Q. Lu

发表

Guidelines for the use and interpretation of assays formonitoring autophagy (3rd edition)

Sangeeta Khare, Xin Qi, Ying Wang, 2016 .

cigarette smoke-induced endothelial apoptosis and lung injury Sustained adenosine exposure causes lung endothelial apoptosis: a possible contributor to

S. Uhlig, M. Blackburn, Q. Lu, 2015 .

Self-assembled Micelle Interfering RNA for Effective and Safe Targeting of Dysregulated Genes in Pulmonary Fibrosis*

Woo Seok Kim, Won-Kyung Cho, Kyuhong Lee, 2016, The Journal of Biological Chemistry.

Effects of cigarette smoke on pulmonary endothelial cells.

Q. Lu, S. Rounds, Eric R Gottlieb, 2018, American journal of physiology. Lung cellular and molecular physiology.

Cigarette smoke alters lung vascular permeability and endothelial barrier function (2017 Grover Conference Series)

Q. Lu, S. Rounds, 2018, Pulmonary circulation.

Alda‐1 Protects Against Acrolein‐Induced Acute Lung Injury and Endothelial Barrier Dysfunction

H. Yao, Q. Lu, S. Rounds, 2017, American journal of respiratory cell and molecular biology.

Blockade of equilibrative nucleoside transporter 1/2 protects against Pseudomonas aeruginosa–induced acute lung injury and NLRP3 inflammasome activation

A. Ayala, M. Blackburn, Zhengke Wang, 2020, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

smoke-induced endothelial apoptosis and lung injury apoptosis: a possible contributor to cigarette Sustained adenosine exposure causes lung endothelial

M. Blackburn, Q. Lu, Mesias Pedroza, 2016 .

Sustained adenosine exposure causes lung endothelial apoptosis: a possible contributor to cigarette smoke-induced endothelial apoptosis and lung injury.

M. Blackburn, Q. Lu, Mesias Pedroza, 2013, American journal of physiology. Lung cellular and molecular physiology.

Sustained adenosine exposure causes lung endothelial barrier dysfunction via nucleoside transporter-mediated signaling.

M. Blackburn, Q. Lu, Mesias Pedroza, 2012, American journal of respiratory cell and molecular biology.

Oxidative-Nitrosative Stress and Post-Translational Protein Modifications : Implications to Lung Structure – Function Relations Inhibition of ICMT Induces Endothelial Cell Apoptosis through GRP 94

Q. Lu, S. Rounds, J. Newton, 2007 .

Inhibition of ICMT induces endothelial cell apoptosis through GRP94.

Q. Lu, S. Rounds, J. Newton, 2007, American journal of respiratory cell and molecular biology.

Transforming growth factor- (cid:1) 1 causes pulmonary microvascular endothelial cell apoptosis via ALK5

Q. Lu, S. Rounds, E. Harrington, 2022 .

Focal adhesion kinase and endothelial cell apoptosis.

Q. Lu, S. Rounds, 2012, Microvascular research.

Mitochondrial Fission Mediated Cigarette Smoke-induced Pulmonary Endothelial Injury.

Zhengke Wang, Junsuk Ko, Q. Lu, 2020, American journal of respiratory cell and molecular biology.

Transforming growth factor-1 protects against pulmonary artery endothelial cell apoptosis via ALK 5

Q. Lu, 2008 .

Effect of (cid:2) 7 nicotinic acetylcholine receptor activation on cardiac ﬁbroblasts: a mechanism underlying RV ﬁbrosis associated with cigarette smoke exposure

R. Clements, H. Chichger, S. Dudley, 2017 .

Apoptosis and lung injury.

Q. Lu, S. Rounds, E. Harrington, 2005, The Keio journal of medicine.

Cigarette smoke-induced lung endothelial apoptosis and emphysema are associated with impairment of FAK and eIF2α.

C. Lee, Q. Lu, S. Rounds, 2014, Microvascular research.

Chitinase 3 like 1 contributes to the development of pulmonary vascular remodeling in pulmonary hypertension

G. Baird, C. Ventetuolo, C. Lee, 2022, JCI insight.

Cigarette smoke causes lung vascular barrier dysfunction via oxidative stress-mediated inhibition of RhoA and focal adhesion kinase.

Q. Lu, S. Rounds, J. Newton, 2011, American journal of physiology. Lung cellular and molecular physiology.

NLRP3 inflammasome activation in cigarette smoke priming for Pseudomonas aeruginosa-induced acute lung injury

F. Sellke, L. Ware, A. Ayala, 2022, Redox biology.

Cigarette Smoke-Induced Lung Endothelial and Alveolar Epithelial Injury Cigarette smoke causes lung vascular barrier dysfunction via oxidative stress-mediated inhibition of RhoA and focal adhesion kinase

Q. Lu, S. Rounds, J. Newton, 2011 .

Pulmonary endothelial cell signaling and function.

B. Casserly, Q. Lu, S. Rounds, 2008, Transactions of the American Clinical and Climatological Association.

Cigarette Smoke Disrupted Lung Endothelial Barrier Integrity and Increased Susceptibility to Acute Lung Injury via Histone Deacetylase 6.

Junsuk Ko, Q. Lu, S. Rounds, 2016, American journal of respiratory cell and molecular biology.

Sustained adenosine exposure causes endothelial mitochondrial dysfunction via equilibrative nucleoside transporters

Junsuk Ko, Q. Lu, S. Rounds, 2020, Pulmonary circulation.

Essential oil from Fructus Alpinia zerumbet (fruit of Alpinia zerumbet (Pers.) Burtt.et Smith) protected against aortic endothelial cell injury and inflammation in vitro and in vivo.

Yan-Yan Zhang, Xiang-chun Shen, Q. Lu, 2019, Journal of ethnopharmacology.

Regulation of Corneal Endothelial Cell Proliferation

T. Fuchsluger, Q. Lu, U. Jurkunas, 2010 .

Isoprenylcysteine Carboxyl Methyltransferase Modulates Endothelial Monolayer Permeability: Involvement of RhoA Carboxyl Methylation

C. Hai, T. Hirase, Q. Lu, 2004, Circulation research.

Pulmonary Endothelial Cell Apoptosis in Emphysema and Acute Lung Injury.

Q. Lu, S. Rounds, E. Chambers, 2018, Advances in anatomy, embryology, and cell biology.

Adenosine protected against pulmonary edema through transporter- and receptor A2-mediated endothelial barrier enhancement.

B. Casserly, M. Blackburn, Michael R Blackburn, 2010, American journal of physiology. Lung cellular and molecular physiology.

Transforming growth factor- (cid:1) 1 causes pulmonary microvascular endothelial cell apoptosis via ALK5

Q. Lu, S. Rounds, E. Harrington, 2022 .

Transforming growth factor- (cid:1) 1 protects against pulmonary artery endothelial cell apoptosis via ALK5

Q. Lu, 2022 .

Brg1 inhibits E-cadherin expression in lung epithelial cells and disrupts epithelial integrity

S. Cai, J. Dai, E. Liu, 2017, Journal of Molecular Medicine.

MicroRNA-98 reduces nerve growth factor expression in nicotine-induced airway remodeling

B. Kang, T. Nyunoya, C. Hart, 2020, The Journal of Biological Chemistry.

Double-hit mouse model of cigarette smoke priming for acute lung injury.

A. Ayala, Zhengke Wang, Joanne L. Lomas-Neira, 2017, American journal of physiology. Lung cellular and molecular physiology.

Isoprenylcysteine carboxyl methyltransferase activity modulates endothelial cell apoptosis.

Kerri‐Lynn Sheahan, Q. Lu, S. Rounds, 2003, Molecular biology of the cell.

activation and subsequent RhoA activation barrier dysfunction involves Smad2-dependent p38

M. Hatziapostolou, W. Zapol, Bo Liu, 2015 .

Transforming growth factor-beta1-induced endothelial barrier dysfunction involves Smad2-dependent p38 activation and subsequent RhoA activation.

Q. Lu, S. Rounds, E. Harrington, 2006, Journal of applied physiology.

Carboxyl Methylation of Small GTPases and Endothelial Cell Function

Q. Lu, S. Rounds, E. Harrington, 2005 .

Where There's Smoke, There's Fire.

Q. Lu, S. Rounds, 2020, Chest.

Adenosine protected against pulmonary edema through transporter- and receptor A2-mediated endothelial barrier enhancement.

B. Casserly, M. Blackburn, R. Warburton, 2010, American journal of physiology. Lung cellular and molecular physiology.

Alterations in molecular chaperones and eIF2alpha during lung endothelial cell apoptosis.

Q. Lu, S. Rounds, J. Newton, 2010, American journal of physiology. Lung cellular and molecular physiology.