S. Michael

发表

Correction for Kong et al., Using distance correlation and SS-ANOVA to assess associations of familial relationships, lifestyle factors, diseases, and mortality

Yeon-Joo Kang, Samuel Andrew Hires, S. Mckercher, 2013, Proceedings of the National Academy of Sciences.

Neuron-released oligomeric α-synuclein is an endogenous agonist of TLR2 for paracrine activation of microglia

E. Masliah, D. Hwang, Junghee Kang, 2013, Nature Communications.

α-Synuclein in Central Nervous System and from Erythrocytes, Mammalian Cells, and Escherichia coli Exists Predominantly as Disordered Monomer*

Bernard Schneider, Niels Lion, Patrick Aebischer, 2012, The Journal of Biological Chemistry.

Lentivirus mediated delivery of neurosin promotes clearance of wild-type α-synuclein and reduces the pathology in an α-synuclein model of LBD.

E. Masliah, E. Rockenstein, B. Spencer, 2013, Molecular therapy : the journal of the American Society of Gene Therapy.

ESCRT-mediated Uptake and Degradation of Brain-targeted α-synuclein Single Chain Antibody Attenuates Neuronal Degeneration In Vivo

E. Masliah, E. Rockenstein, B. Spencer, 2014, Molecular therapy : the journal of the American Society of Gene Therapy.

Detection of peri-synaptic amyloid-β pyroglutamate aggregates in early stages of Alzheimer's disease and in AβPP transgenic mice using a novel monoclonal antibody.

D. Galasko, E. Masliah, E. Rockenstein, 2012, Journal of Alzheimer's disease : JAD.

Phospholipase D1 regulates autophagic flux and clearance of α-synuclein aggregates

E. Masliah, D. Min, S.-J. Lee, 2014, Cell Death and Differentiation.

Microsoft Word-Fauvet and Lashuel et al JBC main text corrected abstract 07Feb12.docx

E. Masliah, P. Aebischer, H. Lashuel, 2012 .

Aβ induces astrocytic glutamate release, extrasynaptic NMDA receptor activation, and synaptic loss

S. Lipton, L. Parsons, S. Heinemann, 2013, Proceedings of the National Academy of Sciences.

Structural diversity of Alzheimer's disease amyloid-β dimers and their role in oligomerization and fibril formation.

E. Masliah, I. Tsigelny, V. Kouznetsova, 2014, Journal of Alzheimer's disease : JAD.

Widespread microRNA dysregulation in multiple system atrophy – disease‐related alteration in miR‐96

D. Galasko, E. Masliah, E. Rockenstein, 2014, The European journal of neuroscience.

Progressive accumulation of amyloid‐β oligomers in Alzheimer’s disease and in amyloid precursor protein transgenic mice is accompanied by selective alterations in synaptic scaffold proteins

J. Yates, D. Salmon, D. Galasko, 2010, The FEBS journal.

Neural stem cells genetically-modified to express neprilysin reduce pathology in Alzheimer transgenic models

E. Masliah, J. Loring, F. LaFerla, 2014, Stem Cell Research & Therapy.

Beneficial effects of a neurotrophic peptidergic mixture persist for a prolonged period following treatment interruption in a transgenic model of Alzheimer's disease

E. Masliah, E. Rockenstein, A. Adame, 2011, Journal of neuroscience research.

Peripheral Delivery of a CNS Targeted, Metalo-Protease Reduces Aβ Toxicity in a Mouse Model of Alzheimer's Disease

E. Masliah, I. Verma, E. Rockenstein, 2011, PloS one.

Alpha-synuclein sequesters Dnmt1 from the nucleus: a novel mechanism for epigenetic alterations in Lewy body diseases.

E. Masliah, E. Rockenstein, B. Spencer, 2011, The Journal of biological chemistry.

A novel Rac1 GAP splice variant relays poly-Ub accumulation signals to mediate Rac1 inactivation

J. Yates, E. Masliah, Timothy Y. Huang, 2013, Molecular biology of the cell.

A novel triple-repeat mutant tau transgenic model that mimics aspects of Pick's disease and frontotemporal tauopathies

E. Masliah, E. Rockenstein, A. Adame, 2013, Alzheimer's & Dementia.

α-Synuclein Sequesters Dnmt1 from the Nucleus

E. Masliah, E. Rockenstein, B. Spencer, 2011, The Journal of Biological Chemistry.