Stefanie Perez-Feliz

Oxytocin exerts harmful cardiac repolarization prolonging effects in drug-induced LQTS

C. Bode, M. Zehender, A. Castiglione, 2022, International journal of cardiology. Heart & vasculature.

Differential Effects of the β‐Adrenoceptor Blockers Carvedilol and Metoprolol on SQT1‐ and SQT2‐Mutant Channels

C. Bode, M. Zehender, M. Brunner, 2013, Journal of cardiovascular electrophysiology.

A computational model of rabbit geometry and ECG: Optimizing ventricular activation sequence and APD distribution

G. Seemann, A. Krafft, T. Hornyik, 2022, PloS one.

Oxytocin exerts harmful cardiac repolarization prolonging effects in drug-induced LQTS

C. Bode, M. Zehender, A. Castiglione, 2022, International journal of cardiology. Heart & vasculature.

A novel LMNA nonsense mutation causes two distinct phenotypes of cardiomyopathy with high risk of sudden cardiac death in a large five-generation family

A. Köttgen, C. Bode, P. Rainer, 2018, Europace : European pacing, arrhythmias, and cardiac electrophysiology : journal of the working groups on cardiac pacing, arrhythmias, and cardiac cellular electrophysiology of the European Society of Cardiology.

Transgenic short-QT syndrome 1 rabbits mimic the human disease phenotype with QT/action potential duration shortening in the atria and ventricles and increased ventricular tachycardia/ventricular fibrillation inducibility

J. Behrends, R. Peyronnet, C. Bode, 2018, European heart journal.

Postpartum hormones oxytocin and prolactin cause pro-arrhythmic prolongation of cardiac repolarization in long QT syndrome type 2.

G. Seemann, C. Bode, John Sorge, 2019, Europace : European pacing, arrhythmias, and cardiac electrophysiology : journal of the working groups on cardiac pacing, arrhythmias, and cardiac cellular electrophysiology of the European Society of Cardiology.

Transgenic LQT2, LQT5, and LQT2‐5 rabbit models with decreased repolarisation reserve for prediction of drug‐induced ventricular arrhythmias

C. Bode, Z. Bősze, M. Zehender, 2020, British journal of pharmacology.

KCNQ1 suppression-replacement gene therapy in transgenic rabbits with type 1 long QT syndrome.

Stefanie Perez-Feliz, C. S. J. Kim, Miriam Barbieri, 2024, European heart journal.