E. Prat

α-MSH Peptides Inhibit Production of Nitric Oxide and Tumor Necrosis Factor-α by Microglial Cells Activated with β-Amyloid and Interferon γ

J. M. Lipton, Daniela Galimberti, D. Galimberti, 1999 .

The immunopathogenesis of multiple sclerosis.

Roland Martin, Roland Martin, Elisabetta Prat, 2002, Journal of rehabilitation research and development.

The human astrocytoma cell line U373MG produces monocyte chemotactic protein (MCP)-1 upon stimulation with β-amyloid protein

Daniela Galimberti, D. Galimberti, G. Ardolino, 2000, Neuroscience Letters.

Peptidic complex mixtures as therapeutic agents in CNS autoimmunity.

Clemencia Pinilla, Claus-Steffen Stuerzebecher, Roland Martin, 2004, Molecular immunology.

HLA-DRB5*0101 and -DRB1*1501 expression in the multiple sclerosis-associated HLA-DR15 haplotype

Deric M. Park, Roland Martin, N. Kruse, 2005, Journal of Neuroimmunology.

Alpha-MSH peptides inhibit production of nitric oxide and tumor necrosis factor-alpha by microglial cells activated with beta-amyloid and interferon gamma.

J. M. Lipton, D. Galimberti, E. Scarpini, 1999, Biochemical and biophysical research communications.

Synergistic effect of beta-amyloid protein and interferon gamma on nitric oxide production by C2C12 muscle cells.

G Scarlato, D. Galimberti, E. Scarpini, 2000, Brain : a journal of neurology.

Proinflammatory profile of cytokine production by human monocytes and murine microglia stimulated with β-amyloid[25–35]

E. Scarpini, P. Baron, G. Scarlato, 1999, Journal of Neuroimmunology.

The immunopathogenesis of multiple sclerosis

R. Martin, E. Prat, 2002 .