L. Scrocchi

发表

Abnormal Cognition, Sleep, EEG and Brain Metabolism in a Novel Knock-In Alzheimer Mouse, PLB1

Emanuele Porcu, Gernot Riedel, Bettina Platt, 2011, PloS one.

Inhibitors of islet amyloid polypeptide fibrillogenesis, and the treatment of type-2 diabetes

P. Fraser, L. Scrocchi, Ling Wu, 2003, Letters in Peptide Science.

Inhibitors of islet amyloid polypeptide fibrillogenesis, and the treatment of type-2 diabetes

P. Fraser, L. Scrocchi, Ling Wu, 2003, Letters in Peptide Science.

Design of peptide-based inhibitors of human islet amyloid polypeptide fibrillogenesis.

P. Fraser, J. McLaurin, L. Scrocchi, 2002, Journal of molecular biology.

Leptin Sensitivity in Nonobese Glucagon-Like Peptide I Receptor −/− Mice

D. Drucker, L. Scrocchi, T. Brown, 1997, Diabetes.

New Developments in the Biology of the Glucagon‐Like Peptides GLP‐1 and GLP‐2

B. Yusta, D. Drucker, Yuanfang Liu, 2000, Annals of the New York Academy of Sciences.

Mouse pancreatic beta-cells exhibit preserved glucose competence after disruption of the glucagon-like peptide-1 receptor gene.

D. Drucker, D. Pipeleers, F. Schuit, 1998, Diabetes.

Exendin-(9-39) is an inverse agonist of the murine glucagon-like peptide-1 receptor: implications for basal intracellular cyclic adenosine 3',5'-monophosphate levels and beta-cell glucose competence.

D. Drucker, B. Thorens, S. Efrat, 1998, Endocrinology.

Exendin-(9-39) Is an Inverse Agonist of the Murine Glucagon-Like Peptide-1 Receptor: Implications for Basal Intracellular Cyclic Adenosine 3',5'-Monophosphate Levels and β-Cell Glucose Competence* *This work was supported by Grant 31-46958.96 from the Swiss National Science Foundation (to B.T.).

D. Drucker, B. Thorens, S. Efrat, 1998, Endocrinology.

Glucocorticoids induce corticosteroid-binding globulin biosynthesis by immature mouse liver and kidney

G. Hammond, L. Scrocchi, Xianya Zhao, 1997, The Journal of Steroid Biochemistry and Molecular Biology.

Neuroendocrine function and response to stress in mice with complete disruption of glucagon-like peptide-1 receptor signaling.

D. Drucker, S. Asa, F. Vaccarino, 2000, Endocrinology.

Enhanced glucose-dependent insulinotropic polypeptide secretion and insulinotropic action in glucagon-like peptide 1 receptor -/- mice.

R. Pederson, D. Drucker, C. Mcintosh, 1998, Diabetes.

Mouse Pancreatic-Cells Exhibit Preserved Glucose Competence After Disruption of the Glucagon-Like Peptide-1 Receptor G e n e

D. Drucker, D. Pipeleers, F. Schuit, 1998 .

Neuroendocrine Function and Response to Stress in Mice with Complete Disruption of Glucagon-Like Peptide-1 Receptor Signaling1.

D. Drucker, S. Asa, F. Vaccarino, 2000, Endocrinology.

Glucose intolerance but normal satiety in mice with a null mutation in the glucagon–like peptide 1 receptor gene

A. Joyner, D. Drucker, N. MacLusky, 1996, Nature Medicine.

Identification of minimal peptide sequences in the (8-20) domain of human islet amyloid polypeptide involved in fibrillogenesis.

P. Fraser, L. Scrocchi, Ling Wu, 2003, Journal of structural biology.

Effects of aging and a high fat diet on body weight and glucose tolerance in glucagon-like peptide-1 receptor -/- mice.

D. Drucker, L. Scrocchi, 1998, Endocrinology.

Targeted delivery of ribavirin improves outcome of murine viral fulminant hepatitis via enhanced anti‐viral activity†

R. Gorczynski, Williams He, I. McGilvray, 2006, Hepatology.

A demonstration of analytical similarity comparing a proposed biosimilar pegfilgrastim and reference pegfilgrastim.

S. Brokx, L. Scrocchi, J. Dowd, 2017, Biologicals : journal of the International Association of Biological Standardization.

Amyloid inhibitors enhance survival of cultured human islets.

P. Fraser, G. Warnock, M. Lipsett, 2009, Biochimica et biophysica acta.

Elimination of glucagon-like peptide 1R signaling does not modify weight gain and islet adaptation in mice with combined disruption of leptin and GLP-1 action.

B Perkins, J Saleh, D. Drucker, 2000, Diabetes.

Corticosteroid-binding globulin biosynthesis in the mouse liver and kidney during postnatal development.

V. Han, G. Hammond, L. Scrocchi, 1993, Endocrinology.

Spatial and temporal distribution of corticosteroid-binding globulin and its messenger ribonucleic acid in embryonic and fetal mice.

V. Han, G. Hammond, L. Scrocchi, 1993, Endocrinology.

Pathological Hallmarks, Clinical Parallels, and Value for Drug Testing in Alzheimer's Disease of the APP[V717I] London Transgenic Mouse Model

T. V. Van Dooren, F. van Leuven, A. Tanghe, 2010, International journal of Alzheimer's disease.

Alteration of proto-oncogene c-fos expression in neonatal estrogenized BALB/c female mice & murine cervicovaginal tumor LJ6195.

L. Jones, L. Scrocchi, 1991, Endocrinology.

Identification of glucagon-like peptide 1 (GLP-1) actions essential for glucose homeostasis in mice with disruption of GLP-1 receptor signaling.

D. Drucker, P. Brubaker, B. Marshall, 1998, Diabetes.

Modulation of γ-secretase by EVP-0015962 reduces amyloid deposition and behavioral deficits in Tg2576 mice

B. de Strooper, H. Zetterberg, L. Leventhal, 2012, Molecular Neurodegeneration.

Modulation of γ-secretase by EVP-0015962 reduces amyloid deposition and behavioral deficits in Tg2576 mice

B. de Strooper, H. Zetterberg, L. Leventhal, 2012, Molecular Neurodegeneration.

Altered cAMP and Ca2+ signaling in mouse pancreatic islets with glucagon-like peptide-1 receptor null phenotype.

D. Drucker, P. Gilon, F. Schuit, 1999, Diabetes.

Effects of Aging and a High Fat Diet on Body Weight and Glucose Tolerance in Glucagon-Like Peptide-1 Receptor-/- Mice.

D. Drucker, L. Scrocchi, 1998, Endocrinology.

Neuroendocrine Function and Response to Stress in Mice with Complete Disruption of Glucagon-Like Peptide-1 Receptor Signaling*

D. Drucker, S. Asa, F. Vaccarino, 1999 .

D. Drucker, B. Thorens, S. Efrat, 1998, Endocrinology.

Identification of glucagon-like peptide 1 (GLP-1) actions essential for glucose homeostasis in mice with disruption of GLP-1 receptor signaling.

D. Drucker, P. Brubaker, B. Marshall, 1998, Diabetes.