N. Rief

DNA Double-Strand Break Repair of Blood Lymphocytes and Normal Tissues Analysed in a Preclinical Mouse Model: Implications for Radiosensitivity Testing

M. Löbrich, C. Rübe, C. Rübe, 2008, Clinical Cancer Research.

A Double-Strand Break Repair Defect in ATM-Deficient Cells Contributes to Radiosensitivity

Kai Rothkamm, Martin Kühne, Markus Löbrich, 2004, Cancer Research.

Efficient Rejoining of Radiation-induced DNA Double-strand Breaks in Centromeric DNA of Human Cells*

M. Löbrich, N. Rief, 2002, The Journal of Biological Chemistry.

ATM and DNA-PK Function Redundantly to Phosphorylate H2AX after Exposure to Ionizing Radiation

P. Jeggo, M. Löbrich, Kuniyoshi Iwabuchi, 2004, Cancer Research.

53BP1-dependent robust localized KAP-1 phosphorylation is essential for heterochromatic DNA double-strand break repair

A. Shibata, P. Jeggo, M. Löbrich, 2010, Nature Cell Biology.

A pathway of double-strand break rejoining dependent upon ATM, Artemis, and proteins locating to gamma-H2AX foci.

Martin Kühne, Markus Löbrich, Penny A Jeggo, 2004, Molecular cell.

In vivo formation and repair of DNA double-strand breaks after computed tomography examinations.

Michael Uder, Christian Rübe, Martin Kühne, 2005, Proceedings of the National Academy of Sciences of the United States of America.

Hyperthermia activates a subset of ataxia-telangiectasia mutated effectors independent of DNA strand breaks and heat shock protein 70 status.

M. Löbrich, T. Pandita, C. Hunt, 2007, Cancer research.

Production and characterization of a rabbit monoclonal antibody against human CDC25C phosphatase.

M. Montenarh, W. Nastainczyk, C. Götz, 1998, Hybridoma.

An additional transcript of the cdc25C gene from A431 cells encodes a functional protein.

M. Montenarh, P. Wagner, A. Jungbluth, 2000, International journal of oncology.

DNA Double-Strand Break Repair of Blood Lymphocytes and Normal Tissues Analysed in a Preclinical Mouse Model: Implications for Radiosensitivity Testing

M. Löbrich, C. Rübe, C. Rübe, 2008, Clinical Cancer Research.