G. S. Hossain

Homocysteine-induced endoplasmic reticulum stress causes dysregulation of the cholesterol and triglyceride biosynthetic pathways.

Yuan-Yuan Shi, N. Maeda, G. Werstuck, 2001, The Journal of clinical investigation.

Altered expression of caveolin-1 and increased cholesterol in detergent insoluble membrane fractions from liver in mice with Niemann-Pick disease type C.

R. Erickson, J. Wilson, R. Heidenreich, 1997, Biochimica et biophysica acta.

Association of Multiple Cellular Stress Pathways With Accelerated Atherosclerosis in Hyperhomocysteinemic Apolipoprotein E-Deficient Mice

E. Falk, G. Werstuck, M. Ritskes-Hoitinga, 2004, Circulation.

Cyclodextrins in the treatment of a mouse model of Niemann-Pick C disease.

F. Camargo, R. Erickson, J. Blanchard, 2001, Life sciences.

The Potential for microRNA Therapeutics and Clinical Research

J. Kocerha, G. S. Hossain, Johora Hanna, 2019, Front. Genet..

The Npc1 mutation causes an altered expression of caveolin-1, annexin II and protein kinases and phosphorylation of caveolin-1 and annexin II in murine livers.

R. Heidenreich, G. S. Hossain, W. Garver, 1999, Biochimica et biophysica acta.

TDAG51 Is Induced by Homocysteine, Promotes Detachment-mediated Programmed Cell Death, and Contributes to the Development of Atherosclerosis in Hyperhomocysteinemia*

E. Falk, R. Kaufman, D. Tang, 2003, Journal of Biological Chemistry.

Deficiency of TDAG51 Protects Against Atherosclerosis by Modulating Apoptosis, Cholesterol Efflux, and Peroxiredoxin‐1 Expression

J. Capone, M. Malloy, C. Pullinger, 2013, Journal of the American Heart Association.

Peroxynitrite Causes Endoplasmic Reticulum Stress and Apoptosis in Human Vascular Endothelium: Implications in Atherogenesis

R. Austin, J. Dickhout, G. S. Hossain, 2005, Arteriosclerosis, thrombosis, and vascular biology.