A. Saha

发表

Omega-3 and omega-6 fatty acids suppress ER- and oxidative stress in cultured neurons and neuronal progenitor cells from mice lacking PPT1

A. Mukherjee, Sung-Jo Kim, Zhongjian Zhang, 2010, Neuroscience Letters.

Production of lysophosphatidylcholine by cPLA2 in the brain of mice lacking PPT1 is a signal for phagocyte infiltration.

Yi-Ching Lee, A. Mukherjee, Sung-Jo Kim, 2007, Human molecular genetics.

RAGE signaling contributes to neuroinflammation in infantile neuronal ceroid lipofuscinosis

Yi-Ching Lee, A. Mukherjee, Sung-Jo Kim, 2008, FEBS letters.

Gene products promoting remyelination are up-regulated in a cell therapy product manufactured from banked human cord blood.

J. Kurtzberg, A. Balber, A. Saha, 2017, Cytotherapy.

Human umbilical cord blood monocytes, but not adult blood monocytes, rescue brain cells from hypoxic-ischemic injury: Mechanistic and therapeutic implications

J. Kurtzberg, A. Filiano, A. Balber, 2019, bioRxiv.

Cln1 gene disruption in mice reveals a common pathogenic link between two of the most lethal childhood neurodegenerative lysosomal storage disorders.

A. Mukherjee, Zhongjian Zhang, A. Saha, 2015, Human molecular genetics.

Disruption of adaptive energy metabolism and elevated ribosomal p-S6K1 levels contribute to INCL pathogenesis: partial rescue by resveratrol.

Z. Quezado, A. Mukherjee, Zhongjian Zhang, 2011, Human molecular genetics.

Cln1‐mutations suppress Rab7‐RILP interaction and impair autophagy contributing to neuropathology in a mouse model of infantile neuronal ceroid lipofuscinosis

A. Mukherjee, A. Saha, C. Sarkar, 2020, Journal of inherited metabolic disease.

FTY720 requires vitamin B12-TCN2-CD320 signaling in astrocytes to reduce disease in an animal model of multiple sclerosis

R. Rivera, E. Quadros, Manisha Ray, 2022, bioRxiv.

In a Model of Batten Disease, Palmitoyl Protein Thioesterase-1 Deficiency Is Associated with Brown Adipose Tissue and Thermoregulation Abnormalities

P. Moya, D. Koziol, Z. Quezado, 2012, PloS one.

The blood-brain barrier is disrupted in a mouse model of infantile neuronal ceroid lipofuscinosis: amelioration by resveratrol.

A. Mukherjee, J. Munasinghe, Zhongjian Zhang, 2012, Human molecular genetics.

Lack of an Endogenous Anti-inflammatory Protein in Mice Enhances Colonization of B16F10 Melanoma Cells in the Lungs*

Yi-Ching Lee, S. Su, A. Mukherjee, 2010, The Journal of Biological Chemistry.

Selective Impairment of a Subset of Ran-GTP-binding Domains of Ran-binding Protein 2 (Ranbp2) Suffices to Recapitulate the Degeneration of the Retinal Pigment Epithelium (RPE) Triggered by Ranbp2 Ablation*

N. Peachey, Minzhong Yu, A. Saha, 2014, The Journal of Biological Chemistry.

A cord blood monocyte-derived cell therapy product accelerates brain remyelination.

J. Troy, J. Kurtzberg, G. Matsushima, 2016, JCI insight.

EpitheliumResponses of the Mouse Retinal Pigment

B. Bell, N. Peachey, T. McIntyre, 2015 .

skeletal muscle fast-twitch fiber-dominant rats Increased wheel-running activity in the genetically

Sanjoy Ghosh, Y. Higaki, I. Laher, 2015 .

Selective loss of RPGRIP1-dependent ciliary targeting of NPHP4, RPGR and SDCCAG8 underlies the degeneration of photoreceptor neurons

N. Tserentsoodol, Y. Hao, A. Saha, 2012, Cell Death and Disease.

Hydrocortisone‐Treated DUOC‐01, a Cord Blood‐Derived Cell Therapy Product, Ameliorates Experimental Autoimmune Encephalomyelitis

J. Kurtzberg, A. Filiano, A. Saha, 2020 .

Human umbilical cord blood monocytes, but not adult blood monocytes, rescue brain cells from hypoxic-ischemic injury: Mechanistic and therapeutic implications

J. Kurtzberg, A. Filiano, A. Balber, 2019, bioRxiv.

Lack of an Endogenous Anti-inflammatory Protein in Mice Enhances Colonization of B16F10 Melanoma Cells in the Lungs*

Yi-Ching Lee, S. Su, A. Mukherjee, 2010, The Journal of Biological Chemistry.

Cln1 gene disruption in mice reveals a common pathogenic link between two of the most lethal childhood neurodegenerative lysosomal storage disorders.

A. Mukherjee, Zhongjian Zhang, A. Saha, 2015, Human molecular genetics.