Anne-Katrine Frank

发表

Disease evolution and outcomes in familial AML with germline CEBPA mutations.

J. Gribben, B. Porse, L. Bullinger, 2015, Blood.

C/EBPα Is Required for Long-Term Self-Renewal and Lineage Priming of Hematopoietic Stem Cells and for the Maintenance of Epigenetic Configurations in Multipotent Progenitors

Philipp S. Hoppe, F. Lauridsen, B. Porse, 2014, PLoS genetics.

Lack of the p42 form of C/EBPα leads to spontaneous immortalization and lineage infidelity of committed myeloid progenitors.

B. Porse, N. Rapin, F. O. Bagger, 2013, Experimental hematology.

Engineering human mini-bones for the standardized modeling of healthy hematopoiesis, leukemia, and solid tumor metastasis

P. Bourgine, B. Porse, Kristina Aaltonen, 2022, Science Translational Medicine.

Phosphorylation of Serine 248 of C/EBPα Is Dispensable for Myelopoiesis but Its Disruption Leads to a Low Penetrant Myeloid Disorder with Long Latency

B. Porse, T. Pedersen, C. Nerlov, 2012, PloS one.

PTBP1 promotes hematopoietic stem cell maintenance and red blood cell development by ensuring sufficient availability of ribosomal constituents.

B. Porse, M. Michaut, N. Rapin, 2022, Cell reports.

Amplification of pico-scale DNA mediated by bacterial carrier DNA for small-cell-number transcription factor ChIP-seq

B. Porse, J. Waage, F. O. Bagger, 2015, BMC Genomics.

Regulation of Trib2 by an E2F1-C/EBPα feedback loop in AML cell proliferation.

Marie S. Hasemann, B. Porse, K. Keeshan, 2014, Blood.

ERG promotes the maintenance of hematopoietic stem cells by restricting their differentiation

Marie S. Hasemann, Lina A. Thoren, B. Porse, 2015, Genes & development.

C/EBPα Is Required for Long-Term Self-Renewal and Lineage Priming of Hematopoietic Stem Cells and for the Maintenance of Epigenetic Configurations in Multipotent Progenitors

Marie S. Hasemann, Philipp S. Hoppe, F. Lauridsen, 2014, PLoS genetics.

Amplification of pico-scale DNA mediated by bacterial carrier DNA for small-cell-number transcription factor ChIP-seq

Marie S. Hasemann, B. Porse, J. Waage, 2015, BMC Genomics.

TET2 lesions enhance the aggressiveness of CEBPA-mutant acute myeloid leukemia by rebalancing GATA2 expression

B. Porse, T. Haferlach, F. Grebien, 2023, Nature communications.

Lack of the p42 form of C/EBPα leads to spontaneous immortalization and lineage infidelity of committed myeloid progenitors.

B. Porse, N. Rapin, F. O. Bagger, 2013, Experimental hematology.

MYELOID NEOPLASIA Disease evolution and outcomes in familial AML with germline CEBPA mutations

J. Gribben, K. Döhner, C. Chelala, 2015 .