H C Lane

Human immunodeficiency virus type 1 quasi species that rebound after discontinuation of highly active antiretroviral therapy are similar to the viral quasi species present before initiation of therapy.

K. Crandall, R. Dewar, J. Metcalf, 2001, The Journal of infectious diseases.

Parallel evolution of drug resistance in HIV: failure of nonsynonymous/synonymous substitution rate ratio to detect selection.

K. Crandall, H. Lane, C. Kelsey, 1999, Molecular biology and evolution.

Bioterrorism on the home front: a new challenge for American medicine.

A. Fauci, H. Lane, A S Fauci, 2001, JAMA.

Characterization of GP120 binding to CD4 and an assay that measures ability of sera to inhibit this binding.

A. Fauci, H. Lane, S. Koenig, 1988, Journal of immunology.

Integrated proviral human immunodeficiency virus type 1 is present in CD4+ peripheral blood lymphocytes in healthy seropositive individuals

M. Baseler, A. Fauci, H. Lane, 1989, Journal of virology.

Longitudinal changes in CD4+ T cell antigen receptor diversity and naive/memory cell phenotype during 9 to 26 months of antiretroviral therapy of HIV-infected patients.

C. Hallahan, M. Baseler, Michael A. Polis, 2000, AIDS research and human retroviruses.

Preferential infection of CD4+ memory T cells by human immunodeficiency virus type 1: evidence for a role in the selective T-cell functional defects observed in infected individuals.

J. Justement, M. Baseler, A. Fauci, 1990, Proceedings of the National Academy of Sciences of the United States of America.

Diagnosis of Pneumocystis carinii pneumonia: improved detection in sputum with use of monoclonal antibodies.

J E Parrillo, F. Ognibene, J. Shelhamer, 1988, The New England journal of medicine.

The reservoir for HIV-1 in human peripheral blood is a T cell that maintains expression of CD4.

C H Fox, C. Fox, M. Baseler, 1989, Science.