Hongquan Jiang

发表

Blood Volatile Organic Compounds as Potential Biomarkers for Amyotrophic Lateral Sclerosis: an Animal Study in the SOD1 G93A Mouse

Xiang Yin, Enyou Li, Hongquan Jiang, 2014, Journal of Molecular Neuroscience.

Differential peroxisome proliferator activated receptors activity in a rodent model of amyotrophic lateral sclerosis.

M. Ren, Hongquan Jiang, Xiang Yin, 2015, International journal of clinical and experimental medicine.

Lithium and valproate acid protect NSC34 cells from H2O2-induced oxidative stress and upregulate expressions of SIRT3 and CARM1.

Hongquan Jiang, H. Feng, Jing Wang, 2013, Neuro endocrinology letters.

Roscovitine treatment caused impairment of fertilizing ability in mice.

S. Cui, M. Ren, Hongquan Jiang, 2015, Toxicology letters.

Astrocyte elevated gene-1 is a novel regulator of astrogliosis and excitatory amino acid transporter-2 via interplaying with nuclear factor-κB signaling in astrocytes from amyotrophic lateral sclerosis mouse model with hSOD1G93A mutation

Hongquan Jiang, Xiang Yin, H. Feng, 2018, Molecular and Cellular Neuroscience.

Spy1, a unique cell cycle regulator, alters viability in ALS motor neurons and cell lines in response to mutant SOD1-induced DNA damage.

Xudong Wang, Hongquan Jiang, Xiang Yin, 2019, DNA repair.

Downregulation of Homer1b/c in SOD1 G93A Models of ALS: A Novel Mechanism of Neuroprotective Effect of Lithium and Valproic Acid

Hongquan Jiang, Xiang Yin, H. Feng, 2016, International journal of molecular sciences.

Corrigendum: PGC-1α silencing compounds the perturbation of mitochondrial function caused by mutant SOD1 in skeletal muscle of ALS mouse model

M. Ren, Hongquan Jiang, Xiang Yin, 2016, Front. Aging Neurosci..

PGC-1α Silencing Compounds the Perturbation of Mitochondrial Function Caused by Mutant SOD1 in Skeletal Muscle of ALS Mouse Model

M. Ren, Hongquan Jiang, Xiang Yin, 2015, Front. Aging Neurosci..

Downregulated AEG-1 together with inhibited PI3K/Akt pathway is associated with reduced viability of motor neurons in an ALS model

D. Chuang, S. Cui, P. Leeds, 2015, Molecular and Cellular Neuroscience.

Comparative Analysis of VOCs in Exhaled Breath of Amyotrophic Lateral Sclerosis and Cervical Spondylotic Myelopathy Patients

Changsong Wang, Lei Guo, E. Li, 2016, Scientific reports.

Allopurinol reduces severity of delayed neurologic sequelae in experimental carbon monoxide toxicity in rats.

M. Ren, Hongquan Jiang, Xiang Yin, 2015, Neurotoxicology.

Left ventricular ejection fraction as an independent predictor of poor outcome in acute intracerebral hemorrhage

Daming Zhang, Shiguang Zhao, Hongquan Jiang, 2020, Brain and behavior.

α-Lipoic acid attenuates oxidative stress and neurotoxicity via the ERK/Akt-dependent pathway in the mutant hSOD1 related Drosophila model and the NSC34 cell line of amyotrophic lateral sclerosis

Hongquan Jiang, H. Feng, Shuyu Wang, 2018, Brain Research Bulletin.

Blood Volatile Organic Compounds as Potential Biomarkers for Amyotrophic Lateral Sclerosis: an Animal Study in the SOD1 G93A Mouse

M. Ren, Chaofu Ke, Changsong Wang, 2014, Journal of Molecular Neuroscience.

Neuroprotection by urate on the mutant hSOD1-related cellular and Drosophila models of amyotrophic lateral sclerosis: Implication for GSH synthesis via activating Akt/GSK3β/Nrf2/GCLC pathways

Hongquan Jiang, H. Feng, Shuyu Wang, 2019, Brain Research Bulletin.

Lithium facilitates removal of misfolded proteins and attenuated faulty interaction between mutant SOD1 and p-CREB (Ser133) through enhanced autophagy in mutant hSOD1G93A transfected neuronal cell lines

Hongquan Jiang, Xiang Yin, H. Feng, 2019, Molecular Biology Reports.

Notch pathway is activated in cell culture and mouse models of mutant SOD1-related familial amyotrophic lateral sclerosis, with suppression of its activation as an additional mechanism of neuroprotection for lithium and valproate

M. Ren, Y. Qi, Hongquan Jiang, 2015, Neuroscience.