Joel A Black

Abnormal sodium channel distribution in optic nerve axons in a model of inflammatory demyelination.

Stephen G Waxman, A. Lo, S. Waxman, 2003, Brain : a journal of neurology.

Voltage-gated Na+ channels in glia: properties and possible functions

Harald Sontheimer, Stephen G Waxman, S. Waxman, 1996, Trends in Neurosciences.

Molecular changes in neurons in multiple sclerosis: altered axonal expression of Nav1.2 and Nav1.6 sodium channels and Na+/Ca2+ exchanger.

Jia Newcombe, Stephen G Waxman, S. Waxman, 2004, Proceedings of the National Academy of Sciences of the United States of America.

Na+ channel expression along axons in multiple sclerosis and its models.

Stephen G Waxman, S. Waxman, Matthew J Craner, 2004, Trends in pharmacological sciences.

Co-localization of sodium channel Nav1.6 and the sodium-calcium exchanger at sites of axonal injury in the spinal cord in EAE.

Stephen G Waxman, A. Lo, S. Waxman, 2004, Brain : a journal of neurology.

Primary cortical motor neurons undergo apoptosis after axotomizing spinal cord injury

Stephen G Waxman, S. Waxman, J. A. Black, 2003, The Journal of comparative neurology.

Introduction. Astrocytes in the injured brain: is gliosis adaptive or maladaptive?

George E Barreto, G. Barreto, Joel A Black, 2014, Neuroscience letters.

Temporal Course of Upregulation of Nav1.8 in Purkinje Neurons Parallels the Progression of Clinical Deficit in Experimental Allergic Encephalomyelitis

Stephen G Waxman, A. Lo, S. Waxman, 2003, Journal of neuropathology and experimental neurology.

Long-term protection of central axons with phenytoin in monophasic and chronic-relapsing EAE.

Stephen G Waxman, S. Waxman, J. A. Black, 2006, Brain : a journal of neurology.