Nicole Weiss

Increased A beta peptides and reduced cholesterol and myelin proteins characterize white matter degeneration in Alzheimer's disease.

Douglas Walker, Thomas Beach, Yu-Min Kuo, 2002, Biochemistry.

The Human Amyloid-β Precursor Protein770 Mutation V717F Generates Peptides Longer Than Amyloid-β-(40-42) and Flocculent Amyloid Aggregates*

B. Ghetti, M. Farlow, C. Esh, 2003, Journal of Biological Chemistry.

APP Transgenic Mice Tg2576 Accumulate Aβ Peptides That Are Distinct from the Chemically Modified and Insoluble Peptides Deposited in Alzheimer's Disease Senile Plaques†

Yu-Min Kuo, Sangram S. Sisodia, M. Staufenbiel, 2002 .

APP transgenic mice Tg2576 accumulate Abeta peptides that are distinct from the chemically modified and insoluble peptides deposited in Alzheimer's disease senile plaques.

M. Staufenbiel, M. Emmerling, W. Kalback, 2002, Biochemistry.

Cortical and Leptomeningeal Cerebrovascular Amyloid and White Matter Pathology in Alzheimer’s Disease

T. Beach, R. Weller, C. Esh, 2003, Molecular medicine.

Increased Aβ Peptides and Reduced Cholesterol and Myelin Proteins Characterize White Matter Degeneration in Alzheimer's Disease†

Yu-Min Kuo, Douglas G. Walker, Thomas G. Beach, 2002 .

The human amyloid-beta precursor protein770 mutation V717F generates peptides longer than amyloid-beta-(40-42) and flocculent amyloid aggregates.

B. Ghetti, M. Farlow, C. Esh, 2004, The Journal of biological chemistry.