S. Bealer

S. Bealer, 1999, News in physiological sciences : an international journal of physiology produced jointly by the International Union of Physiological Sciences and the American Physiological Society.

Paraventricular nucleus histamine increases blood pressure by adrenoreceptor stimulation of vasopressin release.

S. Bealer, S. Abell, 1995, The American journal of physiology.

Hypothalamic histamine release, neuroendocrine and cardiovascular responses during tuberomammillary nucleus stimulation in the conscious rat.

S. Bealer, V. Akins, 1993, Neuroendocrinology.

Brain histamine regulates pressor responses to peripheral hyperosmolality.

S. Bealer, V. Akins, 1990, The American journal of physiology.

The Vasopressor Response to Centrally Administered Ouabain

R. Caldwell, S. Bealer, E. Songu‐Mize, 1984, Circulation research.

Sodium excretion following lesions of preoptic recess periventricular tissue in the rat.

S. Bealer, 1983, The American journal of physiology.

Effect of AV3V lesions on development of DOCA-salt hypertension and vascular Na+-pump activity.

R. Caldwell, S. Bealer, E. Songu‐Mize, 1982, Hypertension.

Preoptic recess alpha-adrenoceptors control cardiovascular responses to hyperosmolality.

S. Bealer, 1997, The American journal of physiology.

Oxytocin release in magnocellular nuclei: neurochemical mediators and functional significance during gestation.

W. Armstrong, S. Bealer, W. R. Crowley, 2010, American journal of physiology. Regulatory, integrative and comparative physiology.

Acute hypertension increases norepinephrine release in the diagonal band of Broca

S. Bealer, 1997, Brain Research.

Ultrastructural effects of anteroventral third ventricle lesions on supraoptic nuclei and neural lobes of rats

M. Brody, A. Johnson, S. Bealer, 1981, Brain Research.

Preoptic-hypothalamic periventricular lesions: Impairment of thirst-motivated behavior

A. Johnson, S. Bealer, 1979, Physiology & Behavior.

Sodium consumption following lesions surrounding the anteroventral third ventricle

A. Johnson, S. Bealer, 1979, Brain Research Bulletin.

Adaptation and recovery of the rat chorda tympani response to NaCl

David V. Smith, S. Bealer, R. L. V. Buskirk, 1978, Physiology & Behavior.

Anteroventral third ventricle lesions reduce antidiuretic responses to angiotensin II.

A. K. Johnson, P. Schmid, S. Bealer, 1979, The American journal of physiology.

Systemic angiotensin II and volume expansion release norepinephrine in the preoptic recess

S. Bealer, 2000, Brain Research.

Anteroventral Third Ventricle Periventricular Tissue Contributes To Cardiac Baroreflex Responses

S. Bealer, 2000, Clinical and experimental pharmacology & physiology.

Preoptic recess ablation selectively increases baroreflex sensitivity to angiotensin II in conscious rats

S. Bealer, 1995, Peptides.

Preoptic Hypothalamic Control of Arteriolar Vasodilatory Responses

K. Proctor, S. Bealer, 1987, Circulation Research.

The effects of transverse cuts caudal to the preoptic recess on the fine structure of paraventricular nuclei in rats

S. Bealer, J. Carithers, 1985, Brain Research.

The effects of tranverse cuts caudal to the preoptic recess on the hypothalamo-neurohypophyseal neurosecretomy system

A. Johnson, S. Bealer, J. Carithers, 1984, Brain Research.

Sensitivity of the rat gustatory system to the rate of stimulus onset

David V. Smith, S. Bealer, 1975, Physiology & Behavior.

Dehydration and fluid balance: central effects of angiotensin.

W. Hoffman, S. Bealer, M. Phillips, 1982, Federation proceedings.

Hypothalamic knife cuts attenuate the pressor responses to angiotensin II.

S. Bealer, 1982, Neuroendocrinology.

Preoptic-hypothalamic periventricular lesions after food-associated drinking and circadian rhythms.

A. K. Johnson, S. Bealer, 1980, Journal of comparative and physiological psychology.

Fine structural evidence of degeneration in supraoptic nucleus and subfornical organ of rats with lesions in the anteroventral third ventricle

M. Brody, A. Johnson, S. Bealer, 1980, Brain Research.

Hypothalamic knife cuts alter vasopressin induced recovery of blood pressure following hemorrhage

S. Bealer, 1986, Experimental Brain Research.

Peripheral hyperosmolality reduces cardiac baroreflex sensitivity

S. Bealer, 2003, Autonomic Neuroscience.

Preoptic recess noradrenergic receptors control modification of baroreflex sensitivity by hypertonicity.

S. Bealer, 1999, American journal of physiology. Regulatory, integrative and comparative physiology.

Preoptic recess noradrenergic receptors control modification of baroreflex sensitivity by hypertonicity.

S. Bealer, 1999, The American journal of physiology.

Arterial baroreceptors and brain histamine contribute to bradycardia to peripheral hyperosmolality.

S. Bealer, M. Kenney, 1993, The American journal of physiology.

Preoptic recess lesions, body fluid compartments, and the renin-aldosterone system.

E. G. Schneider, S. Bealer, 1983, The American journal of physiology.

Central nervous system catecholamine content and norepinephrine release after AV3V ablation.

J. V. Van Huysse, S. Bealer, 1992, The American journal of physiology.

Skeletal muscle vasodilation during electrical stimulation of the preoptic recess.

K. Proctor, S. Bealer, 1986, The American journal of physiology.

Plasma corticosterone and volume after preoptic recess lesions and volume depletion.

E. G. Schneider, S. Bealer, 1985, The American journal of physiology.

Intensity coding in the transient portion of the rat chorda tympani response.

S. Bealer, 1978, Journal of comparative and physiological psychology.

Mechanisms of altered sodium excretion after preoptic hypothalamic lesions.

R. Caldwell, S. Bealer, E. Songu‐Mize, 1988, American Journal of Physiology.

Vascular capacitance following preoptic recess lesions.

S. Bealer, 1993, American Journal of Physiology.

Central depressor action of nitric oxide is deficient in genetic hypertension.

D. Bohr, S. Bealer, C. Cabrera, 1996, American journal of hypertension.

Preoptic hypothalamic lesions reduce adrenergic vascular compensation during hemorrhagic shock.

V. Pugh, S. Bealer, V. Schaumloffel, 1990, Circulatory shock.

Phasic Vascular Sodium Pump Changes in Deoxycorticosterone‐Hypertensive Rats

R. Caldwell, S. Bealer, E. Songu‐Mize, 1984, Circulation research.

Age-dependent behaviors, seizure severity and neuronal damage in response to nerve agents or the organophosphate DFP in immature and adult rats

F. Dudek, M. Lehmkuhle, E. Scholl, 2018, Neurotoxicology.

Autonomic and cellular mechanisms mediating detrimental cardiac effects of status epilepticus

A. Anderson, Cameron S. Metcalf, S. Bealer, 2010, Epilepsy Research.

Central blockade of oxytocin receptors during mid-late gestation reduces amplitude of slow afterhyperpolarization in supraoptic oxytocin neurons.

D. Lipschitz, W. Armstrong, S. Bealer, 2008, American journal of physiology. Endocrinology and metabolism.

β 1-Adrenergic Blockade Prevents Cardiac Dysfunction and Increased Susceptibility to Experimental Arrhythmias Following Status Epilepticus in Rats

Cameron S. Metcalf, S. Bealer, J. G. Little, 2015 .

Seizures following hippocampal kindling induce QT interval prolongation and increased susceptibility to arrhythmias in rats

S. Bealer, J. G. Little, 2013, Epilepsy Research.

Cardiac Myocyte Damage, Electrocardiographic Dysfunction, and Ion Channel Remodeling in Rodent Models of Seizure Disorders

A. Anderson, S. Poelzing, Cameron S. Metcalf, 2015 .

Status epilepticus produces chronic alterations in cardiac sympathovagal balance

P. Radwański, Cameron S. Metcalf, S. Bealer, 2009, Epilepsia.

Effect of DOCA‐Salt Treatment Duration and Anteroventral Third Ventricle Lesions on a Plasma‐Borne Sodium Pump Inhibitor in Rats*

R. Caldwell, S. Bealer, E. Songu‐Mize, 1987, Journal of hypertension.

Redistribution of cardiac output after hypothalamic lesions and hemorrhage

S. Bealer, D. Busija, 2004, Experimental Brain Research.

Multiple sensitivity to chemical stimuli in single human taste papillae

David V. Smith, S. Bealer, 1975, Physiology & Behavior.