J. Weis

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Role of Osteopontin in Murine Lyme Arthritis and Host Defense against Borrelia burgdorferi

C. Teuscher, D. Denhardt, S. Rittling, 2002, Infection and Immunity.

Lymphoid transcription of the murine CD21 gene is positively regulated by histone acetylation.

M. Zabel, J. Weis, J. Weis, 1999, Journal of immunology.

Dual Role of MyD88 in Rapid Clearance of Relapsing Fever Borrelia spp

S. Akira, T. Schwan, J. Weis, 2006, Infection and Immunity.

Altered localization of CXCL13 expressing cells in mice deficient in Pactolus following an inflammatory stimulus

R. Weiss, J. Weis, J. Weis, 2006, Immunology.

Surface Translocation of Pactolus Is Induced by Cell Activation and Death, but Is Not Required for Neutrophil Migration and Function 1

R. Margraf, J. Weis, J. Weis, 2003, The Journal of Immunology.

Transcriptional control of Pactolus: evidence of a negative control region and comparison with its evolutionary paralogue, CD18 (β2 integrin)

R. Margraf, J. S. Hale, J. Weis, 2006, Journal of leukocyte biology.

Borrelia burgdorferi outer surface lipoproteins OspA and OspB possess B-cell mitogenic and cytokine-stimulatory properties

J. Weis, Y. Ma, Ying Ma, 1993, Infection and immunity.

This information is current as Locus Ighm / Ighd within the Abrogating Transcription Termination Zfp 318 Regulates IgD Expression

Peter D. Pioli, J. Weis, I. Debnath, 2014 .

Analysis of the regulatory role of BAFF in controlling the expression of CD21 and CD23.

J. Weis, J. Weis, I. Debnath, 2007, Molecular immunology.

Defining In Vivo Transcription Factor Complexes of the Murine CD21 and CD23 Genes1

J. Weis, J. Weis, I. Debnath, 2007, The Journal of Immunology.

Demonstration of a B-lymphocyte mitogen produced by the Lyme disease pathogen, Borrelia burgdorferi

J. Weis, B. Araneo, R. Schoenfeld, 1992, Infection and immunity.

Overexpression of RANKL Implicates IFN‐β‐Mediated Elimination of B‐Cell Precursors in the Osteopetrotic Bone of Microphthalmic Mice

J. Weis, J. Weis, R. Smith, 2003, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.

Expression of the mouse fragilis gene products in immune cells and association with receptor signaling complexes

J. Weis, J. Weis, R. Smith, 2006, Genes and Immunity.

Overexpression of Snai3 suppresses lymphoid‐ and enhances myeloid‐cell differentiation

G. Spangrude, J. Weis, J. Weis, 2012, European journal of immunology.

High-throughput genotyping of advanced congenic lines by high resolution melting analysis for identification of Bbaa2, a QTL controlling Lyme arthritis.

C. Teuscher, J. Weis, J. Weis, 2012, BioTechniques.

DiseaseResponses in the Murine Model of Lyme Governing Arthritis Severity and Humoral Identification of Quantitative Trait Loci

J. Zachary, C. Teuscher, B. McCracken, 1998 .

Interval-Specific Congenic Lines Reveal Quantitative Trait Loci with Penetrant Lyme Arthritis Phenotypes on Chromosomes 5, 11, and 12

J. Zachary, R. Weiss, D. Dunn, 2009, Infection and Immunity.

T cell infiltration is associated with increased Lyme arthritis in TLR2-/- mice.

J. Zachary, R. Fujinami, J. Weis, 2008, FEMS immunology and medical microbiology.

A Critical Role for Type I IFN in Arthritis Development following Borrelia burgdorferi Infection of Mice1

J. Zachary, R. Schreiber, Jiantao Bian, 2008, The Journal of Immunology.

Gene Expression Profiling Reveals Unique Pathways Associated with Differential Severity of Lyme Arthritis1

R. M. Wooten, J. Zachary, R. Weiss, 2006, The Journal of Immunology.

FunctionRequired for Neutrophil Migration and by Cell Activation and Death, but Is Not Surface Translocation of Pactolus Is Induced

R. Margraf, J. Weis, J. Weis, 2003 .

S. Rogers, J. Weis, J. Weis, 2015, PloS one.

Interferon-inducible transmembrane proteins of the innate immune response act as membrane organizers by influencing clathrin and v-ATPase localization and function

J. Weis, J. Weis, K. Roundy, 2012, Innate immunity.

Control Region Modules Within the Murine CD21 Intronic Repeating, Cyclosporin-Sensitive Regulatory Yin Yang and NFAT-4

J. Weis, W. Wheeler, 2002 .

Yin Yang 1, Oct1, and NFAT-4 Form Repeating, Cyclosporin-Sensitive Regulatory Modules Within the Murine CD21 Intronic Control Region1

M. Zabel, J. Weis, J. Weis, 2002, The Journal of Immunology.

Intronic Elements Gene Depends on Accessibility of Promoter Cell-Specific Expression of the Murine CD 21

M. Zabel, J. Weis, Weis, 2000 .

Cell-Specific Expression of the Murine CD21 Gene Depends on Accessibility of Promoter and Intronic Elements1

M. Zabel, J. Weis, J. Weis, 2000, The Journal of Immunology.

The Lyme Disease Spirochete Borrelia burgdorferi Utilizes Multiple Ligands, Including RNA, for Interferon Regulatory Factor 3-Dependent Induction of Type I Interferon-Responsive Genes

J. Weis, J. Weis, Ying Ma, 2010, Infection and Immunity.

Snai2 and Snai3 transcriptionally regulate cellular fitness and functionality of T cell lineages through distinct gene programs.

Peter D. Pioli, J. Weis, J. Weis, 2016, Immunobiology.

Depletion of Lyn kinase from the BCR complex and inhibition of B cell activation by excess CD21 ligation.

M. Zabel, J. Weis, J. Weis, 2002, International immunology.

Intracellular localization of Borrelia burgdorferi within human endothelial cells

J. Weis, Ying Ma, Y. Ma, 1991, Infection and immunity.

Fatal autoimmunity results from the conditional deletion of Snai2 and Snai3.

Peter D. Pioli, J. Weis, J. Weis, 2015, Cellular immunology.

Gene expression profiling provides insights into the pathways involved in inflammatory arthritis development: murine model of Lyme disease.

J. Weis, Ying Ma, Jennifer C. Miller, 2008, Experimental and molecular pathology.

Relative Contributions of Innate and Acquired Host Responses to Bacterial Control and Arthritis Development in Lyme Disease

J. Zachary, C. Kirschning, J. Weis, 2005, Infection and Immunity.

Effect of Complement Component C3 Deficiency on Experimental Lyme Borreliosis in Mice

R. M. Wooten, J. Zachary, S. Norris, 2003, Infection and Immunity.

Interleukin-4 (IL-4) and IL-13 Signaling Pathways Do Not RegulateBorrelia burgdorferi-Induced Arthritis in Mice: IgG1 Is Not Required for Host Control of Tissue Spirochetes

C. Teuscher, N. Noben-Trauth, J. Weis, 2000, Infection and Immunity.

The role of CD14 in signaling mediated by outer membrane lipoproteins of Borrelia burgdorferi.

R. M. Wooten, R. Thieringer, T. B. Morrison, 1998, Journal of immunology.

Distinct Characteristics of Resistance toBorrelia burgdorferi-Induced Arthritis in C57BL/6N Mice

C. Teuscher, J. Weis, J. Weis, 1998, Infection and Immunity.

Nitric oxide production during murine Lyme disease: lack of involvement in host resistance or pathology

J. Weis, E. Eichwald, J. Hibbs, 1995, Infection and immunity.

Outer surface lipoproteins of Borrelia burgdorferi stimulate nitric oxide production by the cytokine-inducible pathway

M. Woods, J. Weis, K. Tai, 1994, Infection and immunity.

Heritable susceptibility to severe Borrelia burgdorferi-induced arthritis is dominant and is associated with persistence of large numbers of spirochetes in tissues

D. Persing, C. Kolbert, J. Weis, 1994 .

Normal human B lymphocytes and mononuclear cells respond to the mitogenic and cytokine-stimulatory activities of Borrelia burgdorferi and its lipoprotein OspA

J. Weis, K. Tai, Y. Ma, 1994, Infection and immunity.

Evidence for B-lymphocyte mitogen activity in Borrelia burgdorferi-infected mice

J. Weis, E. Eichwald, B. Araneo, 1992, Infection and immunity.

Genetic Control of Lyme Arthritis by Borrelia burgdorferi Arthritis–Associated Locus 1 Is Dependent on Localized Differential Production of IFN-β and Requires Upregulation of Myostatin

J. Zachary, C. Teuscher, J. Weis, 2017, The Journal of Immunology.

Infected Joint Tissue burgdorferi Borrelia Sources of Chemokines in Murine Lyme Disease and Are Major Arthritogenic Type I IFN Response in Endothelial Cells and Fibroblasts Amplify the

J. Zachary, J. Weis, J. Weis, 2014 .

Endothelial Cells and Fibroblasts Amplify the Arthritogenic Type I IFN Response in Murine Lyme Disease and Are Major Sources of Chemokines in Borrelia burgdorferi-Infected Joint Tissue

J. Zachary, J. Weis, J. Weis, 2012, The Journal of Immunology.

Toll-like receptor 2 plays a pivotal role in host defense and inflammatory response to Borrelia burgdorferi.

R. M. Wooten, J. Zachary, C. Kirschning, 2002, Vector borne and zoonotic diseases.

Biological activities of native and recombinant Borrelia burgdorferi outer surface protein A: dependence on lipid modification

J. Weis, Ying Ma, Y. Ma, 1994, Infection and immunity.

Heritable susceptibility to severe Borrelia burgdorferi-induced arthritis is dominant and is associated with persistence of large numbers of spirochetes in tissues.

D. Persing, C. Kolbert, J. Weis, 1994, Infection and immunity.

Identification of a partial cDNA clone for the C3d/Epstein-Barr virus receptor of human B lymphocytes: homology with the receptor for fragments C3b and C4b of the third and fourth components of complement.

D. Fearon, J. Smith, L. Klickstein, 1986, Proceedings of the National Academy of Sciences of the United States of America.

Borrelia burgdorferi outer membrane protein A induces nuclear translocation of nuclear factor-kappa B and inflammatory activation in human endothelial cells.

R. M. Wooten, T. McIntyre, V. Modur, 1996, Journal of immunology.

Tripalmitoyl-S-Glyceryl-Cysteine-Dependent OspA Vaccination of Toll-Like Receptor 2-Deficient Mice Results in Effective Protection from Borrelia burgdorferi Challenge

R. M. Wooten, M. Heilbrun, C. Kirschning, 2003, Infection and Immunity.

Receptor 2 Through Both Human and Murine Toll-Like Lipopolysaccharide Eliminates Signaling Cutting Edge: Repurification of

S. Vogel, J. Weis, J. Weis, 2000 .

Demonstration of Receptor Function of Membrane Proteins by Selection and Immobilization with Monoclonal Antibodies

D. Fearon, J. Weis, 1985 .

Immunity to Lyme disease: protection, pathology and persistence.

J. Weis, K. P. Seiler, 1996, Current opinion in immunology.

Fetal outcome in murine Lyme disease

R. Silver, D. Branch, R. Daynes, 1995, Infection and immunity.

The identification of N-linked oligosaccharides on the human CR2/Epstein-Barr virus receptor and their function in receptor metabolism, plasma membrane expression, and ligand binding.

D. Fearon, J. Weis, 1985, The Journal of biological chemistry.

The C3d Receptor Identified by the HB-5 Monoclonal Antibody: Expression and Role as a Receptor for Epstein-Barr Virus

D. Fearon, M. Cooper, T. Tedder, 1986 .

Identification of a 145,000 Mr membrane protein as the C3d receptor (CR2) of human B lymphocytes.

D. Fearon, T. Tedder, J. Weis, 1984, Proceedings of the National Academy of Sciences of the United States of America.

The role of receptors for complement in the induction of polyclonal B-cell proliferation and differentiation

D. Fearon, M. Cooper, T. Tedder, 2004, Journal of Clinical Immunology.

Murine complement receptor 1 is required for germinal center B cell maintenance but not initiation.

J. Weis, J. Weis, Luke R. Donius, 2014, Immunobiology.

Partial rescue of B cells in microphthalmic osteopetrotic marrow by loss of response to type I IFNs.

G. Spangrude, J. Weis, J. Weis, 2005, International immunology.

Mice Lacking CD21 and CD35 Proteins Mount Effective Immune Responses against Borrelia burgdorferi Infection

J. Zachary, J. Weis, J. Weis, 2007, Infection and Immunity.

The in vitro derivation of phenotypically mature and diverse B cells from immature spleen and bone marrow precursors

J. Weis, J. Weis, K. Roundy, 2010, European journal of immunology.

Variability of CR2 gene products is due to alternative exon usage and different CR2 alleles.

J. Weis, L. Toothaker, A. J. Henjes, 1989, Journal of immunology.

Host-pathogen interactions promoting inflammatory Lyme arthritis: use of mouse models for dissection of disease processes.

R. M. Wooten, J. Weis, R. Wooten, 2001, Current opinion in microbiology.

Epstein-Barr virus receptor of human B lymphocytes is the C3d receptor CR2.

P. A. Biro, J. Strominger, D. Fearon, 1984, Proceedings of the National Academy of Sciences of the United States of America.

Cutting Edge: Repurification of Lipopolysaccharide Eliminates Signaling Through Both Human and Murine Toll-Like Receptor 21

S. Vogel, Stefanie N. Vogel, J. Weis, 2000, The Journal of Immunology.

Cutting edge: inflammatory signaling by Borrelia burgdorferi lipoproteins is mediated by toll-like receptor 2.

R. M. Wooten, C. Kirschning, J. Weis, 1999, Journal of immunology.

Signaling by Toll-Like Receptor 2 and 4 Agonists Results in Differential Gene Expression in Murine Macrophages

M. J. Cody, S. Michalek, S. Vogel, 2001, Infection and Immunity.

Deletion of Snai2 and Snai3 Results in Impaired Physical Development Compounded by Lymphocyte Deficiency

Peter D. Pioli, J. Weis, J. Weis, 2013, PloS one.

Zfp318 Regulates IgD Expression by Abrogating Transcription Termination within the Ighm/Ighd Locus

Peter D. Pioli, J. Weis, J. Weis, 2014, Journal of Immunology.

Microphthalmic mice display a B cell deficiency similar to that seen for mast and NK cells.

A. Kollhoff, J. Weis, J. Weis, 1999, Journal of immunology.

Epstein-barr virus gp350/220 binding to the B lymphocyte C3d receptor mediates adsorption, capping, and endocytosis

E. Kieff, D. Fearon, J. Weis, 1987, Cell.

Mouse chromosome 11 harbors genetic determinants of hippocampal strain‐specific nicotinic receptor expression

S. Rogers, C. Teuscher, J. Weis, 2008, Hippocampus.

Bystander T Cells: A Balancing Act of Friends and Foes.

Matthew A. Williams, J. Weis, Jeremy P. Snook, 2018, Trends in immunology.

Functional Characterization of Pactolus, a β-Integrin-like Protein Preferentially Expressed by Neutrophils*

J. Weis, J. Weis, S. Garrison, 2001, The Journal of Biological Chemistry.

Lyme arthritis: linking infection, inflammation and autoimmunity

A. Steere, J. Weis, K. Strle, 2021, Nature Reviews Rheumatology.

Resistance to Lyme disease in decorin-deficient mice.

B. P. Guo, R. M. Wooten, Amanda P. Smith, 2001, The Journal of clinical investigation.

Dual Role of Interleukin-10 in Murine Lyme Disease: Regulation of Arthritis Severity and Host Defense

R. M. Wooten, J. Zachary, C. Teuscher, 1999, Infection and Immunity.

Rapid and Sensitive Quantification ofBorrelia burgdorferi-Infected Mouse Tissues by Continuous Fluorescent Monitoring of PCR

J. Weis, J. Weis, Ying Ma, 1999, Journal of Clinical Microbiology.

Pathological manifestations in murine Lyme disease: association with tissue invasion and spirochete persistence.

R. Silver, J. Weis, L. Yang, 1997, Clinical infectious diseases : an official publication of the Infectious Diseases Society of America.

Localized Production of IL-10 Suppresses Early Inflammatory Cell Infiltration and Subsequent Development of IFN-γ–Mediated Lyme Arthritis

J. Zachary, G. Spangrude, Xiaosong Huang, 2012, The Journal of Immunology.

Identification of a 145 , 000 Mr membrane protein as the C 3 d receptor ( CR 2 ) of human

D. Fearon, J. Weis, B. Iymphocytes, 2022 .

Subsequent Development of IFN- Early Inflammatory Cell Infiltration and Localized Production of IL-10 Suppresses

J. Zachary, G. Spangrude, Xiaosong Huang, 2013 .

MyD88 Plays a Unique Role in Host Defense but Not Arthritis Development in Lyme Disease1

S. Akira, J. Zachary, C. Kirschning, 2004, The Journal of Immunology.

Defining a transcriptional fingerprint of murine splenic B-cell development

R. Weiss, J. Weis, J. Weis, 2008, Genes and Immunity.

Molecular Mimicry toBorrelia burgdorferi: Pathway to Autoimmunity?

J. Weis, Devin D Bolz, 2004, Autoimmunity.

Host-pathogen interactions and the pathogenesis of murine Lyme disease.

J. Weis, Janis J Weis, 2002, Current opinion in rheumatology.

IL-10 Deficiency Reveals a Role for TLR2-Dependent Bystander Activation of T Cells in Lyme Arthritis

J. Zachary, J. Round, Matthew A. Williams, 2018, The Journal of Immunology.

Antagonistic Interplay between MicroRNA-155 and IL-10 during Lyme Carditis and Arthritis

Ryan M. O’Connell, J. Zachary, J. Weis, 2015, PloS one.

Borrelia burgdorferi Arthritis–Associated Locus Bbaa1 Regulates Lyme Arthritis and K/B×N Serum Transfer Arthritis through Intrinsic Control of Type I IFN Production

J. Zachary, C. Teuscher, J. Weis, 2014, The Journal of Immunology.

MicroRNA-146a Provides Feedback Regulation of Lyme Arthritis but Not Carditis during Infection with Borrelia burgdorferi

Ryan M. O’Connell, D. Baltimore, J. Zachary, 2014, PLoS pathogens.

Forward genetic approaches for elucidation of novel regulators of Lyme arthritis severity

C. Teuscher, J. Weis, Kenneth K. C. Bramwell, 2014, Front. Cell. Infect. Microbiol..

Lysosomal β-glucuronidase regulates Lyme and rheumatoid arthritis severity.

J. Zachary, C. Teuscher, J. Weis, 2014, The Journal of clinical investigation.

Impaired host defense to infection and Toll-like receptor 2-independent killing of Borrelia burgdorferi clinical isolates in TLR2-deficient C3H/HeJ mice.

Guiqing Wang, I. Schwartz, J. Weis, 2004, FEMS microbiology letters.

Defining In Vivo Transcription Factor Complexes of the Murine CD21 and CD23 Genes1

J. Weis, J. Weis, I. Debnath, 2007, The Journal of Immunology.

Toll-like receptors mediate induction of hepcidin in mice infected with Borrelia burgdorferi.

L. Bockenstedt, J. Kushner, J. Kaplan, 2009, Blood.

Generation of a novel Cr2 gene allele by homologous recombination that abrogates production of Cr2 but is sufficient for expression of Cr1.

J. Weis, J. Weis, Luke R. Donius, 2014, Immunobiology.

Optimal Germinal Center B Cell Activation and T-Dependent Antibody Responses Require Expression of the Mouse Complement Receptor Cr1

J. Weis, J. Weis, Luke R. Donius, 2013, The Journal of Immunology.

Bone marrow-induced Mef2c deficiency delays B-cell development and alters the expression of key B-cell regulatory proteins.

Peter D. Pioli, J. Weis, J. Weis, 2013, International immunology.

Complement Receptor Cr 1 Require Expression of the Mouse and T-Dependent Antibody Responses Optimal Germinal Center B Cell Activation

J. Weis, Luke R. Donius, J. Handy, 2013 .

Deletion of putative intronic control sequences does not alter cell or stage specific expression of Cr2.

J. Weis, J. Weis, K. Roundy, 2009, Molecular immunology.

Expression of the murine CD21 gene is regulated by promoter and intronic sequences.

J. Weis, J. Weis, H. Hu, 1997, Journal of immunology.

β-Glucuronidase, a Regulator of Lyme Arthritis Severity, Modulates Lysosomal Trafficking and MMP-9 Secretion in Response to Inflammatory Stimuli

C. Teuscher, J. Weis, J. Weis, 2015, The Journal of Immunology.

Loss of human CR1- and murine Crry-like exons in human CR2 transcripts due to CR2 gene mutations.

C. Kurtz, C. Parker, J. Weis, 1990, Journal of immunology.

Complement Receptors 1 and 2 Influence the Immune Environment in a B Cell Receptor-Independent Manner12

J. Weis, J. Weis, A. Jacobson, 2008, The Journal of Immunology.

A complement receptor locus: genes encoding C3b/C4b receptor and C3d/Epstein-Barr virus receptor map to 1q32.

C. Morton, G. Bruns, D. Fearon, 1987, Journal of immunology.

Murine complement receptor gene family. II. Identification and characterization of the murine homolog (Cr2) to human CR2 and its molecular linkage to Crry.

M. Aegerter, C. Kurtz, J. Weis, 1989, Journal of immunology.

Borrelia burgdorferi outer surface protein A (OspA) activates and primes human neutrophils.

T. B. Morrison, J. Weis, J. Weis, 1997, Journal of immunology.

Genomic organization, chromosomal localization, and transcriptional variants of the murine pactolus gene

R. Margraf, J. Weis, J. Weis, 1999, Mammalian Genome.

Bb2Bb3 regulation of murine Lyme arthritis is distinct from Ncf1 and independent of the phagocyte nicotinamide adenine dinucleotide phosphate oxidase.

J. Zachary, R. Weiss, D. Dunn, 2005, The American journal of pathology.

Delineating the Requirement for the Borrelia burgdorferi Virulence Factor OspC in the Mammalian Host

J. A. Carroll, P. Stewart, P. Rosa, 2006, Infection and Immunity.

Toll-Like Receptor 2 Is Required for Innate, But Not Acquired, Host Defense to Borrelia burgdorferi1

R. M. Wooten, J. Zachary, C. Kirschning, 2002, The Journal of Immunology.

Association of Persistent Symptoms after Lyme Neuroborreliosis and Increased Levels of Interferon-α in Blood

F. Strle, K. Kelley, Andrej Kastrin, 2023, Emerging infectious diseases.

The Cdkn2a gene product p19 alternative reading frame (p19ARF) is a critical regulator of IFNβ-mediated Lyme arthritis

J. Zachary, C. Teuscher, J. Weis, 2022, PLoS pathogens.

Toll-Like Receptor 2 Is Required for Innate, But Not Acquired, Host Defense toBorrelia burgdorferi

J. Zachary, C. Kirschning, J. Weis, 2001 .

Identification of Pactolus, an Integrin β Subunit-like Cell-surface Protein Preferentially Expressed by Cells of the Bone Marrow*

J. Weis, J. Weis, S. Garrison, 1998, The Journal of Biological Chemistry.

Receptor-Independent Manner Immune Environment in a B Cell Complement Receptors 1 and 2 Influence the

J. Weis, A. Jacobson, 2008 .

Purification of the B lymphocyte receptor for the C3d fragment of complement and the Epstein-Barr virus by monoclonal antibody affinity chromatography, and assessment of its functional capacities.

D. Fearon, J. Smith, J. Weis, 1986, Journal of immunological methods.

CD28 expression by mouse mast cells is modulated by lipopolysaccharide and outer surface protein A lipoprotein from Borrelia burgdorferi.

E. Marietta, J. Weis, J. Weis, 1997, Journal of immunology.

Regulation of Murine Splenic B Cell CR3 Expression by Complement Component 31

J. Weis, J. Weis, K. Roundy, 2009, The Journal of Immunology.

Expression by Complement Component 3 Regulation of Murine Splenic B Cell CR 3

J. Weis, J. Weis, K. Roundy, 2009 .

Quantification of low-copy transcripts by continuous SYBR Green I monitoring during amplification.

T. B. Morrison, C. Wittwer, J. Weis, 1998, BioTechniques.

Identification of quantitative trait loci governing arthritis severity and humoral responses in the murine model of Lyme disease.

R. Doerge, J. Zachary, T. B. Morrison, 1999, Journal of immunology.

CD21 signaling via C3 regulates Purkinje cell protein 4 expression.

J. Weis, J. Weis, A. Jacobson, 2009, Molecular immunology.

Interval-Specific Congenic Lines Reveal Quantitative Trait Loci with Penetrant Lyme Arthritis Phenotypes on Chromosomes 5, 11, and 12

J. Zachary, R. Weiss, D. Dunn, 2009, Infection and Immunity.

Structure of the human B lymphocyte receptor for C3d and the Epstein- Barr virus and relatedness to other members of the family of C3/C4 binding proteins [published erratum appears in J Exp Med 1988 Nov 1;168(5):1953-4]

D. Fearon, J. Smith, J. Weis, 1988, The Journal of experimental medicine.

Lysosomal β-glucuronidase regulates Lyme and rheumatoid arthritis severity.

J. Zachary, C. Teuscher, J. Weis, 2014, The Journal of clinical investigation.

Generation of a novel Cr2 gene allele by homologous recombination that abrogates production of Cr2 but is sufficient for expression of Cr1.

J. Weis, J. Weis, Luke R. Donius, 2014, Immunobiology.

Structure of the human B lymphocyte receptor for C3d and the Epstein-Barr virus and relatedness to other members of the family of C3/C4 binding protein

J. Weis, Lorraine E. Toothaker, Douglas T Fearon, 1988 .

This information is current as Infection of Mice Borrelia burgdorferi Development following A Critical Role for Type I IFN in Arthritis

J. Zachary, J. Weis, Jenn Miller, 2008 .

Genetic control of susceptibility to experimental Lyme arthritis is polygenic and exhibits consistent linkage to multiple loci on chromosome 5 in four independent mouse crosses

R. Doerge, J. Zachary, C. Teuscher, 2001, Genes and Immunity.

Control Region Modules Within the Murine CD21 Intronic Repeating, Cyclosporin-Sensitive Regulatory Yin Yang and NFAT-4 Form

J. Weis, W. Wheeler, 2002 .

Control Region Modules Within the Murine CD21 Intronic Repeating, Cyclosporin-Sensitive Regulatory Yin Yang and NFAT-4 Form

J. Weis, W. Wheeler, 2002 .

by Toll-Like Receptor 2 Lipoproteins Is Mediated Borrelia burgdorferi Cutting Edge: Inflammatory Signaling by

C. Kirschning, J. Weis, J. Weis, 2015 .

Toll-Like Receptor 2 Lipoproteins Is Mediated Borrelia burgdorferi Cutting Edge : Inflammatory Signaling by Wooten and

R. M. Wooten, C. Kirschning, J. Weis, 1999 .

E and P Selectins Are Not Required for Resistance to Severe Murine Lyme Arthritis

J. Weis, K. P. Seiler, Ying Ma, 1998, Infection and Immunity.