J Pfeilschifter

Induction of inducible nitric oxide synthase and its corresponding tetrahydrobiopterin-cofactor-synthesizing enzyme GTP-cyclohydrolase I during cutaneous wound repair.

S. Werner, J. Pfeilschifter, S Frank, 1998, The Journal of investigative dermatology.

Large and sustained induction of chemokines during impaired wound healing in the genetically diabetic mouse: prolonged persistence of neutrophils and macrophages during the late phase of repair.

J. Pfeilschifter, B. Stallmeyer, S Frank, 2000, The Journal of investigative dermatology.

Regulation of interleukin-18 (IL-18) expression in keratinocytes (HaCaT): implications for early wound healing.

H. Mühl, J. Pfeilschifter, U. Kalina, 2000, European cytokine network.

Macrophage-Derived Heme-Oxygenase-1: Expression, Regulation, and Possible Functions in Skin Repair

J. Pfeilschifter, S Frank, J Pfeilschifter, 2001, Molecular medicine.

The function of nitric oxide in wound repair: inhibition of inducible nitric oxide-synthase severely impairs wound reepithelialization.

J. Pfeilschifter, B. Stallmeyer, S Frank, 1999, The Journal of investigative dermatology.

Activation of protein kinase C subtypes alpha, gamma, delta, epsilon, zeta, and eta by tumor-promoting and nontumor-promoting agents.

D. Fabbro, T. Meyer, J. Pfeilschifter, 1997, Biochemical pharmacology.

Selective inhibition of cyclooxygenase 2.

J. Pfeilschifter, V. Ullrich, R. Nüsing, 1994, Biochemical pharmacology.

Leptin enhances wound re-epithelialization and constitutes a direct function of leptin in skin repair.

J. Pfeilschifter, B. Stallmeyer, S Frank, 2000, The Journal of clinical investigation.