Resham Chhabra

Emerging Use of Nanotechnology in the Treatment of Neurological Disorders.

G. Tosi, A. Grabrucker, Resham Chhabra, 2015, Current pharmaceutical design.

Nanoparticles as Blood-Brain Barrier Permeable CNS Targeted Drug Delivery Systems

B. Ruozi, G. Tosi, F. Forni, 2013 .

Characterization of lysosome-destabilizing DOPE/PLGA nanoparticles designed for cytoplasmic drug release.

T. Boeckers, B. Ruozi, G. Tosi, 2014, International journal of pharmaceutics.

Insight on the fate of CNS-targeted nanoparticles. Part II: Intercellular neuronal cell-to-cell transport.

Michele Zoli, Barbara Ruozi, Maria Angela Vandelli, 2014, Journal of controlled release : official journal of the Controlled Release Society.

Loss of COMMD1 and copper overload disrupt zinc homeostasis and influence an autism-associated pathway at glutamatergic synapses

T. Boeckers, A. Grabrucker, Resham Chhabra, 2014, BioMetals.

PTBP1 and PTBP2 Repress Nonconserved Cryptic Exons.

P. Wong, S. Wheelan, J. Corden, 2016, Cell reports.

Zinc deficiency dysregulates the synaptic ProSAP/Shank scaffold and might contribute to autism spectrum disorders.

T. Boeckers, M. Kreutz, M. Schmeisser, 2014, Brain : a journal of neurology.

The PSD protein ProSAP2/Shank3 displays synapto-nuclear shuttling which is deregulated in a schizophrenia-associated mutation

T. Boeckers, M. Schmeisser, A. Grabrucker, 2014, Experimental Neurology.

Activity and circadian rhythm influence synaptic Shank3 protein levels in mice

M. Zoli, A. Grabrucker, Resham Chhabra, 2016, Journal of neurochemistry.

Depletion of TDP-43 decreases fibril and plaque β-amyloid and exacerbates neurodegeneration in an Alzheimer’s mouse model

P. Wong, L. Martin, Resham Chhabra, 2016, Acta Neuropathologica.

Calpain cleavage and inactivation of the sodium calcium exchanger-3 occur downstream of Aβ in Alzheimer’s disease

A. Jeromin, W. Noble, D. Hanger, 2013, Aging cell.