B. Wang

发表

Insulin-like growth factor-1 directly affects cardiac cellular remodelling via distinct pathways

H. Krum, P. Fu, Kevin W. Huang, 2021, International journal of cardiology. Heart & vasculature.

Attenuating PI3K/Akt- mTOR pathway reduces dihydrosphingosine 1 phosphate mediated collagen synthesis and hypertrophy in primary cardiac cells.

D. Liew, D. Kaye, R. Magaye, 2021, The international journal of biochemistry & cell biology.

Inhibition of Apoptosis Signal-Regulating Kinase 1 Attenuates Myocyte Hypertrophy and Fibroblast Collagen Synthesis.

H. Krum, R. Willette, Zhiliang Li, 2017, Heart, lung & circulation.

The Challenges of Stem Cell Therapy in Myocardial Infarction and Heart Failure and the Potential Strategies to Improve the Outcomes

D. Liew, Zhongmin Liu, C. Reid, 2018, Nano LIFE.

Cardiorenal syndrome: Multi‐organ dysfunction involving the heart, kidney and vasculature

D. Liew, D. Kaye, R. Magaye, 2020, British journal of pharmacology.

Myocardial infarction impairs renal function, induces renal interstitial fibrosis, and increases renal KIM-1 expression: implications for cardiorenal syndrome.

Yuan Zhang, H. Krum, D. Kelly, 2012, American journal of physiology. Heart and circulatory physiology.

Cardiorenal Syndrome: The Emerging Role of Protein-Bound Uremic Toxins

H. Krum, D. Kelly, B. Wang, 2012, Circulation research.

The Uremic Toxin Adsorbent AST-120 Abrogates Cardiorenal Injury Following Myocardial Infarction

H. Krum, D. Kelly, B. Wang, 2013, PloS one.

Cost-Effectiveness of Switching Patients With Heart Failure and Reduced Ejection Fraction to Sacubitril/Valsartan: The Australian Perspective.

D. Liew, K. Chin, E. Zomer, 2019, Heart, Lung and Circulation.

Soluble epoxide hydrolase inhibition exerts beneficial anti-remodeling actions post-myocardial infarction.

Yuan Zhang, H. Krum, D. Behm, 2013, International journal of cardiology.

Chronic urotensin II receptor antagonist treatment does not alter hypertrophy or fibrosis in a rat model of pressure-overload hypertrophy

David G. Behm, H. Krum, S. Douglas, 2010, Peptides.

Chronic urotensin-II infusion induces diastolic dysfunction and enhances collagen production in rats.

H. Krum, A. Phrommintikul, L. Tran, 2010, American journal of physiology. Heart and circulatory physiology.

Effects of a Rho kinase inhibitor on pressure overload induced cardiac hypertrophy and associated diastolic dysfunction.

H. Krum, A. Phrommintikul, L. Tran, 2008, American journal of physiology. Heart and circulatory physiology.

Peptidomic profiles of post myocardial infarction rats affinity depleted plasma using matrix-assisted laser desorption/ionization time of flight (MALDI-ToF) mass spectrometry

H. Krum, G. Rice, B. Wang, 2012, Clinical and Translational Medicine.

TRAF 2 regulates TNF and NF-κB signalling to suppress apoptosis and skin 1 inflammation independently of Sphingosine kinase-1 2 3

D. Vaux, G. Smyth, S. Nutt, 2015 .

Sequencing of modified oligonucleotides using in-source fragmentation and delayed pulsed ion extraction matrix-assisted laser desorption ionization time-of-flight mass spectrometry

B. Wang, A. Cohen, C. E. Hopkins, 1997 .

VCP746, a novel A1 adenosine receptor biased agonist, reduces hypertrophy in a rat neonatal cardiac myocyte model

Shane M. Devine, H. Krum, P. White, 2016, Clinical and experimental pharmacology & physiology.

Full title : VCP 746 , a Novel A 1 Adenosine Receptor Biased Agonist , Reduces Hypertrophy in a Rat Neonatal Cardiac Myocyte Model Short title : VCP 746 and Cardiac Myocyte Hypertrophy

Shane M. Devine, H. Krum, P. White, 2016 .

Chronic Kidney Disease-Induced Cardiac Fibrosis Is Ameliorated by Reducing Circulating Levels of a Non-Dialysable Uremic Toxin, Indoxyl Sulfate

H. Krum, D. Kelly, R. Langham, 2011, PloS one.

The hybrid molecule, VCP746, is a potent adenosine A2B receptor agonist that stimulates anti-fibrotic signalling.

Joanne Baltos, P. White, A. Christopoulos, 2016, Biochemical pharmacology.

Does indoxyl sulfate, a uraemic toxin, have direct effects on cardiac fibroblasts and myocytes?

H. Krum, D. Kelly, B. Wang, 2010, European heart journal.

Apoptosis signal-regulating kinase 1 inhibition attenuates cardiac hypertrophy and cardiorenal fibrosis induced by uremic toxins: Implications for cardiorenal syndrome

H. Krum, R. Willette, Zhiliang Li, 2017, PloS one.

Expression of Active α1B-Adrenergic Receptors in the Heart does not Alleviate Ischemic Reperfusion Injury

E. Woodcock, Xiao-Ming Gao, Xiaojun Du, 2000 .

Combined angiotensin receptor blockade and neprilysin inhibition attenuates angiotensin-II mediated renal cellular collagen synthesis.

D. Atar, H. Krum, P. Jordaan, 2015, International journal of cardiology.

Celecoxib, but not rofecoxib or naproxen, attenuates cardiac hypertrophy and fibrosis induced in vitro by angiotensin and aldosterone

H. Krum, B. Wang, R. Cheung, 2010, Clinical and experimental pharmacology & physiology.

Molecular mechanisms of protein-bound uremic toxin-mediated cardiac, renal and vascular effects: underpinning intracellular targets for cardiorenal syndrome therapy.

T. Marwick, D. Liew, D. Kaye, 2019, Toxicology letters.

Antagonists of organic anion transporters 1 and 3 ameliorate adverse cardiac remodelling induced by uremic toxin indoxyl sulfate.

H. Krum, B. Wang, S. Lekawanvijit, 2012, International journal of cardiology.

Evaluation of the effects of urotensin II and soluble epoxide hydrolase inhibitor on skin microvessel tone in healthy controls and heart failure patients.

H. Krum, L. Tran, B. Wang, 2012, Cardiovascular therapeutics.

Inositol Polyphosphate 1-Phosphatase Is a Novel Antihypertrophic Factor*

R. Hannan, E. Woodcock, J. Brown, 2002, The Journal of Biological Chemistry.

Exogenous dihydrosphingosine 1 phosphate mediates collagen synthesis in cardiac fibroblasts through JAK/STAT signalling and regulation of TIMP1.

D. Liew, D. Kaye, R. Magaye, 2020, Cellular signalling.

Early and delayed tranilast treatment reduces pathological fibrosis following myocardial infarction.

H. Krum, A. Boyle, R. Gilbert, 2013, Heart, lung & circulation.

RE: Inhibition of apoptosis signal-regulating kinase 1 might be a novel therapeutic target in the treatment of cardiorenal syndrome.

H. Krum, R. Willette, B. Jucker, 2020, International journal of cardiology.

Vitamin D supplementation and inflammatory and metabolic biomarkers in patients with knee osteoarthritis: post hoc analysis of a randomised controlled trial

F. Cicuttini, L. Blizzard, A. Wluka, 2018, British Journal of Nutrition.

The Effects of a Calcium-Rich Pre-Exercise Meal on Biomarkers of Calcium Homeostasis in Competitive Female Cyclists: A Randomised Crossover Trial

Eric C. Haakonssen, F. Cicuttini, A. Wluka, 2015, PloS one.

Efficacy of intra-articular injections of platelet-rich plasma as a symptom- and disease-modifying treatment for knee osteoarthritis - the RESTORE trial protocol

R. Buchbinder, F. Cicuttini, K. Paterson, 2018, BMC Musculoskeletal Disorders.

Cardiac fibrosis in the ageing heart: Contributors and mechanisms

D. Liew, R. Magaye, Ying-Chun Zhou, 2017, Clinical and experimental pharmacology & physiology.

Vitamin C-enriched gelatin supplementation before intermittent activity augments collagen synthesis.

K. Baar, M. Ross, B. Wang, 2017, The American journal of clinical nutrition.

Cardiorenal syndrome: Pathophysiology, preclinical models, management and potential role of uraemic toxins

H. Krum, D. Kelly, B. Wang, 2012, Clinical and experimental pharmacology & physiology.

Angiotensin Receptor Neprilysin Inhibitor LCZ696 Attenuates Cardiac Remodeling and Dysfunction After Myocardial Infarction by Reducing Cardiac Fibrosis and Hypertrophy

D. Atar, H. Krum, P. Jordaan, 2015, Circulation. Heart failure.

Potential mechanisms underlying the cardiovascular benefits of sodium glucose cotransporter 2 inhibitors: a systematic review of data from preclinical studies

C. Reid, R. Ofori-Asenso, S. Zoungas, 2018, Cardiovascular research.

Association of expression of ZNF606 gene from monocytes with the risk of coronary artery disease.

Zhiliang Li, Q. Fu, Xianbao Wang, 2018, Clinical biochemistry.

Vasculopathy in the setting of cardiorenal syndrome: roles of protein-bound uremic toxins.

H. Fan, Zhongmin Liu, Q. Fu, 2017, American journal of physiology. Heart and circulatory physiology.

The role of dihydrosphingolipids in disease

D. Liew, R. Magaye, B. Flynn, 2018, Cellular and Molecular Life Sciences.

Renin-angiotensin blockade combined with natriuretic peptide system augmentation: novel therapeutic concepts to combat heart failure.

D. Atar, H. Krum, J. Burnett, 2013, Circulation. Heart failure.

Subtotal nephrectomy accelerates pathological cardiac remodeling post-myocardial infarction: implications for cardiorenal syndrome.

H. Krum, D. Kelly, B. Wang, 2013, International journal of cardiology.

Angiotensin receptor neprilysin inhibition provides superior cardioprotection compared to angiotensin converting enzyme inhibition after experimental myocardial infarction.

H. Krum, T. von Lueder, D. Kelly, 2018, International journal of cardiology.

Chronic kidney disease with comorbid cardiac dysfunction exacerbates cardiac and renal damage†

H. Krum, D. Kelly, B. Wang, 2017, Journal of cellular and molecular medicine.

Angiotensin receptor neprilysin inhibitor LCZ696: pharmacology, pharmacokinetics and clinical development.

B. Liu, D. Liew, Ying-Chun Zhou, 2017, Future cardiology.

Design, Synthesis, and Biological Evaluation of Tetra-Substituted Thiophenes as Inhibitors of p38α MAPK

Shane M. Devine, H. Krum, D. Chalmers, 2014, ChemistryOpen.

Contribution of microRNA to pathological fibrosis in cardio-renal syndrome: impact of uremic toxins

H. Krum, F. Charchar, J. Skommer, 2015, Physiological reports.

Cardiorenal fibrosis and dysfunction in aging: Imbalance in mediators and regulators of collagen

H. Krum, J. Burnett, B. Wang, 2016, Peptides.

RE: Blockade of apoptosis signal-regulating kinase 1 ameliorates cardiac dysfunction in cardiorenal syndrome via enhancing angiogenesis.

R. Willette, B. Jucker, D. Kelly, 2020, International journal of cardiology.

Inhibition of apoptosis signal-regulating kinase 1 ameliorates left ventricular dysfunction by reducing hypertrophy and fibrosis in a rat model of cardiorenal syndrome.