K. Hochrainer

C. Iadecola, J. Anrather, Josef Anrather, 2015, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

Elevated post-ischemic ubiquitination results from suppression of deubiquitinase activity and not proteasome inhibition

C. Iadecola, P. Zhou, T. Kahles, 2020, Cellular and Molecular Life Sciences.

Monoubiquitination of nuclear RelA negatively regulates NF-κB activity independent of proteasomal degradation

C. Iadecola, J. Anrather, Gianfranco Racchumi, 2012, Cellular and Molecular Life Sciences.

Hypo‐phosphorylation leads to nuclear retention of NF‐κB p65 due to impaired IκBα gene synthesis

J. Anrather, Gianfranco Racchumi, K. Hochrainer, 2007 .

Cerebral ischemia induces the aggregation of proteins linked to neurodegenerative diseases

Shenmin Zhang, C. Iadecola, S. Zhang, 2018, Scientific Reports.

Reperfusion Rather than Ischemia Drives the Formation of Ubiquitin Aggregates After Middle Cerebral Artery Occlusion

C. Iadecola, J. Anrather, K. Jackman, 2012, Stroke.

Dietary salt promotes cognitive impairment through tau phosphorylation

Hong Jiang, D. Holtzman, C. Iadecola, 2018, Nature.

The human HERC family of ubiquitin ligases: novel members, genomic organization, expression profiling, and evolutionary aspects.

B. Binder, U. Baranyi, H. Mayer, 2005, Genomics.

Site-specific Phosphorylation of the p65 Protein Subunit Mediates Selective Gene Expression by Differential NF-κB and RNA Polymerase II Promoter Recruitment*

J. Anrather, Gianfranco Racchumi, K. Hochrainer, 2012, The Journal of Biological Chemistry.

Regulation of Nuclear Factor κB (NF-κB) Transcriptional Activity via p65 Acetylation by the Chaperonin Containing TCP1 (CCT)

J. Anrather, M. Soares, K. Hochrainer, 2012, PloS one.

Tau induces PSD95-nNOS uncoupling and neurovascular dysfunction independent of neurodegeneration

C. Iadecola, D. Eliezer, J. Anrather, 2020, Nature Neuroscience.

Phosphatidylethanolamine critically supports internalization of cell-penetrating protein C inhibitor

P. Schwille, E. Bielek, J. Huntington, 2007, The Journal of cell biology.

Lipoprotein Receptor–Related Protein-6 Protects the Brain From Ischemic Injury

M. Ross, C. Iadecola, J. Anrather, 2013, Stroke.

Nuclear Factor-κB Activation and Postischemic Inflammation Are Suppressed in CD36-Null Mice after Middle Cerebral Artery Occlusion

C. Iadecola, J. Anrather, Gianfranco Racchumi, 2008, The Journal of Neuroscience.

Cerebral ischemia induces the aggregation of proteins linked to neurodegenerative diseases

C. Iadecola, S. Zhang, K. Jackman, 2018, Scientific Reports.

Elevated post-ischemic ubiquitination results from suppression of deubiquitinase activity and not proteasome inhibition

C. Iadecola, P. Zhou, T. Kahles, 2020, Cellular and Molecular Life Sciences.

Post-ischemic ubiquitination at the postsynaptic density reversibly influences the activity of ischemia-relevant kinases

K. Hochrainer, Luvna Dhawka, Victoria Palfini, 2023, bioRxiv.

Protein Modifications with Ubiquitin as Response to Cerebral Ischemia-Reperfusion Injury

K. Hochrainer, Karin Hochrainer, 2017, Translational Stroke Research.

Reperfusion Rather than Ischemia Drives the Formation of Ubiquitin Aggregates After Middle Cerebral Artery Occlusion

C. Iadecola, J. Anrather, K. Hochrainer, 2012, Stroke.