K Gerdes

Antisense RNA-regulated programmed cell death.

A. Gultyaev, K. Gerdes, T. Franch, 1997, Annual review of genetics.

Mechanism of killer gene activation. Antisense RNA-dependent RNase III cleavage ensures rapid turn-over of the stable hok, srnB and pndA effector messenger RNAs.

E. Wagner, K. Gerdes, A. Nielsen, 1992, Journal of molecular biology.

Metastable structures and refolding kinetics in hok mRNA of plasmid R1.

C. Pleij, A. Gultyaev, K. Gerdes, 1999, RNA.

Programmed cell death in bacteria: proteic plasmid stabilization systems

R. B. Jensen, K. Gerdes, K Gerdes, 1995, Molecular microbiology.

RelE, a global inhibitor of translation, is activated during nutritional stress

K. Gerdes, K Gerdes, K Pedersen, 2001, Proceedings of the National Academy of Sciences of the United States of America.

Unique type of plasmid maintenance function: postsegregational killing of plasmid-free cells.

S. Molin, K. Gerdes, S Molin, 1986, Proceedings of the National Academy of Sciences of the United States of America.

Antisense RNA regulation in prokaryotes: rapid RNA/RNA interaction facilitated by a general U-turn loop structure.

E. Wagner, K. Gerdes, T. Franch, 1999, Journal of molecular biology.

Combining the hok/sok, parDE, and pnd postsegregational killer loci to enhance plasmid stability

T. Wood, K. Wu, C. Kim, 1997, Applied and environmental microbiology.

Plasmid and chromosome segregation in prokaryotes.

R. B. Jensen, K. Gerdes, J. Møller-Jensen, 2000, Trends in microbiology.

Programmed cell death by hok/sok of plasmid R1: processing at the hok mRNA 3'-end triggers structural rearrangements that allow translation and antisense RNA binding.

A. Gultyaev, K. Gerdes, T. Franch, 1997, Journal of molecular biology.