J. Jacobson

发表

Effect of an amber mutation in the herpes simplex virus thymidine kinase gene on polypeptide synthesis and stability.

M. Manos, D. Coen, J. Gibbs, 1989, Virology.

Nonnucleoside Pyrrolopyrimidines with a Unique Mechanism of Action against Human Cytomegalovirus

M. Nassiri, J. Jacobson, T. Renau, 1999, Antimicrobial Agents and Chemotherapy.

Evidence for a novel regulatory pathway for herpes simplex virus gene expression in trigeminal ganglion neurons

D. Coen, D. Knipe, J. Jacobson, 1993, Journal of virology.

Very late activation antigens on rheumatoid synovial fluid T lymphocytes. Association with stages of T cell activation.

D. Glass, M. Hemler, J. Coblyn, 1986, The Journal of clinical investigation.

VLA‐1: a T cell surface antigen which defines a novel late stage of human T cell activation

J. Strominger, D. Mann, M. Brenner, 1985, European journal of immunology.

Biochemical characterization of VLA-1 and VLA-2. Cell surface heterodimers on activated T cells.

J. Strominger, M. Hemler, J. Jacobson, 1985, The Journal of biological chemistry.

Herpes simplex virus thymidine kinase and specific stages of latency in murine trigeminal ganglia

K. Wobbe, D. Coen, D. Knipe, 1993, Journal of virology.

Importance of the herpes simplex virus UL24 gene for productive ganglionic infection in mice.

D. Coen, W. Cook, Shun-Hua Chen, 1998, Virology.

Synergistic effects on ganglionic herpes simplex virus infections by mutations or drugs that inhibit the viral polymerase and thymidine kinase.

D. Coen, F. Rozenberg, J. Jacobson, 1995, Virology.

Thymidine kinase-negative herpes simplex virus mutants establish latency in mouse trigeminal ganglia but do not reactivate.

K. Tyler, P. Schaffer, D. Coen, 1989, Proceedings of the National Academy of Sciences of the United States of America.

A herpes simplex virus ribonucleotide reductase deletion mutant is defective for productive acute and reactivatable latent infections of mice and for replication in mouse cells.

S. Weller, P. Schaffer, D. Coen, 1989, Virology.

A conserved open reading frame that overlaps the herpes simplex virus thymidine kinase gene is important for viral growth in cell culture

D. Coen, S. Martin, J. Jacobson, 1989, Journal of virology.

Low levels of herpes simplex virus thymidine- thymidylate kinase are not limiting for sensitivity to certain antiviral drugs or for latency in a mouse model.

D. Coen, K. Kerns, A. Irmiere, 1989, Virology.

Linker Insertion Mutations in the Herpes Simplex Virus Type 1 UL28 Gene: Effects on UL28 Interaction with UL15 and UL33 and Identification of a Second-Site Mutation in the UL15 Gene That Suppresses a Lethal UL28 Mutation

F. Homa, J. Baines, J. Jacobson, 2006, Journal of Virology.

Thymidine kinase-negative herpes simplex virus mutants establish latency in mouse trigeminal ganglia but do not reactivate.

D. Knipe, J. Jacobson, Connie L. Bogard, 1989, Proceedings of the National Academy of Sciences of the United States of America.