Y. Kishimoto

发表

Hippocampal CA3 NMDA Receptors Are Crucial for Adaptive Timing of Trace Eyeblink Conditioned Response

S. Tonegawa, K. Nakazawa, M. Kano, 2006, The Journal of Neuroscience.

A Role for Myosin Va in Cerebellar Plasticity and Motor Learning: A Possible Mechanism Underlying Neurological Disorder in Myosin Va Disease

Yasushi Kishimoto, Mariko Miyata, Masanobu Kano, 2011, The Journal of Neuroscience.

Traumatic brain injury accelerates amyloid-β deposition and impairs spatial learning in the triple-transgenic mouse model of Alzheimer’s disease

T. Tamiya, N. Kawai, M. Ueno, 2016, Neuroscience Letters.

mGluR5 Is Substitutable for mGluR1 in Cerebellar Purkinje Cells for Motor Coordination, Developmental Synapse Elimination, and Motor Learning

M. Kano, A. Aiba, K. Nakao, 2022, Cells.

Phospholipase Cβ4 and Protein Kinase Cα and/or Protein Kinase CβI Are Involved in the Induction of Long Term Depression in Cerebellar Purkinje Cells*

S. Kawahara, Y. Kirino, M. Katsuki, 2001, The Journal of Biological Chemistry.

The Synaptic Targeting of mGluR1 by Its Carboxyl-Terminal Domain Is Crucial for Cerebellar Function

Masahiko Watanabe, M. Kano, T. Tabata, 2014, The Journal of Neuroscience.

Impairment of eyeblink conditioning in GluRδ2‐mutant mice depends on the temporal overlap between conditioned and unconditioned stimuli

S. Kawahara, Y. Kirino, M. Mishina, 2001, The European journal of neuroscience.

Impairment of cerebellar long-term depression and GABAergic transmission in prion protein deficient mice ectopically expressing PrPLP/Dpl

Y. Kirino, T. Yoshioka, S. Katamine, 2020, Scientific Reports.

Age-Dependent Impairment of Eyeblink Conditioning in Prion Protein-Deficient Mice

Yasushi Kishimoto, Yutaka Kirino, Tohru Yoshioka, 2013, PloS one.

Classical eyeblink conditioning in glutamate receptor subunit δ2 mutant mice is impaired in the delay paradigm but not in the trace paradigm

S Kawahara, Y Kirino, S. Kawahara, 2001, The European journal of neuroscience.

MDGA1-deficiency attenuates prepulse inhibition with alterations of dopamine and serotonin metabolism: An ex vivo HPLC-ECD analysis

T. Kubota, Y. Kishimoto, Tohru Yamamoto, 2019, Neuroscience Letters.

Data on amyloid precursor protein accumulation, spontaneous physical activity, and motor learning after traumatic brain injury in the triple-transgenic mouse model of Alzheimer׳s disease

Y. Kirino, T. Tamiya, N. Kawai, 2016, Data in brief.

Functional maintenance of calcium store by ShcB adaptor protein in cerebellar Purkinje cells

Masahiko Watanabe, R. Sakai, Shinichiro Yamamoto, 2020, Scientific Reports.

Conditioned eyeblink learning is formed and stored without cerebellar granule cell transmission

Yasushi Kishimoto, Masanobu Kano, Dai Watanabe, 2007, Proceedings of the National Academy of Sciences.

Endogenous Cannabinoid Signaling through the CB1 Receptor Is Essential for Cerebellum-Dependent Discrete Motor Learning

M. Kano, Y. Kishimoto, 2006, The Journal of Neuroscience.

mGluR1 in cerebellar Purkinje cells is essential for the formation but not expression of associative eyeblink memory

Masahiko Watanabe, M. Kano, K. Kitamura, 2019, Scientific Reports.

Dataset of eyeblink conditioning in mice treated with the selective mGluR1 antagonist JNJ16259685

Y. Kishimoto, Shoichi Tohyama, 2023, Data in brief.

Implicit Memory in Monkeys: Development of a Delay Eyeblink Conditioning System with Parallel Electromyographic and High-Speed Video Measurements

Haruyoshi Toyoda, Yasushi Kishimoto, Hideo Tsukada, 2015, PloS one.

Involvement of protein‐tyrosine phosphatase PTPMEG in motor learning and cerebellar long‐term depression

M. Kano, M. Iino, T. Manabe, 2007, The European journal of neuroscience.

mGluR1 in cerebellar Purkinje cells is required for normal association of temporally contiguous stimuli in classical conditioning

M. Kano, S. Kawahara, Y. Kirino, 2002, The European journal of neuroscience.

Impaired delay but normal trace eyeblink conditioning in PLCβ4 mutant mice

S. Kawahara, Y. Kirino, M. Katsuki, 2001, Neuroreport.

Long‐trace interval eyeblink conditioning is impaired in mutant mice lacking the NMDA receptor subunit ε1

S. Kawahara, Y. Kirino, M. Mishina, 2001 .

SHORT COMMUNICATION Classical eyeblink conditioning in glutamate receptor subunit d2 mutant mice is impaired in the delay paradigm but not in the trace paradigm

S. Kawahara, Y. Kirino, M. Mishina, 2001 .

Endocannabinoid Signaling from 2-Arachidonoylglycerol to CB1 Cannabinoid Receptor Facilitates Reward-based Learning of Motor Sequence

M. Kano, K. Sakimura, T. Ohno‐Shosaku, 2019, Neuroscience.

Effects of the noncompetitive NMDA receptor antagonist MK-801 on classical eyeblink conditioning in mice

S. Kawahara, Y. Kirino, K. Takehara, 2001, Neuropharmacology.

Altered Cortical Dynamics and Cognitive Function upon Haploinsufficiency of the Autism-Linked Excitatory Synaptic Suppressor MDGA2

Yasushi Kishimoto, Yutaka Kirino, Ann Marie Craig, 2016, Neuron.

Early Contextual Fear Memory Deficits in a Double-Transgenic Amyloid-β Precursor Protein/Presenilin 2 Mouse Model of Alzheimer's Disease

Y. Kishimoto, M. Nagai, Kai Fukumoto, 2017, International journal of Alzheimer's disease.

Conditioned eyeblink response is impaired in mutant mice lacking NMDA receptor subunit NR2A

S Kawahara, Y Kirino, S. Kawahara, 1997, Neuroreport.

Task-specific enhancement of hippocampus-dependent learning in mice deficient in monoacylglycerol lipase, the major hydrolyzing enzyme of the endocannabinoid 2-arachidonoylglycerol

M. Kano, Y. Kirino, K. Sakimura, 2015, Front. Behav. Neurosci..

Inhibition of MAO-A and stimulation of behavioural activities in mice by the inactive prodrug form of the anti-influenza agent oseltamivir

Y. Kirino, M. Kanai, M. Hiasa, 2013, British journal of pharmacology.

mGluR1 in cerebellar Purkinje cells is essential for the formation but not expression of associative eyeblink memory

Masahiko Watanabe, M. Kano, K. Kitamura, 2019, Scientific Reports.

Loss of Synapse Repressor MDGA1 Enhances Perisomatic Inhibition, Confers Resistance to Network Excitation, and Impairs Cognitive Function.

Steven A. Connor, Y. T. Wang, A. Craig, 2017, Cell reports.

Junctophilin‐mediated channel crosstalk essential for cerebellar synaptic plasticity

Yasushi Kishimoto, Masahiko Watanabe, Masanobu Kano, 2007, The EMBO journal.

Visually cued fear conditioning test for memory impairment related to cortical function

M. Yamada, Kazuya Kuboyama, Y. Kishimoto, 2020, Neuropsychopharmacology reports.

Age-dependent impairment of delay and trace eyeblink conditioning in mice

S. Kawahara, Y. Kirino, Y. Kishimoto, 2001, Neuroreport.

Traumatic Brain Injury by Weight-Drop Method Causes Transient Amyloid-β Deposition and Acute Cognitive Deficits in Mice

Y. Kirino, T. Tamiya, N. Kawai, 2019, Behavioural neurology.

Early impairment in a water-finding test in a longitudinal study of the Tg2576 mouse model of Alzheimer's disease

Y. Kirino, Y. Kishimoto, Erina Higashihara, 2013, Brain Research.

Chemical equilibrium model comprising calcaneus bone mineral density, low-density lipoprotein cholesterol, and physical work capacity in premenopausal women

N. Ishii, Y. Kishimoto, Kazuto Mitsuhashi, 2022, F1000Research.

Presenilin 2 mutation accelerates the onset of impairment in trace eyeblink conditioning in a mouse model of Alzheimer's disease overexpressing human mutant amyloid precursor protein

Y. Kirino, Y. Kishimoto, 2013, Neuroscience Letters.

Experimental Verification of Classical Conditioning Models by Inducible On/Off Control of Gene Expression

Y. Kishimoto, 2010 .

Long-trace interval eyeblink conditioning is impaired in mutant mice lacking the NMDA receptor subunit epsilon 1.

S. Kawahara, Y. Kirino, M. Mishina, 2001, The European journal of neuroscience.

Impaired long-trace eyeblink conditioning in a Tg2576 mouse model of Alzheimer's disease

Y. Kirino, Y. Kishimoto, Ikuko Oku, 2012, Neuroscience Letters.

Phospholipase Cbeta4 and protein kinase Calpha and/or protein kinase CbetaI are involved in the induction of long term depression in cerebellar Purkinje cells.

S. Kawahara, Y. Kirino, M. Katsuki, 2001, The Journal of biological chemistry.

Spatial and latent memory data in PS2Tg2576 alzheimer's disease mouse model after memantine treatment

T. Kubota, Y. Kishimoto, Kana Sato, 2021, Data in brief.

Loss of Synapse Repressor MDGA1 Enhances Perisomatic Inhibition, Confers Resistance to Network Excitation, and Impairs Cognitive Function.

Steven A. Connor, Y. T. Wang, A. Craig, 2017, Cell reports.