N. Hoshi

发表

Loss of AKAP150 perturbs distinct neuronal processes in mice

Fang Zhang, Naoto Hoshi, Jacob Raber, 2008, Proceedings of the National Academy of Sciences.

Sulfhydryl modification inhibits K+ (M) current with kinetics close to acetylcholine in rodent NG108-15 cells

H. Higashida, M. Noda, N. Hoshi, 1997, Neuroscience Research.

M-Current Suppression, Seizures and Lipid Metabolism: A Potential Link Between Neuronal Kv7 Channel Regulation and Dietary Therapies for Epilepsy

N. Hoshi, 2020, Frontiers in Physiology.

A Change in Configuration of the Calmodulin-KCNQ Channel Complex Underlies Ca2+-Dependent Modulation of KCNQ Channel Activity

N. Hoshi, A. Kosenko, 2013, PloS one.

12-Lipoxygenase increases neuronal excitability by inhibiting M-type potassium channels

H. Higashida, Yoshitaka Takahashi, T. Yoshimoto, 2002 .

Channel-anchored Protein Kinase CK2 and Protein Phosphatase 1 Reciprocally Regulate KCNQ2-containing M-channels via Phosphorylation of Calmodulin*

E. Cooper, Seungwoo Kang, N. Hoshi, 2014, The Journal of Biological Chemistry.

Activation of m1 muscarinic acetylcholine receptor induces surface transport of KCNQ channels through a CRMP-2-mediated pathway

Lan Huang, Xuejun Li, N. Hoshi, 2015, Journal of Cell Science.

Ion selectivity of Ba2+ inward current oscillations in ras-transformed fibroblasts that elicit cytoplasmic Ca2+ oscillations by bradykinin.

Y. Nozawa, H. Higashida, M. Noda, 1992, Biochemical and biophysical research communications.

Ba2+ current oscillations modulated by cyclic AMP and phorbol esters in ras-transformed fibroblasts.

Y. Nozawa, H. Higashida, M. Noda, 1992, Biochemical and biophysical research communications.

Ba2+ current oscillation evoked by bradykinin in ras-transformed fibroblasts.

Y. Nozawa, H. Higashida, M. Noda, 1991, Biochemical and biophysical research communications.

Bradykinin B2 receptors and signal transduction analyzed in NG108-15 neuroblastoma x glioma hybrid cells, B2 receptor-transformed CHO cells and ras-transformed NIH/3T3 fibroblasts.

S. Nakashima, Y. Minabe, Y. Nozawa, 1996, Progress in brain research.

Microheterogeneity in heteromultimeric assemblies formed by Shaker (Kv1) and Shaw (Kv3) subfamilies of voltage-gated K+ channels

S. Yokoyama, N. Hoshi, M. Shahidullah, 1995, Proceedings of the Royal Society of London. Series B: Biological Sciences.

Slow inactivation conserved in heteromultimeric voltage‐dependent K+ channels between Shaker (Kv1) and Shaw (Kv3) subfamilies

H. Higashida, S. Yokoyama, N. Hoshi, 1995, FEBS letters.

Inositol trisphosphate/Ca2+ as messengers of bradykinin B2 and muscarinic acetylcholine m1-m4 receptors in neuroblastoma-derived hybrid cells.

D. A. Brown, H. Higashida, S. Yokoyama, 1996, Journal of lipid mediators and cell signalling.

Clinical and electrophysiological characterization of a novel mutation (F193L) in the KCNQ1 gene associated with long QT syndrome.

K. Hayashi, H. Higashida, H. Mabuchi, 2002, Clinical science.

M-current preservation contributes to anticonvulsant effects of valproic acid.

Seungwoo Kang, N. Hoshi, A. Kosenko, 2015, The Journal of clinical investigation.

Activity-Dependent Synaptogenesis: Regulation by a CaM-Kinase Kinase/CaM-Kinase I/βPIX Signaling Complex

T. Soderling, D. Fortin, T. Natsume, 2008, Neuron.

Hydrogen Sulfide Relaxes Human Uterine Artery via Activating Smooth Muscle BKCa Channels

Dong-bao Chen, N. Hoshi, Yi-Hua Yang, 2020, Antioxidants.

Coupling of m2 and m4 muscarinic acetylcholine receptor subtypes to Ca2+-dependent K+ channels in transformed NL308 neuroblastoma x fibroblast hybrid cells

K. Fukuda, S. Yokoyama, M. Noda, 1993, Proceedings of the Royal Society of London. Series B: Biological Sciences.

[Presynaptic and postsynaptic mechanisms of phosphoinositide metabolites].

D. A. Brown, A. Ogura, Y. Nozawa, 1990, Tanpakushitsu kakusan koso. Protein, nucleic acid, enzyme.

Distinct enzyme combinations in AKAP signalling complexes permit functional diversity

L. Langeberg, John D. Scott, N. Hoshi, 2005, Nature Cell Biology.

Cyclic ADP-ribose as a second messenger revisited from a new aspect of signal transduction from receptors to ADP-ribosyl cyclase.

H. Higashida, S. Yokoyama, M. Noda, 2001, Pharmacology & therapeutics.

KCR1, a Membrane Protein That Facilitates Functional Expression of Non-inactivating K+ Currents Associates with Rat EAG Voltage-dependent K+Channels*

Hiroto Takahashi, H. Higashida, N. Hoshi, 1998, The Journal of Biological Chemistry.

Single nucleotide polymorphisms alter kinase anchoring and the subcellular targeting of A-kinase anchoring proteins

Manuel F. Navedo, L. Langeberg, John D. Scott, 2018, Proceedings of the National Academy of Sciences.

Molecular basis of AKAP specificity for PKA regulatory subunits.

C. Carlson, D. Barford, John D. Scott, 2006, Molecular cell.

Modulation of Kv7 channels and excitability in the brain

Naoto Hoshi, N. Hoshi, Derek L Greene, 2016, Cellular and Molecular Life Sciences.

Modulation of voltage-gated K+ channels by the sodium channel β1 subunit

Brian J. Smith, K. Chandy, N. Nukina, 2012, Proceedings of the National Academy of Sciences.

Overexpression of human CD38/ADP-ribosyl cyclase enhances acetylcholine-induced Ca2+ signalling in rodent NG108-15 neuroblastoma cells

S. Takasawa, H. Higashida, S. Yokoyama, 2007, Neuroscience Research.

Streptozotocin, an inducer of NAD+ decrease, attenuates M‐potassium current inhibition by ATP, bradykinin, angiotensin II, endothelin 1 and acetylcholine in NG108‐15 cells

H. Higashida, M. Noda, N. Hoshi, 1996, FEBS letters.

Interaction with AKAP79 modifies the cellular pharmacology of PKC.

A. Newton, L. Langeberg, John D. Scott, 2010, Molecular cell.

Coupling of Muscarinic Receptor Subtypes to Ion Channels: Experiments on Neuroblastoma Hybrid Cells a

D. A. Brown, K. Kameyama, H. Higashida, 1993, Annals of the New York Academy of Sciences.

Subtype‐specific coupling with ADP‐ribosyl cyclase of metabotropic glutamate receptors in retina, cervical superior ganglion and NG108‐15 cells

S. Nakanishi, R. Shigemoto, Y. Fukuda, 2003, Journal of neurochemistry.

Cyclic ADP‐ribose as a potential second messenger for neuronal Ca2+ signaling

H. Higashida, S. Yokoyama, N. Hoshi, 2001, Journal of neurochemistry.

Bradykinin induces inositol 1,4,5‐trisphosphate‐dependent hyperpolarization in K+ M‐current‐deficient hybrid NL308 cells: Comparison with NG108–15 neuroblastoma × glioma hybrid cells

Y. Nozawa, Y. Okano, H. Higashida, 1990, Glia.

In Vivo Attenuation of M-Current Suppression Impairs Consolidation of Object Recognition Memory

Anastasia Kosenko, Shirin Moftakhar, Marcelo A Wood, 2020, The Journal of Neuroscience.

Signal Transduction from Bradykinin, Angiotensin, Adrenergic and Muscarinic Receptors to Effector Enzymes, Including ADP-Ribosyl Cyclase

Hiroto Takahashi, H. Higashida, S. Yokoyama, 2001, Biological chemistry.

Dynamic regulation of cAMP synthesis through anchored PKA-adenylyl cyclase V/VI complexes.

Naoto Hoshi, D. Cooper, L. Langeberg, 2006, Molecular cell.

AKAP150 signaling complex promotes suppression of the M-current by muscarinic agonists

L. Langeberg, John D. Scott, H. Higashida, 2003, Nature Neuroscience.

XE991 and Linopirdine Are State-Dependent Inhibitors for Kv7/KCNQ Channels that Favor Activated Single Subunits

Seungwoo Kang, N. Hoshi, Derek L. Greene, 2017, The Journal of Pharmacology and Experimental Therapeutics.

Attenuating M‐current suppression in vivo by a mutant Kcnq2 gene knock‐in reduces seizure burden and prevents status epilepticus–induced neuronal death and epileptogenesis

N. Hoshi, A. Kosenko, Derek L. Greene, 2018, Epilepsia.

Coordinated signal integration at the M‐type potassium channel upon muscarinic stimulation

B. Reinhart, D. Bartel, M. Jones-Rhoades, 2012, The EMBO journal.

12‐Lipoxygenase overexpression in rodent NG108‐15 cells enhances membrane excitability by inhibiting M‐type K+ channels

H. Higashida, Yoshitaka Takahashi, T. Yoshimoto, 1999, The Journal of physiology.

Delineation of Type I Protein Kinase A-selective Signaling Events Using an RI Anchoring Disruptor*

C. Carlson, John D. Scott, K. Taskén, 2006, Journal of Biological Chemistry.

Bioinformatic design of A-kinase anchoring protein-in silico: A potent and selective peptide antagonist of type II protein kinase A anchoring

Naoto Hoshi, L. Langeberg, John D. Scott, 2003, Proceedings of the National Academy of Sciences of the United States of America.

Potassium Channels Cloned from NG108‐15 Neuroblastoma‐Glioma Hybrid Cells

H. Higashida, S. Yokoyama, N. Hoshi, 1993, Annals of the New York Academy of Sciences.

Protein kinase C bound with A-kinase anchoring protein is involved in muscarinic receptor-activated modulation of M-type KCNQ potassium channels

H. Higashida, S. Yokoyama, M. Noda, 2005, Neuroscience Research.

Endomorphins inhibit high‐threshold Ca2+ channel currents in rodent NG108‐15 cells overexpressing μ‐opioid receptors

A. Kastin, H. Higashida, J. Zadina, 1998, The Journal of physiology.

Compound heterozygosity for mutations Asp611-->Tyr in KCNQ1 and Asp609-->Gly in KCNH2 associated with severe long QT syndrome.

K. Hayashi, H. Higashida, H. Mabuchi, 2005, Clinical science.

Probucol aggravates long QT syndrome associated with a novel missense mutation M124T in the N-terminus of HERG.

K. Hayashi, H. Higashida, H. Mabuchi, 2004, Clinical science.

Adrenergic Regulation of HCN4 Channel Requires Protein Association with β2-Adrenergic Receptor*

Seungwoo Kang, N. Hoshi, A. Kosenko, 2012, The Journal of Biological Chemistry.

ATP Competitive Protein Kinase C Inhibitors Demonstrate Distinct State-Dependent Inhibition

N. Hoshi, Ida M. Smith, 2011, PloS one.

Activation of m1 muscarinic acetylcholine receptor induces surface transport of KCNQ channels through a CRMP-2-mediated pathway

Lan Huang, Xuejun Li, N. Hoshi, 2015, Journal of Cell Science.

Coordinated signal integration at the M‐type potassium channel upon muscarinic stimulation

L. Langeberg, Seungwoo Kang, N. Hoshi, 2012, The EMBO journal.

Characterization of a novel missense mutation E637K in the pore-S6 loop of HERG in a patient with long QT syndrome.

K. Hayashi, H. Higashida, H. Mabuchi, 2002, Cardiovascular research.

Whole-cell analysis of NGK2 (mKv3.1a) K+ channels stably expressed in mouse fibroblast cells

H. Higashida, S. Yokoyama, N. Hoshi, 1995, Neuroscience Letters.

Muscarinic acetylcholine m1–m4 receptors overexpressed in neuroblastoma-glioma hybrid NG108-15 cells as a cholinergic model

H. Higashida, S. Yokoyama, N. Hoshi, 1998, Journal of Physiology-Paris.