

HIV protease inhibitors promote atherosclerotic lesion formation independent of dyslipidemia by increasing CD36-dependent cholesteryl ester accumulation in macrophages.

Xiang-An Li, A. Uittenbogaard, S. Matveev, 2003, The Journal of clinical investigation.

Cholesteryl Ester Is Transported from Caveolae to Internal Membranes as Part of a Caveolin-Annexin II Lipid-Protein Complex*

A. Uittenbogaard, S. Matveev, Annette Uittenbogaard, 2002, The Journal of Biological Chemistry.

Iron Toxicity in the Retina Requires Alu RNA and the NLRP 3 Inflammasome Graphical Abstract Highlights

Youn Seon Han, B. Gelfand, W. McDonald, 2018 .

Palmitoylation of caveolin-1 is required for cholesterol binding, chaperone complex formation, and rapid transport of cholesterol to caveolae.

A. Uittenbogaard, E. Smart, Annette M. Uittenbogaard, 2013, The Journal of Biological Chemistry.

Iron Toxicity in the Retina Requires Alu RNA and the NLRP3 Iron Toxicity in the Retina Requires Alu RNA and the NLRP3 Inflammasome Inflammasome

W. McDonald, J. Ambati, B. Fowler, 2022 .

Temperature-dependence of yadBC phenotypes in Yersinia pestis.

B. J. Hinnebusch, Amanda A. Gorman, A. Uittenbogaard, 2014, Microbiology.