E. Avdievich

发表

MutS homolog 4 localization to meiotic chromosomes is required for chromosome pairing during meiosis in male and female mice.

R Kucherlapati, R. Kucherlapati, J. Pollard, 2000, Genes & development.

EXO1 suppresses double-strand break induced homologous recombination between diverged sequences in mammalian cells.

W. Edelmann, M. Jasin, E. Avdievich, 2017, DNA repair.

The DNA mismatch repair genes Msh3 and Msh6 cooperate in intestinal tumor suppression.

R. Kucherlapati, T. Kunkel, M. Kucherlapati, 2000, Cancer research.

Advances in Brief The DNA Mismatch Repair Genes Msh 3 and Msh 6 Cooperate in Intestinal Tumor Suppression 1

R. Kucherlapati, T. Kunkel, M. Kucherlapati, 2000 .

Mbd4 inactivation increases Cright-arrowT transition mutations and promotes gastrointestinal tumor formation.

W. Edelmann, M. Fazzari, Kan Yang, 2002, Proceedings of the National Academy of Sciences of the United States of America.

Apoptotic function of human PMS2 compromised by the nonsynonymous single-nucleotide polymorphic variant R20Q

R. Kolodner, W. Edelmann, H. Shimodaira, 2008, Proceedings of the National Academy of Sciences.

Genome-wide recombination map construction from single individuals using linked-read sequencing

Felicity C. Jones, Vrinda Venu, F. Jones, 2018, Nature Communications.

Mammalian Exo1 encodes both structural and catalytic functions that play distinct roles in essential biological processes

T. Kunkel, W. Edelmann, R. Sellers, 2013, Proceedings of the National Academy of Sciences.

Ubiquitylated PCNA plays a role in somatic hypermutation and class-switch recombination and is required for meiotic progression

A. Bergman, P. Cohen, W. Edelmann, 2008, Proceedings of the National Academy of Sciences.

The ATPase activity of MLH1 is required to orchestrate DNA double-strand breaks and end processing during class switch recombination

W. Edelmann, R. Chahwan, E. Avdievich, 2012, The Journal of experimental medicine.

The ATPase activity of MLH 1 is required to orchestrate DNA double-strand breaks and end processing during class switch recombination

W. Edelmann, R. Chahwan, E. Avdievich, 2012 .

Comparative Analysis of Meiotic Progression in Female Mice Bearing Mutations in Genes of the DNA Mismatch Repair Pathway1

P. Cohen, W. Edelmann, N. Kolas, 2008, Biology of reproduction.

An Msh2 Point Mutation Uncouples DNA Mismatch Repair and Apoptosis

R. Kucherlapati, T. Kunkel, W. Edelmann, 2004, Cancer Research.

The distinct spectra of tumor-associated Apc mutations in mismatch repair-deficient Apc1638N mice define the roles of MSH3 and MSH6 in DNA repair and intestinal tumorigenesis.

R. Kucherlapati, R. Kolodner, A. Brown, 2001, Cancer research.

Distinct effects of the recurrent Mlh1G67R mutation on MMR functions, cancer, and meiosis

A. Rosenwald, R. Kucherlapati, P. Cohen, 2008, Proceedings of the National Academy of Sciences.

Mbd4 inactivation increases C→T transition mutations and promotes gastrointestinal tumor formation

W. Edelmann, M. Fazzari, Kan Yang, 2002, Proceedings of the National Academy of Sciences of the United States of America.

Paradoxical enhancement of atherosclerosis by probucol treatment in apolipoprotein E-deficient mice.

N. Maeda, R. Reddick, S. H. Zhang, 1997, The Journal of clinical investigation.

Functional consequences of DNA mismatch repair missense mutations in murine models and their impact on cancer predisposition.

W. Edelmann, S. Scherer, E. Avdievich, 2005, Biochemical Society transactions.

An Msh2 Point Mutation Uncouples DNA Mismatch Repair and Apoptosis

R. Kucherlapati, T. Kunkel, W. Edelmann, 2004, Cancer Research.