E. D. Crown

发表

Changes in Motoneuron Properties and Synaptic Inputs Related to Step Training after Spinal Cord Transection in Rats

V. Edgerton, D. Jindrich, R. Roy, 2007, The Journal of Neuroscience.

Instrumental learning within the spinal cord: underlying mechanisms and implications for recovery after injury.

Adam R Ferguson, Rajesh C Miranda, J. Grau, 2006, Behavioral and cognitive neuroscience reviews.

Impact of Behavioral Control on the Processing of Nociceptive Stimulation

Adam R Ferguson, J. Grau, E. D. Crown, 2012, Front. Physio..

Instrumental learning within the spinal cord: V. Evidence the behavioral deficit observed after noncontingent nociceptive stimulation reflects an intraspinal modification

Adam R Ferguson, J. Grau, R. Joynes, 2003, Behavioural Brain Research.

Instrumental learning within the spinal cord: IV. Induction and retention of the behavioral deficit observed after noncontingent shock.

Adam R Ferguson, J. Grau, R. Joynes, 2002, Behavioral neuroscience.

Preserving and restoring behavioral potential within the spinal cord using an instrumental training paradigm.

J. Grau, E. D. Crown, 2001, Journal of neurophysiology.

Nociceptive plasticity inhibits adaptive learning in the spinal cord

Adam R Ferguson, J. Grau, E. D. Crown, 2006, Neuroscience.

Instrumental Learning within the Rat Spinal Cord: Localization of the Essential Neural Circuit

Adam R Ferguson, J. Grau, R. Miranda, 2005 .

The behavioral deficit observed following noncontingent shock in spinalized rats is prevented by the protein synthesis inhibitor cycloheximide.

Adam R Ferguson, J. Grau, E. D. Crown, 2004, Behavioral neuroscience.

Central nociceptive sensitization vs. spinal cord training: opposing forms of plasticity that dictate function after complete spinal cord injury

Adam R Ferguson, J. Grau, E. D. Crown, 2012, Front. Physio..

Mechanisms of chronic central neuropathic pain after spinal cord injury

C. Hulsebosch, B. C. Hains, E. D. Crown, 2009, Brain Research Reviews.

The role of mitogen activated protein kinase signaling in microglia and neurons in the initiation and maintenance of chronic pain

E. D. Crown, 2012, Experimental Neurology.

Activation of p38 MAP kinase is involved in central neuropathic pain following spinal cord injury

C. Hulsebosch, Kathia M. Johnson, E. D. Crown, 2008, Experimental Neurology.

Preemptive analgesia with lidocaine prevents Failed Back Surgery Syndrome

C. Hulsebosch, D. McAdoo, E. D. Crown, 2007, Experimental Neurology.

Increases in the activated forms of ERK 1/2, p38 MAPK, and CREB are correlated with the expression of at-level mechanical allodynia following spinal cord injury

C. Hulsebosch, D. McAdoo, Kathia M. Johnson, 2006, Experimental Neurology.

BOLD and blood volume-weighted fMRI of rat lumbar spinal cord during non-noxious and noxious electrical hindpaw stimulation

Fuqiang Zhao, Mangay Williams, Xiangjun Meng, 2008, NeuroImage.

HC-030031, a TRPA1 selective antagonist, attenuates inflammatory- and neuropathy-induced mechanical hypersensitivity

D. Henze, M. Urban, S. R. Eid, 2008, Molecular pain.

Maladaptive spinal plasticity opposes spinal learning and recovery in spinal cord injury

Adam R Ferguson, J. Grau, E. D. Crown, 2012, Front. Physio..

Two chronic motor training paradigms differentially influence acute instrumental learning in spinally transected rats

Adam R Ferguson, V. Edgerton, R. Roy, 2007, Behavioural Brain Research.

Evidence that descending serotonergic systems protect spinal cord plasticity against the disruptive effect of uncontrollable stimulation

J. Grau, E. D. Crown, 2005, Experimental Neurology.

Calcium/calmodulin dependent kinase II contributes to persistent central neuropathic pain following spinal cord injury

C. Hulsebosch, D. McAdoo, Kathia M. Johnson, 2012, PAIN.

Shock-induced hyperalgesia: IV. Generality.

Adam R Ferguson, J. Grau, M. Meagher, 2001, Journal of experimental psychology. Animal behavior processes.

Propentofylline attenuates allodynia, glial activation and modulates GABAergic tone after spinal cord injury in the rat

C. Hulsebosch, G. Unabia, E. D. Crown, 2008, PAIN.

Upregulation of the phosphorylated form of CREB in spinothalamic tract cells following spinal cord injury: Relation to central neuropathic pain

C. Hulsebosch, D. McAdoo, K. Westlund, 2005, Neuroscience Letters.

Shock-induced hyperalgesia: Evidence forebrain systems play an essential role

J. Grau, M. Meagher, R. Joynes, 1999, Behavioural Brain Research.

GABA(A) receptor activation is involved in noncontingent shock inhibition of instrumental conditioning in spinal rats.

Adam R Ferguson, J. Grau, E. D. Crown, 2003, Behavioral neuroscience.

Shock-induced hyperalgesia: III. Role of the bed nucleus of the stria terminalis and amygdaloid nuclei.

J. Grau, M. Meagher, E. D. Crown, 2000, Behavioral neuroscience.

Pain in a balance: noxious events engage opposing processes that concurrently modulate nociceptive reactivity.

J. Grau, M. Meagher, E. D. Crown, 2004, Behavioral neuroscience.

Uncontrollable stimulation undermines recovery after spinal cord injury.

Adam R Ferguson, K. Bolding, J. Grau, 2004, Journal of neurotrauma.

Instrumental learning within the spinal cord II. Evidence for central mediation

Adam R Ferguson, J. Grau, R. Joynes, 2002, Physiology & Behavior.

Propentofylline attenuates allodynia, glial activation and modulates GABAergic tone after spinal cord injury in the rat

C. Hulsebosch, G. Unabia, E. D. Crown, 2008, PAIN.

Calcium/calmodulin dependent kinase II contributes to persistent central neuropathic pain following spinal cord injury

C. Hulsebosch, D. McAdoo, Kathia M. Johnson, 2012, PAIN.