T. Figueiredo

发表

Acetylcholinesterase inhibitors (nerve agents) as weapons of mass destruction: History, mechanisms of action, and medical countermeasures

V. Aroniadou-Anderjaska, J. P. Apland, T. Figueiredo, 2020, Neuropharmacology.

Diazepam administration after prolonged status epilepticus reduces neurodegeneration in the amygdala but not in the hippocampus during epileptogenesis

V. Aroniadou-Anderjaska, J. P. Apland, T. Figueiredo, 2008, Amino Acids.

Presynaptic facilitation of glutamate release in the basolateral amygdala: A mechanism for the anxiogenic and seizurogenic function of GluK1 receptors

V. Aroniadou-Anderjaska, V. Pidoplichko, T. Figueiredo, 2012, Neuroscience.

Acetylcholinesterase inhibition in the basolateral amygdala plays a key role in the induction of status epilepticus after soman exposure.

V. Aroniadou-Anderjaska, J. P. Apland, T. Figueiredo, 2013, Neurotoxicology.

Efficacy of the GluK1/AMPA Receptor Antagonist LY293558 against Seizures and Neuropathology in a Soman-Exposure Model without Pretreatment and its Pharmacokinetics after Intramuscular Administration

V. Aroniadou-Anderjaska, C. Green, J. P. Apland, 2013, The Journal of Pharmacology and Experimental Therapeutics.

Acute and long‐term consequences of exposure to organophosphate nerve agents in humans

A. Marini, J. P. Apland, T. Figueiredo, 2018, Epilepsia.

GABAergic circuits of the basolateral amygdala and generation of anxiety after traumatic brain injury

J. Juranek, L. Eiden, T. Figueiredo, 2022, Amino Acids.

Repeated Isoflurane Exposures Impair Long-Term Potentiation and Increase Basal GABAergic Activity in the Basolateral Amygdala

V. Aroniadou-Anderjaska, V. Pidoplichko, T. Figueiredo, 2016, Neural plasticity.

Antiseizure and Neuroprotective Efficacy of Midazolam in Comparison with Tezampanel (LY293558) against Soman-Induced Status Epilepticus

V. Aroniadou-Anderjaska, V. Pidoplichko, J. P. Apland, 2022, Toxics.

The GluK1 (GluR5) Kainate/α-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acid Receptor Antagonist LY293558 Reduces Soman-Induced Seizures and Neuropathology

V. Aroniadou-Anderjaska, J. P. Apland, T. Figueiredo, 2011, Journal of Pharmacology and Experimental Therapeutics.

The Recovery of GABAergic Function in the Hippocampus CA1 Region After mTBI

A. Marini, V. Pidoplichko, T. Figueiredo, 2019, Molecular Neurobiology.

Targeting the glutamatergic system to counteract organophosphate poisoning: A novel therapeutic strategy

V. Aroniadou-Anderjaska, J. P. Apland, T. Figueiredo, 2019, Neurobiology of Disease.

Comparing the Antiseizure and Neuroprotective Efficacy of LY293558, Diazepam, Caramiphen, and LY293558-Caramiphen Combination against Soman in a Rat Model Relevant to the Pediatric Population

V. Aroniadou-Anderjaska, V. Pidoplichko, J. P. Apland, 2018, The Journal of Pharmacology and Experimental Therapeutics.

Susceptibility to Soman Toxicity and Efficacy of LY293558 Against Soman-Induced Seizures and Neuropathology in 10-Month-Old Male Rats

V. Aroniadou-Anderjaska, J. P. Apland, T. Figueiredo, 2017, Neurotoxicity Research.

Pathological alterations in GABAergic interneurons and reduced tonic inhibition in the basolateral amygdala during epileptogenesis

B. Fritsch, M. Rogawski, V. Aroniadou-Anderjaska, 2009, Neuroscience.

Electroconvulsive shocks exacerbate the heightened acoustic startle response in stressed rats.

Sean T. Manion, V. Aroniadou-Anderjaska, T. Figueiredo, 2010, Behavioral neuroscience.

Electroencephalographic analysis in soman‐exposed 21‐day‐old rats and the effects of midazolam or LY293558 with caramiphen

V. Aroniadou-Anderjaska, J. P. Apland, T. Figueiredo, 2020, Annals of the New York Academy of Sciences.

Full Protection Against Soman-Induced Seizures and Brain Damage by LY293558 and Caramiphen Combination Treatment in Adult Rats

V. Aroniadou-Anderjaska, J. P. Apland, T. Figueiredo, 2018, Neurotoxicity Research.

Full Protection Against Soman-Induced Seizures and Brain Damage by LY293558 and Caramiphen Combination Treatment in Adult Rats

V. Aroniadou-Anderjaska, J. P. Apland, T. Figueiredo, 2018, Neurotoxicity Research.

Alpha-Linolenic Acid Treatment Reduces the Contusion and Prevents the Development of Anxiety-Like Behavior Induced by a Mild Traumatic Brain Injury in Rats

A. Marini, Hongna Pan, V. Pidoplichko, 2017, Molecular Neurobiology.

A rat model of nerve agent exposure applicable to the pediatric population: The anticonvulsant efficacies of atropine and GluK1 antagonists.

V. Aroniadou-Anderjaska, J. P. Apland, T. Figueiredo, 2015, Toxicology and applied pharmacology.

LY293558 prevents soman-induced pathophysiological alterations in the basolateral amygdala and the development of anxiety

V. Aroniadou-Anderjaska, J. P. Apland, T. Figueiredo, 2015, Neuropharmacology.

The Limitations of Diazepam as a Treatment for Nerve Agent–Induced Seizures and Neuropathology in Rats: Comparison with UBP302

V. Aroniadou-Anderjaska, Franco Rossetti, J. P. Apland, 2014, The Journal of Pharmacology and Experimental Therapeutics.

Reduced GABAergic Inhibition in the Basolateral Amygdala and the Development of Anxiety-Like Behaviors after Mild Traumatic Brain Injury

Eric M. Prager, A. Marini, L. Eiden, 2014, PloS one.

Long‐term neuropathological and behavioral impairments after exposure to nerve agents

V. Aroniadou-Anderjaska, V. Pidoplichko, J. P. Apland, 2016, Annals of the New York Academy of Sciences.

Repeated systemic administration of the nutraceutical alpha-linolenic acid exerts neuroprotective efficacy, an antidepressant effect and improves cognitive performance when given after soman exposure.

N. Grunberg, A. Marini, Jun Chen, 2015, Neurotoxicology.

α-Linolenic Acid, A Nutraceutical with Pleiotropic Properties That Targets Endogenous Neuroprotective Pathways to Protect against Organophosphate Nerve Agent-Induced Neuropathology

A. Marini, Hongna Pan, T. Figueiredo, 2015, Molecules.

Intranasal delivery of obidoxime to the brain prevents mortality and CNS damage from organophosphate poisoning.

J. Moffett, P. Arun, A. Namboodiri, 2016, Neurotoxicology.

Efficacy of the GluK 1 / AMPA Receptor Antagonist LY 293558 against Seizures and Neuropathology in a Soman-Exposure Model without Pretreatment and its Pharmacokinetics

V. Aroniadou-Anderjaska, C. Green, T. Figueiredo, 2013 .

Primary brain targets of nerve agents: the role of the amygdala in comparison to the hippocampus.

V. Aroniadou-Anderjaska, J. P. Apland, T. Figueiredo, 2009, Neurotoxicology.

Higher susceptibility of the ventral versus the dorsal hippocampus and the posteroventral versus anterodorsal amygdala to soman-induced neuropathology.

V. Aroniadou-Anderjaska, J. P. Apland, T. Figueiredo, 2010, Neurotoxicology.

Brief isoflurane administration as a post‐exposure treatment for organophosphate poisoning

J. Moffett, P. Arun, A. Namboodiri, 2017, Neurotoxicology.

Oscillatory Synchronous Inhibition in the Basolateral Amygdala and its Primary Dependence on NR2A-containing NMDA Receptors

V. Aroniadou-Anderjaska, V. Pidoplichko, T. Figueiredo, 2018, Neuroscience.

The recovery of acetylcholinesterase activity and the progression of neuropathological and pathophysiological alterations in the rat basolateral amygdala after soman-induced status epilepticus: Relation to anxiety-like behavior

V. Aroniadou-Anderjaska, Franco Rossetti, J. P. Apland, 2014, Neuropharmacology.

ASIC1a Activation Enhances Inhibition in the Basolateral Amygdala and Reduces Anxiety

V. Aroniadou-Anderjaska, V. Pidoplichko, T. Figueiredo, 2014, The Journal of Neuroscience.

The M1 Muscarinic Receptor Antagonist VU0255035 Delays the Development of Status Epilepticus after Organophosphate Exposure and Prevents Hyperexcitability in the Basolateral Amygdala

V. Aroniadou-Anderjaska, V. Pidoplichko, J. P. Apland, 2017, The Journal of Pharmacology and Experimental Therapeutics.

GABAergic interneuronal loss and reduced inhibitory synaptic transmission in the hippocampal CA1 region after mild traumatic brain injury

A. Marini, L. Eiden, V. Pidoplichko, 2015, Experimental Neurology.

Susceptibility to Soman Toxicity and Efficacy of LY293558 Against Soman-Induced Seizures and Neuropathology in 10-Month-Old Male Rats

V. Aroniadou-Anderjaska, C. Olsen, J. P. Apland, 2017, Neurotoxicity Research.

Alpha-linolenic acid enhances the facilitation of GABAergic neurotransmission in the BLA and CA1

Hongna Pan, T. Figueiredo, Ann M Marini, 2023, Experimental biology and medicine.

GABAergic interneuronal loss and reduced inhibitory synaptic transmission in the hippocampal CA1 region after mild traumatic brain injury

A. Marini, L. Eiden, V. Pidoplichko, 2015, Experimental Neurology.

The Recovery of GABAergic Function in the Hippocampus CA1 Region After mTBI

A. Marini, V. Pidoplichko, T. Figueiredo, 2019, Molecular Neurobiology.

V. Aroniadou-Anderjaska, Franco Rossetti, J. P. Apland, 2014, Neuropharmacology.

Neuroprotective efficacy of caramiphen against soman and mechanisms of its action

V. Aroniadou-Anderjaska, V. Pidoplichko, J. P. Apland, 2011, British journal of pharmacology.