C. Talora

发表

PLK1 targets NOTCH1 during DNA damage and mitotic progression

I. Screpanti, D. Bellavia, S. Checquolo, 2019, The Journal of Biological Chemistry.

High commitment of embryonic keratinocytes to terminal differentiation through a Notch1-caspase 3 regulatory mechanism.

G. Dotto, M. Woo, H. Tagami, 2004, Developmental cell.

A PKC-eta/Fyn-dependent pathway leading to keratinocyte growth arrest and differentiation.

P. Stein, G. Dotto, T. Kuroki, 2000, Molecular cell.

Functional cooperation between ASK1 and p21Waf1/Cip1 in the balance of cell-cycle arrest, cell death and tumorigenesis of stressed keratinocytes

I. Screpanti, S. Cialfi, H. Ichijo, 2021, Cell death discovery.

Identification of a novel chalcone derivative that inhibits Notch signaling in T-cell acute lymphoblastic leukemia

I. Screpanti, M. Mori, C. Ingallina, 2017, Scientific Reports.

Inhibition of ErbB-2 Mitogenic and Transforming Activity by RALT, a Mitogen-Induced Signal Transducer Which Binds to the ErbB-2 Kinase Domain

M. Crescenzi, O. Segatto, C. Talora, 2000, Molecular and Cellular Biology.

Yeast-Based Screen to Identify Natural Compounds with a Potential Therapeutic Effect in Hailey-Hailey Disease

S. Cialfi, D. Uccelletti, Azzurra Zonfrilli, 2018, International journal of molecular sciences.

WC-1 and the Proximal GATA Sequence Mediate a Cis-/Trans-Acting Repressive Regulation of Light-Dependent Gene Transcription in the Dark

Andrea Brenna, C. Talora, 2019, International journal of molecular sciences.

Autophagy processes are dependent on EGF receptor signaling

S. Martinotti, P. Conti, V. Flati, 2018, Oncotarget.

Maml1 acts cooperatively with Gli proteins to regulate sonic hedgehog signaling pathway

L. Di Marcotullio, I. Screpanti, D. Bellavia, 2017, Cell Death & Disease.

Notch Signal Mediates the Cross-Interaction between M2 Muscarinic Acetylcholine Receptor and Neuregulin/ErbB Pathway: Effects on Schwann Cell Proliferation

C. Talora, A. Tata, S. Loreti, 2022, Biomolecules.

The deregulated expression of miR-125b in acute myeloid leukemia is dependent on the transcription factor C/EBPα

I. Screpanti, D. Bellavia, S. Checquolo, 2015, Leukemia.

Glutathione S-transferase ϴ-subunit as a phenotypic suppressor of pmr1Δ strain, the Kluyveromyces lactis model for Hailey-Hailey disease.

S. Cialfi, D. Uccelletti, G. Biolcati, 2016, Biochimica et biophysica acta.

The Golgi Ca2+-ATPase KlPmr1p function is required for oxidative stress response by controlling the expression of the heat-shock element HSP60 in Kluyveromyces lactis.

P. Pinton, R. Rizzuto, D. Uccelletti, 2005, Molecular biology of the cell.

miR ‐ 125 b / NRF 2 / HO ‐ 1 axis is involved in protection against oxidative stress of cystic fibrosis : A pilot study

I. Screpanti, S. Cialfi, A. Pizzuti, 2021 .

The Translation Factor eIF6 Is a Notch-Dependent Regulator of Cell Migration and Invasion

S. Cialfi, P. Londei, D. Benelli, 2012, PloS one.

Pre‐TCR‐triggered ERK signalling‐dependent downregulation of E2A activity in Notch3‐induced T‐cell lymphoma

A. F. Campese, I. Screpanti, D. Bellavia, 2003, EMBO reports.

Cross talk among Notch3, pre-TCR, and Tal1 in T-cell development and leukemogenesis.

I. Screpanti, S. Cialfi, L. Frati, 2006, Blood.

Inhibition of microbial growth by carbon nanotube networks.

M. S. Sarto, M. Monthioux, S. Fiorito, 2013, Nanoscale.

M2 muscarinic receptor activation regulates schwann cell differentiation and myelin organization

B. Avallone, C. Talora, V. Magnaghi, 2014, Developmental neurobiology.

Cross talk among calcineurin, Sp1/Sp3, and NFAT in control of p21WAF1/CIP1 expression in keratinocyte differentiation

G. Dotto, C. Talora, M. P. Santini, 2001, Proceedings of the National Academy of Sciences of the United States of America.

Complex multipathways alterations and oxidative stress are associated with Hailey–Hailey disease

I. Screpanti, D. Taruscio, S. Cialfi, 2010, The British journal of dermatology.

Notch3 and the Notch3‐upregulated RNA‐binding protein HuD regulate Ikaros alternative splicing

A. F. Campese, Luigi Frati, I. Screpanti, 2007, The EMBO journal.

Hypotonic, Acidic Oxidizing Solution Containing Hypochlorous Acid (HClO) as a Potential Treatment of Hailey-Hailey Disease

I. Screpanti, S. Cialfi, Azzurra Zonfrilli, 2019, Molecules.

Corrigendum: The loss of ATP2C1 impairs the DNA damage response and induces altered skin homeostasis: Consequences for epidermal biology in Hailey-Hailey disease

I. Screpanti, S. Cialfi, D. Uccelletti, 2017, Scientific reports.

The loss of ATP2C1 impairs the DNA damage response and induces altered skin homeostasis: Consequences for epidermal biology in Hailey-Hailey disease

I. Screpanti, S. Cialfi, D. Uccelletti, 2016, Scientific Reports.

Efficacy of the melanocortin analogue Nle4-D-Phe7-α-melanocyte-stimulating hormone in the treatment of patients with Hailey–Hailey disease

I. Screpanti, S. Cialfi, G. Biolcati, 2013, Clinical and experimental dermatology.

Oxidative stress activation of miR‐125b is part of the molecular switch for Hailey–Hailey disease manifestation

I. Screpanti, G. Melino, D. Taruscio, 2011, Experimental dermatology.

Notch and NF-kB signaling pathways regulate miR-223/FBXW7 axis in T-cell acute lymphoblastic leukemia

A Gulino, I. Screpanti, D. Bellavia, 2014, Leukemia.

Glucocorticoid sensitivity of T-cell lymphoblastic leukemia/lymphoma is associated with glucocorticoid receptor-mediated inhibition of Notch1 expression

I. Screpanti, D. Bellavia, S. Checquolo, 2013, Leukemia.

Differential subcellular localization regulates c-Cbl E3 ligase activity upon Notch3 protein in T-cell leukemia

G. Ferrara, I. Screpanti, D. Bellavia, 2010, Oncogene.

Notch signaling and diseases: an evolutionary journey from a simple beginning to complex outcomes.

I. Screpanti, D. Bellavia, A. Gulino, 2008, Biochimica et biophysica acta.

Constitutively active Notch1 induces growth arrest of HPV-positive cervical cancer cells via separate signaling pathways.

I. Screpanti, S. Cialfi, L. Frati, 2005, Experimental cell research.

Specific down-modulation of Notch1 signaling in cervical cancer cells is required for sustained HPV-E6/E7 expression and late steps of malignant transformation.

D. Sgroi, G. Dotto, C. Crum, 2002, Genes & development.

Corrigendum: The loss of ATP2C1 impairs the DNA damage response and induces altered skin homeostasis: Consequences for epidermal biology in Hailey-Hailey disease

I. Screpanti, S. Cialfi, D. Uccelletti, 2017, Scientific Reports.

Human papilloma virus-dependent HMGA1 expression is a relevant step in cervical carcinogenesis.

I. Screpanti, G. Giannini, L. Frati, 2008, Neoplasia.

Targeting Notch to Maximize Chemotherapeutic Benefits: Rationale, Advanced Strategies, and Future Perspectives

I. Screpanti, S. Lazzari, M. P. Felli, 2021, Cancers.

Notch signaling is a direct determinant of keratinocyte growth arrest and entry into differentiation

Freddy Radtke, Daniel Metzger, Michel Aguet, 2001, The EMBO journal.

Role of a white collar‐1–white collar‐2 complex in blue‐light signal transduction

G. Macino, H. Linden, L. Franchi, 1999, The EMBO journal.

Roles in dimerization and blue light photoresponse of the PAS and LOV domains of Neurospora crassa white collar proteins

G. Macino, H. Linden, P. Ballario, 1998, Molecular microbiology.

NOTCH3 inactivation increases triple negative breast cancer sensitivity to gefitinib by promoting EGFR tyrosine dephosphorylation and its intracellular arrest

M. V. Giuli, E. Giuliani, D. Bellavia, 2018, Oncogenesis.

Mir-34a-5p Mediates Cross-Talk between M2 Muscarinic Receptors and Notch-1/EGFR Pathways in U87MG Glioblastoma Cells: Implication in Cell Proliferation

Pietro Laneve, E. Caffarelli, C. Talora, 2018, International journal of molecular sciences.

Novel TONSL variants cause SPONASTRIME dysplasia and associate with spontaneous chromosome breaks, defective cell proliferation and apoptosis.

D. Taruscio, S. Cialfi, S. Castellana, 2020, Human molecular genetics.

Inhibition of Eukaryotic Translation Initiation Factor 5A (eIF5A) Hypusination Suppress p53 Translation and Alters the Association of eIF5A to the Ribosomes

Caterina Catalanotto, P. Londei, D. Benelli, 2020, International journal of molecular sciences.

Prolyl-isomerase Pin1 controls Notch3 protein expression and regulates T-ALL progression

M. V. Giuli, I. Screpanti, D. Bellavia, 2016, Oncogene.

The epigenetic factor BORIS/CTCFL regulates the NOTCH3 gene expression in cancer cells.

I. Screpanti, S. Chiaretti, S. Cialfi, 2014, Biochimica et biophysica acta.

The Impact of the Ubiquitin System in the Pathogenesis of Squamous Cell Carcinomas

G. Melino, F. Bernassola, A. Peschiaroli, 2020, Cancers.

White collar‐1, a central regulator of blue light responses in Neurospora, is a zinc finger protein.

G. Macino, P. Ballario, P. Vittorioso, 1996, The EMBO journal.

DNA Damage Stress: Cui Prodest?

S. Cialfi, R. Palermo, Azzurra Zonfrilli, 2019, International journal of molecular sciences.

A threshold level of NFATc1 activity facilitates thymocyte differentiation and opposes notch-driven leukaemia development

I. Screpanti, K. Knobeloch, P. Cauchy, 2016, Nature Communications.

Identification of a Novel Curcumin Derivative Influencing Notch Pathway and DNA Damage as a Potential Therapeutic Agent in T-ALL

I. Screpanti, D. Bellavia, S. Checquolo, 2022, Cancers.

Histone Modifications Drive Aberrant Notch3 Expression/Activity and Growth in T-ALL

I. Screpanti, D. Bellavia, S. Checquolo, 2019, Front. Oncol..

Loss of Notch1-dependent p21Waf1/Cip1 expression influences the Notch1 outcome in tumorigenesis

I. Screpanti, D. Bellavia, S. Checquolo, 2014, Cell cycle.

“In vitro toxicity studies of zinc oxide nano- and microrods on mammalian cells: A comparative analysis”

M. S. Sarto, S. Cialfi, D. Uccelletti, 2016 .

Notch3/Jagged1 Circuitry Reinforces Notch Signaling and Sustains T-ALL

L. Di Marcotullio, I. Screpanti, D. Bellavia, 2014, Neoplasia.

SOD1, a New Kluyveromyces lactis Helper Gene for Heterologous Protein Secretion

S. Raimondi, D. Uccelletti, M. Rossi, 2008, Applied and Environmental Microbiology.

Notch3 contributes to T-cell leukemia growth via regulation of the unfolded protein response

M. V. Giuli, E. Giuliani, I. Screpanti, 2020, Oncogenesis.

Pmr-1 gene affects susceptibility of Caenorhabditis elegans to Staphylococcus aureus infection through glycosylation and stress response pathways' alterations

Emily Schifano, D. Uccelletti, P. Mancini, 2019, Virulence.

Regulation of proapoptotic proteins Bak1 and p53 by miR-125b in an experimental model of Alzheimer's disease: Protective role of 17β-estradiol

F. Micheli, R. Palermo, C. Talora, 2016, Neuroscience Letters.

Kras/ADAM17-dependent Jag1-ICD reverse signalling sustains colorectal cancer progression and chemoresistance.

L. Di Marcotullio, I. Screpanti, D. Bellavia, 2019, Cancer research.

The molecular basis of notch signaling regulation: a complex simplicity.

I. Screpanti, D. Bellavia, S. Checquolo, 2014, Current molecular medicine.

PKCθ mediates pre-TCR signaling and contributes to Notch3-induced T-cell leukemia

I. Screpanti, D. Bellavia, S. Checquolo, 2005, Oncogene.

Loss of ATP2C1 function promotes trafficking and degradation of NOTCH1: Implications for Hailey‐Hailey disease

I. Screpanti, D. Bellavia, S. Checquolo, 2023, Experimental dermatology.

PKC theta mediates pre-TCR signaling and contributes to Notch3-induced T-cell leukemia.

I. Screpanti, D. Bellavia, S. Checquolo, 2005, Oncogene.

The deregulated expression of miR-125b in acute myeloid leukemia is dependent on the transcription factor C/EBPα

C. Talora, P. V. Romero, 2015 .

Notch signaling and diseases: an evolutionary journey from a simple beginning to complex outcomes.

I. Screpanti, D. Bellavia, A. Gulino, 2008, Biochimica et biophysica acta.