E. Dragicevic

发表

Predicting cardiac safety using human induced pluripotent stem cell-derived cardiomyocytes combined with multi-electrode array (MEA) technology: A conference report.

Jean-Pierre Valentin, Udo Kraushaar, Elena Dragicevic, 2018, Journal of pharmacological and toxicological methods.

NEUROSCIENCE FOREFRONT REVIEW DOPAMINE MIDBRAIN NEURONS IN HEALTH AND PARKINSON'S DISEASE: EMERGING ROLES OF VOLTAGE-GATED CALCIUM CHANNELS AND ATP-SENSITIVE POTASSIUM CHANNELS

B. Liss, J. Schiemann, E. Dragicevic, 2015 .

Integration of mechanical conditioning into a high throughput contractility assay for cardiac safety assessment.

N. Fertig, M. George, S. Stoelzle-Feix, 2020, Journal of pharmacological and toxicological methods.

TRPC3 Channels Are Required for Synaptic Transmission and Motor Coordination

Marc Freichel, Jana Hartmann, Alexander Dietrich, 2008, Neuron.

Dopamine midbrain neurons in health and Parkinson’s disease: Emerging roles of voltage-gated calcium channels and ATP-sensitive potassium channels

J. Schiemann, B. Liss, J. Schiemann, 2015, Neuroscience.

Pain relief in a neuropathy patient by lacosamide: Proof of principle of clinical translation from patient-specific iPS cell-derived nociceptors

J. Schüttler, A. Winterpacht, B. Winner, 2018, EBioMedicine.

Cav2.3 channels contribute to dopaminergic neuron loss in a model of Parkinson’s disease

O. Pongs, B. Liss, P. Kloppenburg, 2019, Nature Communications.

Orchestrated increase of dopamine and PARK mRNAs but not miR-133b in dopamine neurons in Parkinson's disease

Jan Gründemann, J. Hardy, B. Liss, 2014, Neurobiology of Aging.

Cav1.3 channels control D2-autoreceptor responses via NCS-1 in substantia nigra dopamine neurons

Masahiko Watanabe, S. Lammel, R. Malenka, 2014, Brain : a journal of neurology.

Compensatory T-type Ca2+ channel activity alters D2-autoreceptor responses of Substantia nigra dopamine neurons from Cav1.3 L-type Ca2+ channel KO mice

B. Liss, J. Striessnig, J. Benkert, 2015, Scientific Reports.

Cav2.3 channels contribute to dopaminergic neuron loss in a model of Parkinsonâ€TMs disease

O. Pongs, B. Liss, P. Kloppenburg, 2019 .