Samuel H Wilson

Coincidentally, this first issue marks the 50th anniversary of the seminal discovery of the excision-repair of damaged DNA. Preface.

Philip C Hanawalt, Samuel H Wilson, 2014, DNA repair.

In situ analysis of repair processes for oxidative DNA damage in mammalian cells.

Samuel H. Wilson, Satoshi Okano, Mitsuko Masutani, 2004, Proceedings of the National Academy of Sciences of the United States of America.

Modifying the beta,gamma leaving-group bridging oxygen alters nucleotide incorporation efficiency, fidelity, and the catalytic mechanism of DNA polymerase beta.

Arieh Warshel, Samuel H. Wilson, Jan Florián, 2007, Biochemistry.

Framework for environmental exposure research: the disease-first approach.

Samuel H. Wilson, William A Suk, W. Suk, 2005, Molecular interventions.

Alkylation DNA damage in combination with PARP inhibition results in formation of S-phase-dependent double-strand breaks.

Samuel H. Wilson, J. Horton, M. Heacock, 2010, DNA repair.

Ochratoxin A-induced mutagenesis in mammalian cells is consistent with the production of oxidative stress.

Samuel H. Wilson, Eugenia Dogliotti, E. Dogliotti, 2007, Chemical research in toxicology.

Structural insights into the origins of DNA polymerase fidelity.

Samuel H. Wilson, W. Beard, Samuel H Wilson, 2003, Structure.

Influence of DNA structure on DNA polymerase beta active site function: extension of mutagenic DNA intermediates.

Samuel H. Wilson, W. Beard, D. D. Shock, 2004, The Journal of biological chemistry.

Poly(ADP-ribose) Polymerase Activity Prevents Signaling Pathways for Cell Cycle Arrest after DNA Methylating Agent Exposure*

Samuel H. Wilson, J. Horton, P. Kedar, 2005, Journal of Biological Chemistry.

DNA polymerase beta fidelity: halomethylene-modified leaving groups in pre-steady-state kinetic analysis reveal differences at the chemical transition state.

Arieh Warshel, Samuel H. Wilson, Jan Florián, 2008, Biochemistry.