Naoko Minematsu-Ikeguchi

Atg8L/Apg8L is the fourth mammalian modifier of mammalian Atg8 conjugation mediated by human Atg4B, Atg7 and Atg3

Yu-shin Sou, T. Ueno, E. Kominami, 2006, The FEBS journal.

Lysosomal Turnover, but Not a Cellular Level, of Endogenous LC3 is a Marker for Autophagy

T. Ueno, E. Kominami, I. Tanida, 2005, Autophagy.

Lysosomal Turnover of GABARAP-Phospholipid Conjugate is Activated During Differentiation of C2C12 Cells to Myotubes without Inactivation of the mTor Kinase-Signaling Pathway

M. Hirata, Yu-shin Sou, T. Ueno, 2006, Autophagy.

Characterization of CAA0225, a novel inhibitor specific for cathepsin L, as a probe for autophagic proteolysis.

T. Ueno, E. Kominami, I. Tanida, 2009, Biological & pharmaceutical bulletin.

HsAtg4B/HsApg4B/Autophagin-1 Cleaves the Carboxyl Termini of Three Human Atg8 Homologues and Delipidates Microtubule-associated Protein Light Chain 3- and GABAA Receptor-associated Protein-Phospholipid Conjugates*

T. Ueno, J. Ezaki, E. Kominami, 2004, Journal of Biological Chemistry.

The Mouse APG10 Homologue, an E2-like Enzyme for Apg12p Conjugation, Facilitates MAP-LC3 Modification*

T. Nemoto, T. Ueno, E. Kominami, 2003, Journal of Biological Chemistry.