W. Martins

发表

Patterns of chemokines and chemokine receptors expression in different forms of human periodontal disease.

G. Garlet, J. Silva, B. Ferreira, 2003, Journal of periodontal research.

Differential expression of chemokines and chemokine receptors in inflammatory periapical diseases.

G. Garlet, F. Cunha, W. Martins, 2005, Oral microbiology and immunology.

An Interleukin-1β (IL-1β) Single-Nucleotide Polymorphism at Position 3954 and Red Complex Periodontopathogens Independently and Additively Modulate the Levels of IL-1β in Diseased Periodontal Tissues

G. Garlet, A. Trombone, J. Silva, 2008, Infection and Immunity.

Matrix metalloproteinases, their physiological inhibitors and osteoclast factors are differentially regulated by the cytokine profile in human periodontal disease.

G. Garlet, J. Silva, B. A. Fonseca, 2004, Journal of clinical periodontology.

The broad effects of the functional IL‐10 promoter‐592 polymorphism: modulation of IL‐10, TIMP‐3, and OPG expression and their association with periodontal disease outcome

G. Garlet, A. Trombone, J. Silva, 2008, Journal of leukocyte biology.

A Role for the Laminin Receptor in Leukocyte Chemotaxis

L. Liotta, C. N. Rao, M. Brentani, 1987, Journal of leukocyte biology.

Association of human T lymphotropic virus 1 amplification of periodontitis severity with altered cytokine expression in response to a standard periodontopathogen infection.

G. Garlet, A. Trombone, E. Carvalho, 2010, Clinical infectious diseases : an official publication of the Infectious Diseases Society of America.

Tumor necrosis factor-alpha -308G/A single nucleotide polymorphism and red-complex periodontopathogens are independently associated with increased levels of tumor necrosis factor-alpha in diseased periodontal tissues.

G. Garlet, A. Trombone, W. Martins, 2009, Journal of periodontal research.

Strong and persistent microbial and inflammatory stimuli overcome the genetic predisposition to higher matrix metalloproteinase-1 (MMP-1) expression: a mechanistic explanation for the lack of association of MMP1-1607 single-nucleotide polymorphism genotypes with MMP-1 expression in chronic periodont

G. Garlet, A. Trombone, J. Silva, 2009, Journal of clinical periodontology.

An Interleukin-1 (cid:2) (IL-1 (cid:2) ) Single-Nucleotide Polymorphism at Position 3954 and Red Complex Periodontopathogens Independently and Additively Modulate the Levels of IL-1 (cid:2) in Diseased Periodontal Tissues (cid:1)

G. Garlet, A. Trombone, J. Silva, 2008 .

TBX21-1993T/C (rs4794067) polymorphism is associated with increased risk of chronic periodontitis and increased T-bet expression in periodontal lesions, but does not significantly impact the IFN-g transcriptional level or the pattern of periodontophatic bacterial infection

G. Garlet, W. Martins, A. Letra, 2015, Virulence.

Expression of suppressors of cytokine signaling in diseased periodontal tissues: a stop signal for disease progression?

G. Garlet, W. Martins, J. Silva, 2006, Journal of periodontal research.

CCR5&Dgr;32 (rs333) polymorphism is associated with decreased risk of chronic and aggressive periodontitis: A case‐control analysis based in disease resistance and susceptibility phenotypes

G. Garlet, W. Martins, C. Santos, 2017, Cytokine.

The use of chronic gingivitis as reference status increases the power and odds of periodontitis genetic studies: a proposal based in the exposure concept and clearer resistance and susceptibility phenotypes definition.

G. Garlet, A. Trombone, A. E. Vieira, 2012, Journal of clinical periodontology.

An Interleukin-1β (IL-1β) Single-Nucleotide Polymorphism at Position 3954 and Red Complex Periodontopathogens Independently and Additively Modulate the Levels of IL-1β in Diseased Periodontal Tissues

G. Garlet, A. Trombone, J. Silva, 2008, Infection and Immunity.