Robert W Mahley

Lipid- and receptor-binding regions of apolipoprotein E4 fragments act in concert to cause mitochondrial dysfunction and neurotoxicity.

R. Mahley, Yadong Huang, Yadong Huang, 2005, Proceedings of the National Academy of Sciences of the United States of America.

Neuron-Specific Apolipoprotein E4 Proteolysis Is Associated with Increased Tau Phosphorylation in Brains of Transgenic Mice

Tony Wyss-Coray, L. Mucke, R. Mahley, 2004, The Journal of Neuroscience.

Putting cholesterol in its place: apoE and reverse cholesterol transport.

R. Mahley, K. Weisgraber, Yadong Huang, 2006, The Journal of clinical investigation.

Genetic Variants Influencing Circulating Lipid Levels and Risk of Coronary Artery Disease

Inês Barroso, Michael Inouye, Panagiotis Deloukas, 2010, Arteriosclerosis, thrombosis, and vascular biology.

Apolipoprotein E4 Potentiates Amyloid β Peptide-induced Lysosomal Leakage and Apoptosis in Neuronal Cells*

R. Mahley, K. Weisgraber, Yadong Huang, 2002, The Journal of Biological Chemistry.

Apolipoprotein (apo) E4 enhances amyloid beta peptide production in cultured neuronal cells: apoE structure as a potential therapeutic target.

Irwin D Kuntz, Liming Dong, Elaine C Meng, 2005, Proceedings of the National Academy of Sciences of the United States of America.

Apolipoprotein E4: a causative factor and therapeutic target in neuropathology, including Alzheimer's disease.

R. Mahley, K. Weisgraber, Yadong Huang, 2006, Proceedings of the National Academy of Sciences of the United States of America.

Profile and Regulation of Apolipoprotein E (ApoE) Expression in the CNS in Mice with Targeting of Green Fluorescent Protein Gene to the ApoE Locus

David Walker, R. Mahley, Yadong Huang, 2006, The Journal of Neuroscience.