L. Cappabianca

发表

Reconstitution of TIMP-2 expression in SH-SY5Y neuroblastoma cells by 5-azacytidine is mediated transcriptionally by NF-Y through an inverted CCAAT site.

R. Mantovani, A. Gulino, L. Cappabianca, 2003, Experimental cell research.

Mechanisms involved in selecting and maintaining neuroblastoma cancer stem cell populations, and perspectives for therapeutic targeting

L. Cappabianca, A. Farina, A. Mackay, 2021, World journal of stem cells.

Alendronate promotes plasmin‐mediated MMP‐9 inactivation by exposing cryptic plasmin degradation sites within the MMP‐9 catalytic domain

A. Gulino, Natalia Di Ianni, L. Cappabianca, 2012, FEBS letters.

Retrograde TrkAIII transport from ERGIC to ER: a re-localisation mechanism for oncogenic activity

R. Maccarone, L. Cappabianca, A. Farina, 2015, Oncotarget.

The TrkAIII Oncoprotein Inhibits Mitochondrial Free Radical ROS-Induced Death of SH-SY5Y Neuroblastoma Cells by Augmenting SOD2 Expression and Activity at the Mitochondria, within the Context of a Tumour Stem Cell-like Phenotype

A. Gulino, Natalia Di Ianni, L. Cappabianca, 2014, PloS one.

Alternative TrkAIII splicing: a potential regulated tumor-promoting switch and therapeutic target in neuroblastoma.

A. Gulino, L. Cappabianca, A. Farina, 2005, Future oncology.

In vivo analysis of the model tyrosine aminotransferase gene reveals multiple sequential steps in glucocorticoid receptor action

T. Grange, L. Cappabianca, H. Thomassin, 2001, Oncogene.

Genomic footprinting using nucleases.

R. Pictet, T. Grange, L. Cappabianca, 1999, Methods in molecular biology.

Follicle-stimulating hormone increases the expression of tissue inhibitors of metalloproteinases TIMP-1 and TIMP-2 and induces TIMP-1 AP-1 site binding complex(es) in prepubertal rat Sertoli cells.

W. Stetler-Stevenson, G. D’Orazi, E. Jannini, 1994, Endocrinology.

Retinoic acid‐enhanced invasion through reconstituted basement membrane by human SK‐N‐SH neuroblastoma cells involves membrane‐associated tissue‐type plasminogen activator

A. Gulino, A. Tiberio, L. Cappabianca, 1997, International journal of cancer.

Positive and Negative Regulation of the Composite Octamer Motif of the Interleukln 2 Enhancer by AP-1 , OCT-2 , and Retinoic Acid Receptor By Ugo

I. Screpanti, L. Frati, A. Gulino, 2003 .

Positive and negative regulation of the composite octamer motif of the interleukin 2 enhancer by AP-1, Oct-2, and retinoic acid receptor

I. Screpanti, L. Frati, A. Gulino, 1994, The Journal of experimental medicine.

Positive and Negative Regulation of the Composite Octamer Motif of the Interleukln 2 Enhancer by AP-1, OCT-2, and Retinoic Acid Receptor By Ugo de Grazia,~ll Maria Pia Felli,* Alessandra Vacca,~

I. Screpanti, L. Frati, A. Gulino, 1994 .

Doxorubicin-Induced TrkAIII Activation: A Selection Mechanism for Resistant Dormant Neuroblastoma Cells

L. Cappabianca, A. Farina, A. Mackay, 2022, International journal of molecular sciences.

Inhibition of human MDA-MB-231 breast cancer cell invasion by matrix metalloproteinase 3 involves degradation of plasminogen.

A. Gulino, L. Cappabianca, A. Farina, 2002, European journal of biochemistry.

Thioredoxin alters the matrix metalloproteinase/tissue inhibitors of metalloproteinase balance and stimulates human SK-N-SH neuroblastoma cell invasion.

A. Holmgren, A. Gulino, G. Beckett, 2001, European journal of biochemistry.

Advanced Merkel cell carcinoma of the lower extremity treated with surgery and isolated pelvic and limb perfusion using Melphalan: A case of unexpected long-term survival

L. Cappabianca, A. Farina, A. Mackay, 2019, International journal of surgery case reports.

Nutraceutical Preventative and Therapeutic Potential in Neuroblastoma: From Pregnancy to Early Childhood

M. Ruggieri, L. Cappabianca, A. Farina, 2022, Life.

Identification of plasminogen in Matrigel and its activation by reconstitution of this basement membrane extract.

A. Gulino, A. Tiberio, L. Cappabianca, 1996, BioTechniques.

Hypoxia-induced alternative splicing: the 11th Hallmark of Cancer

L. Cappabianca, A. Farina, A. Mackay, 2020, Journal of experimental & clinical cancer research : CR.

Discovery, characterization and potential roles of a novel NF-YAx splice variant in human neuroblastoma

L. Di Marcotullio, L. Cappabianca, A. Farina, 2019, Journal of Experimental & Clinical Cancer Research.

The oncogenic neurotrophin receptor tropomyosin-related kinase variant, TrkAIII

M. Fargnoli, L. Cappabianca, A. Farina, 2018, Journal of Experimental & Clinical Cancer Research.

TrkA alternative splicing: a regulated tumor-promoting switch in human neuroblastoma.

I. Screpanti, A. Tessitore, A. Gulino, 2004, Cancer cell.

Alternative TrkA Splicing and Neuroblastoma

A. Gulino, L. Cappabianca, A. Farina, 2012 .

All-trans-retinoic acid induces nuclear factor kappaB activation and matrix metalloproteinase-9 expression and enhances basement membrane invasivity of differentiation-resistant human SK-N-BE 9N neuroblastoma Cells.

A. Gulino, L. Cappabianca, A. Farina, 2002, Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research.

All-trans-Retinoic Acid Induces Nuclear Factor B Activation and Matrix Metalloproteinase-9 Expression and Enhances Basement Membrane Invasivity of Differentiation-resistant Human SK-NBE 9 N Neuroblastoma Cells 1

A. Gulino, L. Cappabianca, A. Farina, 2002 .

Transcriptional regulation of intercellular adhesion molecule 1 by phorbol ester in human neuroblastoma cell line SK-N-SH involves jun- and fos-containing activator protein 1 site binding complex(es).

A. Tessitore, L. Frati, A. Gulino, 1997, Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research.

The neuroblastoma tumour-suppressor TrkAI and its oncogenic alternative TrkAIII splice variant exhibit geldanamycin-sensitive interactions with Hsp90 in human neuroblastoma cells

A Gulino, A. Gulino, A. Romanelli, 2009, Oncogene.

TrkAIII: A Novel Hypoxia-regulated Alternative TrkA Splice Variant of Potential Physiological and Pathological Importance

A. Gulino, L. Cappabianca, A. Farina, 2005, Cell cycle.

A pilot study of alternative TrkAIII splicing in Merkel cell carcinoma: a potential oncogenic mechanism and novel therapeutic target

R. Maccarone, L. Cappabianca, A. Farina, 2019, Journal of Experimental & Clinical Cancer Research.

Nature of the Accessible Chromatin at a Glucocorticoid-Responsive Enhancer

T. Grange, L. Cappabianca, H. Thomassin, 2004, Molecular and Cellular Biology.

Multidisciplinary Treatment, Including Locoregional Chemotherapy, for Merkel-Polyomavirus-Positive Merkel Cell Carcinomas: Perspectives for Patients Exhibiting Oncogenic Alternative Δ exon 6–7 TrkAIII Splicing of Neurotrophin Receptor Tropomyosin-Related Kinase A

R. Maccarone, G. Fiorentini, E. Ricevuto, 2020, International journal of molecular sciences.

Thioredoxin stimulates MMP‐9 expression, de‐regulates the MMP‐9/TIMP‐1 equilibrium and promotes MMP‐9 dependent invasion in human MDA‐MB‐231 breast cancer cells

A. Gulino, L. Cappabianca, A. Farina, 2011, FEBS letters.

The Alternative TrkAIII Splice Variant Targets the Centrosome and Promotes Genetic Instability

A. Gulino, C. Pedone, A. Romanelli, 2009, Molecular and Cellular Biology.

Thioredoxin inhibits microvascular endothelial capillary tubule formation.

A. Gulino, L. Cappabianca, A. Farina, 2003, Experimental cell research.

TrkAIII Promotes Microtubule Nucleation and Assembly at the Centrosome in SH-SY5Y Neuroblastoma Cells, Contributing to an Undifferentiated Anaplastic Phenotype

A. Gulino, L. Cappabianca, A. Farina, 2013, BioMed research international.

The Alternative TrkAIII Splice Variant, a Targetable Oncogenic Participant in Human Cutaneous Malignant Melanoma

R. Maccarone, M. Fargnoli, L. Cappabianca, 2023, Cells.

TrkAIII signals endoplasmic reticulum stress to the mitochondria in neuroblastoma cells, resulting in glycolytic metabolic adaptation

L. Cappabianca, A. Farina, A. Mackay, 2017, Oncotarget.

NGF sensitizes TrkA SH-SY5Y neuroblastoma cells to TRAIL-induced apoptosis

L. Cappabianca, A. Farina, A. Mackay, 2016, Cell Death Discovery.

Constitutive autotaxin transcription by Nmyc‐amplified and non‐amplified neuroblastoma cells is regulated by a novel AP‐1 and SP‐mediated mechanism and abrogated by curcumin

A. Gulino, J. Horowitz, K. Sano, 2012, FEBS letters.

TRAIL induces pro-apoptotic crosstalk between the TRAIL-receptor signaling pathway and TrkAIII in SH-SY5Y cells, unveiling a potential therapeutic “Achilles heel” for the TrkAIII oncoprotein in neuroblastoma

L. Cappabianca, A. Farina, A. Mackay, 2016, Oncotarget.

TrkAIII expression in the thymus

I. Screpanti, A. Gulino, A. Vetuschi, 2007, Journal of Neuroimmunology.

The oncogenic neurotrophin receptor tropomyosin-related kinase variant, TrkAIII

M. Fargnoli, L. Cappabianca, A. Farina, 2018, Journal of experimental & clinical cancer research : CR.

A pilot study of alternative TrkAIII splicing in Merkel cell carcinoma: a potential oncogenic mechanism and novel therapeutic target

R. Maccarone, L. Cappabianca, A. Farina, 2019, Journal of experimental & clinical cancer research : CR.

NGF FLIPs TrkA onto the death TRAIL in neuroblastoma cells

L. Cappabianca, A. Farina, A. Mackay, 2016, Cell Death and Disease.

Molecular Characterization and Inhibition of a Novel Stress-Induced Mitochondrial Protecting Role for Misfolded TrkAIII in Human SH-SY5Y Neuroblastoma Cells

L. Cappabianca, A. Farina, A. Mackay, 2024, International journal of molecular sciences.