S. Lomoio

发表

Functional and Sustainable 3D Human Neural Network Models from Pluripotent Stem Cells.

David L. Kaplan, D. Kaplan, M. Hunter, 2018, ACS biomaterials science & engineering.

Gga3 deletion and a GGA3 rare variant associated with late onset Alzheimer’s disease trigger BACE1 accumulation in axonal swellings

R. Tanzi, D. Prokopenko, M. Kennedy, 2020, Science Translational Medicine.

Pathophysiology of neurodegenerative diseases: An interplay among axonal transport failure, oxidative stress, and inflammation?

G. Tesco, S. Lomoio, 2022, Seminars in immunology.

A 3D bioengineered neural tissue model generated from patient-derived iPSCs develops Alzheimer‘s disease-related phenotypes

Ravi S. Pandey, D. Kaplan, P. Haydon, 2022, bioRxiv.

Dysfunction of the ubiquitin–proteasome system in the cerebellum of aging Ts65Dn mice

D. Necchi, S. Lomoio, E. Scherini, 2011, Experimental Neurology.

1 3 D bioengineered neural tissue generated from patient-derived iPSCs develops time-1 dependent phenotypes and transcriptional features of Alzheimer ’ s disease 2 3

Ravi S. Pandey, D. Kaplan, P. Haydon, 2022 .

Uneven distribution of NG2 cells in the rat cerebellar vermis and changes in aging

D. Necchi, S. Lomoio, E. Scherini, 2012, European journal of histochemistry : EJH.

A single episode of neonatal seizures alters the cerebellum of immature rats

D. Necchi, V. Mareš, S. Lomoio, 2011, Epilepsy Research.

β-Secretase BACE1 Promotes Surface Expression and Function of Kv3.4 at Hippocampal Mossy Fiber Synapses

C. Alzheimer, C. D'Avanzo, Doo Yeon Kim, 2018, The Journal of Neuroscience.

β-Amyloid overload does not directly correlate with SAPK/JNK activation and tau protein phosphorylation in the cerebellar cortex of Ts65Dn mice

D. Necchi, S. Lomoio, E. Scherini, 2009, Brain Research.

Axonal abnormalities in cerebellar Purkinje cells of the Ts65Dn mouse

D. Necchi, S. Lomoio, Selene Lomoio, 2008, Brain Research.

BACE1 elevation engendered by GGA3 deletion increases β-amyloid pathology in association with APP elevation and decreased CHL1 processing in 5XFAD mice

P. Haydon, Jinghui Dong, G. Tesco, 2018, Molecular Neurodegeneration.

Spatiotemporal processing of neural cell adhesion molecules 1 and 2 by BACE1 in vivo

G. Tesco, S. Lomoio, WonHee Kim, 2021, The Journal of biological chemistry.

Amyloid Generation and Dysfunctional Immunoproteasome Activation with Disease Progression in Animal Model of Familial Alzheimer's Disease

I. Ferrer, M. Portero-Otín, R. Pamplona, 2012, Brain pathology.

Genetic Deletion of the Clathrin Adaptor GGA3 Reduces Anxiety and Alters GABAergic Transmission

P. Haydon, S. Moss, G. Tesco, 2016, PloS one.

Cerebellar amyloid-β plaques: disturbed cortical circuitry in AβPP/PS1 transgenic mice as a model of familial Alzheimer's disease.

I. Ferrer, M. Carmona, E. Asó, 2012, Journal of Alzheimer's disease : JAD.

3D bioengineered neural tissue generated from patient-derived iPSCs mimics time-dependent phenotypes and transcriptional features of Alzheimer's disease.

D. Kaplan, P. Haydon, G. Carter, 2023, Molecular psychiatry.

BACE1 elevation engendered by GGA3 deletion increases β-amyloid pathology in association with APP elevation and decreased CHL1 processing in 5XFAD mice

P. Haydon, Jinghui Dong, G. Tesco, 2018, Molecular Neurodegeneration.