Carlos Opazo

Stable Complexes Involving Acetylcholinesterase and Amyloid-β Peptide Change the Biochemical Properties of the Enzyme and Increase the Neurotoxicity of Alzheimer’s Fibrils

B. P. Doctor, N. Inestrosa, C. Opazo, 1998, The Journal of Neuroscience.

A novel functional low-density lipoprotein receptor-related protein 6 gene alternative splice variant is associated with Alzheimer's disease

Eduardo Pérez-Palma, Randall T. Moon, R. Moon, 2013, Neurobiology of Aging.

Metalloenzyme-like activity of Alzheimer's disease beta-amyloid. Cu-dependent catalytic conversion of dopamine, cholesterol, and biological reducing agents to neurotoxic H(2)O(2).

Xudong Huang, Colin L Masters, Roberto Cappai, 2002, The Journal of biological chemistry.

Copper reduction by copper binding proteins and its relation to neurodegenerative diseases

N. Inestrosa, C. Opazo, Nibaldo C. Inestrosa, 2003, Biometals.

β-Amyloid Causes Depletion of Synaptic Vesicles Leading to Neurotransmission Failure*

N. Inestrosa, J. Parodi, C. Opazo, 2009, The Journal of Biological Chemistry.

Nature of the neurotoxic membrane actions of amyloid-β on hippocampal neurons in Alzheimer's disease

Robert W. Peoples, C. Opazo, L. Aguayo, 2014, Neurobiology of Aging.