V. Lawson

发表

Conservation of a Glycine-rich Region in the Prion Protein Is Required for Uptake of Prion Infectivity*

Andrew F Hill, Colin L Masters, Roberto Cappai, 2010, The Journal of Biological Chemistry.

Markers of A1 astrocytes stratify to molecular sub-types in sporadic Creutzfeldt–Jakob disease brain

A. Hill, C. Mclean, V. Lewis, 2020, Brain communications.

Flexible N-terminal Region of Prion Protein Influences Conformation of Protease-resistant Prion Protein Isoforms Associated with Cross-species Scrapie Infection in Vivo and in Vitro*

B. Chesebro, S. Priola, K. Meade-White, 2004, Journal of Biological Chemistry.

Glycosylation influences cross‐species formation of protease‐resistant prion protein

S. Priola, V. Lawson, 2001, The EMBO journal.

Evidence for prion protein expression in enteroglial cells of the myenteric plexus of mouse intestine

Giovanni Di Guardo, A. Hill, R. Cappai, 2008, Autonomic Neuroscience.

Enzymatic detergent treatment protocol that reduces protease-resistant prion protein load and infectivity from surgical-steel monofilaments contaminated with a human-derived prion strain.

C. Masters, V. Lawson, James D. Stewart, 2007, The Journal of general virology.

Acute exposure to prion infection induces transient oxidative stress progressing to be cumulatively deleterious with chronic propagation in vitro.

C. Masters, C. Haigh, A. McGlade, 2011, Free radical biology & medicine.

Correlative studies support lipid peroxidation is linked to PrPres propagation as an early primary pathogenic event in prion disease

C. Masters, A. Hill, R. Cappai, 2006, Brain Research Bulletin.

Gene Knockout of tau Expression Does Not Contribute to the Pathogenesis of Prion Disease

C. Masters, R. Cappai, G. Ciccotosto, 2011, Journal of neuropathology and experimental neurology.

Near-infrared fluorescence imaging of apoptotic neuronal cell death in a live animal model of prion disease.

C. Masters, C. Haigh, B. Roberts, 2010, ACS chemical neuroscience.

Extended period of asymptomatic prion disease after low dose inoculation: Assessment of detection methods and implications for infection control

C. Masters, A. Hill, G. Klug, 2005, Neurobiology of Disease.

PrPSc Oligomerization Appears Dynamic, Quickly Engendering Inherent M1000 Acute Synaptotoxicity.

Blaine R. Roberts, P. Adlard, D. Finkelstein, 2020, Biophysical journal.

PrPC-related signal transduction is influenced by copper, membrane integrity and the alpha cleavage site

C. Masters, A. Hill, C. Haigh, 2009, Cell Research.

Mouse-adapted sporadic human Creutzfeldt-Jakob disease prions propagate in cell culture.

C. Masters, A. Hill, R. Cappai, 2008, The international journal of biochemistry & cell biology.

Preparation and Immunostaining of the Myenteric Plexus of Prion-Infected Mice.

V. Lawson, L. Ellett, 2017, Methods in molecular biology.

Quantitative bioassay of surface-bound prion infectivity.

V. Lawson, 2008, Methods in molecular biology.

The brain to gut pathway: a possible route of prion transmission

A. Hill, J. Furness, V. Lawson, 2010, Gut.

Apoptosis induced in synchronized human immunodeficiency virus type 1-infected primary peripheral blood mononuclear cells is detected after the peak of CD4+ T-lymphocyte loss and is dependent on the tropism of the gp120 envelope glycoprotein.

D. Purcell, A. Greenway, D. McPhee, 2004, Virology.

Blood vessel cell death during prion disease: implications for disease management and infection control.

C. Haigh, V. Lawson, S. Drew, 2014, Experimental hematology.

Rapid Full-Length Genomic Sequencing of Two Cytopathically Heterogeneous Australian Primary HIV-1 Isolates

D. McPhee, N. Deacon, R. Oelrichs, 2000, Journal of Biomedical Science.

Rapid full-length genomic sequencing of two cytopathically heterogeneous Australian primary HIV-1 isolates.

D. McPhee, N. Deacon, R. Oelrichs, 2000, Journal of biomedical science.

Early existence and biochemical evolution characterise acutely synaptotoxic PrPSc

Blaine R. Roberts, P. Adlard, D. Finkelstein, 2019, PLoS pathogens.

N-terminal Truncation of Prion Protein Affects Both Formation and Conformation of Abnormal Protease-resistant Prion Protein Generatedin Vitro *

B. Chesebro, S. Priola, V. Lawson, 2001, The Journal of Biological Chemistry.

Dominant roles of the polybasic proline motif and copper in the PrP23-89-mediated stress protection response

C. Masters, C. Haigh, Martin P Boland, 2009, Journal of Cell Science.

Increased Proportions of C1 Truncated Prion Protein Protect Against Cellular M1000 Prion Infection

C. Masters, A. Hill, C. Haigh, 2009, Journal of neuropathology and experimental neurology.

Transmissible spongiform encephalopathies

C. Masters, S. Collins, V. Lawson, 2004, The Lancet.

Adaptive changes after human immunodeficiency virus type 1 transmission.

D. Cooper, D. McPhee, A. Imrie, 2002, AIDS research and human retroviruses.

Pathogenic mechanisms of prion protein, amyloid‐β and α‐synuclein misfolding: the prion concept and neurotoxicity of protein oligomers

A. Hill, D. Finkelstein, V. Lawson, 2016, Journal of neurochemistry.

The role of lipids in α-synuclein misfolding and neurotoxicity

A. Hill, D. Finkelstein, V. Lawson, 2019, The Journal of Biological Chemistry.

Restoration of intestinal function in an MPTP model of Parkinson’s Disease

J. Culvenor, J. Furness, D. Finkelstein, 2016, Scientific Reports.

Genomic Structure of an Attenuated Quasi Species of HIV-1 from a Blood Transfusion Donor and Recipients

J. S. Sullivan, A. Tsykin, J. Mills, 1995, Science.

An intact membrane is essential for small extracellular vesicle‐induced modulation of α‐synuclein fibrillization

Shane E. Gordon, A. Hill, M. Perugini, 2020, Journal of extracellular vesicles.

The prion protein regulates beta-amyloid-mediated self-renewal of neural stem cells in vitro

Qiao-Xin Li, C. Haigh, T. Ryan, 2015, Stem Cell Research & Therapy.

Viral Phenotypes and Antibody Responses in Long-Term Survivors Infected with Attenuated Human Immunodeficiency Virus Type 1 Containing Deletions in the nef and Long Terminal Repeat Regions

M. Churchill, S. Wesselingh, D. McPhee, 2007, Journal of Virology.

Prion Infection Impairs Cholesterol Metabolism in Neuronal Cells*

P. Meikle, M. Bukrinsky, A. Hill, 2013, The Journal of Biological Chemistry.

Distribution of microRNA profiles in pre-clinical and clinical forms of murine and human prion disease

Lesley Cheng, A. Hill, I. Zerr, 2021, Communications biology.

Prion subcellular fractionation reveals infectivity spectrum, with a high titre-low PrPres level disparity

C. Masters, A. Hill, C. Haigh, 2012, Molecular Neurodegeneration.

Prion acute synaptotoxicity is largely driven by protease-resistant PrPSc species

Blaine R. Roberts, P. Adlard, D. Finkelstein, 2018, PLoS pathogens.

Pathogenic Mutations within the Hydrophobic Domain of the Prion Protein Lead to the Formation of Protease-Sensitive Prion Species with Increased Lethality

C. Masters, A. Hill, Christopher F. Harrison, 2013, Journal of Virology.

Strain variation in treatment and prevention of human prion diseases.

V. Lawson, L. Ellett, Zoe T Revill, 2020, Progress in molecular biology and translational science.

Glycosaminoglycan sulfation determines the biochemical properties of prion protein aggregates.

C. Masters, A. Hill, Mitch Shambrook, 2015, Glycobiology.

Residues Surrounding the Glycosylphosphatidylinositol Anchor Attachment Site of PrP Modulate Prion Infection: Insight from the Resistance of Rabbits to Prion Disease

C. Masters, A. Hill, R. Cappai, 2010, Journal of Virology.

Prion protein glycosylation

A. Hill, S. Collins, V. Lawson, 2005, Journal of neurochemistry.

Distribution of microRNA profiles in pre-clinical and clinical forms of murine and human prion disease

Lesley Cheng, A. Hill, I. Zerr, 2021, Communications Biology.

Misfolded α-synuclein causes hyperactive respiration without functional deficit in live neuroblastoma cells

Shane E. Gordon, A. Hill, P. Fisher, 2019, Disease Models & Mechanisms.

Prion‐infected cells regulate the release of exosomes with distinct ultrastructural features

A. Hill, E. Hanssen, V. Lawson, 2012, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Optical Imaging Detects Apoptosis in the Brain and Peripheral Organs of Prion-Infected Mice

C. Masters, C. Haigh, K. Barnham, 2011, Journal of neuropathology and experimental neurology.

The Prion Protein Preference of Sporadic Creutzfeldt-Jakob Disease Subtypes*

C. Masters, A. Hill, G. Klug, 2012, The Journal of Biological Chemistry.

Glycosaminoglycan Sulphation Affects the Seeded Misfolding of a Mutant Prion Protein

C. Masters, A. Hill, D. Hoke, 2010, PloS one.

Chronic isolation stress is associated with increased colonic and motor symptoms in the A53T mouse model of Parkinson’s disease

J. Furness, D. Finkelstein, M. Ringuet, 2019, Neurogastroenterology and motility : the official journal of the European Gastrointestinal Motility Society.

ATH434 Reverses Colorectal Dysfunction in the A53T Mouse Model of Parkinson’s Disease

R. Cherny, P. Adlard, J. Furness, 2021, Journal of Parkinson's disease.

Visualisation of phenotypically mixed HIV-1 and HTLV-I virus particles by electron microscopy.

D. McPhee, V. Lawson, J. Doultree, 2000, Journal of biomedical science.

Visualisation of Phenotypically Mixed HIV-1 and HTLV-I Virus Particles by Electron Microscopy

D. McPhee, V. Lawson, J. Doultree, 2000, Journal of Biomedical Science.

In Vivo-Near Infrared Imaging of Neurodegeneration.

C. Haigh, V. Lawson, S. Drew, 2017, Methods in molecular biology.

Increased Proportions of C1 Truncated Prion Protein Protect Against Cellular M1000 Prion Infection

C. Masters, A. Hill, C. Haigh, 2009, Journal of neuropathology and experimental neurology.

Pathogenic Mutations within the Hydrophobic Domain of the Prion Protein Lead to the Formation of Protease-Sensitive Prion Species with Increased Lethality

C. Masters, A. Hill, Christopher F. Harrison, 2013, Journal of Virology.