S. Ramanadham

发表

Guidelines for the use and interpretation of assays formonitoring autophagy (3rd edition)

Sangeeta Khare, Xin Qi, Ying Wang, 2016 .

Group VIA Phospholipase A2 (iPLA2β) Modulates Bcl-x 5′-Splice Site Selection and Suppresses Anti-apoptotic Bcl-x(L) in β-Cells*

C. Chalfant, Margaret A. Park, D. Wijesinghe, 2015, The Journal of Biological Chemistry.

Characterization of N-terminal processing of group VIA phospholipase A2 and of potential cleavage sites of amyloid precursor protein constructs by automated identification of signature peptides in LC/MS/MS analyses of proteolytic digests

John Turk, Ilan Vidavsky, Haowei Song, 2004, Journal of the American Society for Mass Spectrometry.

Metabolic Effects of Selective Deletion of Group VIA Phospholipase A2 from Macrophages or Pancreatic Islet Beta-Cells

Haowei Song, J. Turk, X. Lei, 2020, Biomolecules.

Male Mice That Do Not Express Group VIA Phospholipase A2 Produce Spermatozoa with Impaired Motility and Have Greatly Reduced Fertility*

David J. Miller, J. Turk, Zhongming Ma, 2004, Journal of Biological Chemistry.

Insulin Secretory Responses and Phospholipid Composition of Pancreatic Islets from Mice That Do Not Express Group VIA Phospholipase A2 and Effects of Metabolic Stress on Glucose Homeostasis*

Haowei Song, J. Turk, A. Bohrer, 2006, Journal of Biological Chemistry.

Mass spectrometric characterization of arachidonate-containing plasmalogens in human pancreatic islets and in rat islet beta-cells and subcellular membranes.

R. Gross, J. Turk, A. Bohrer, 1993, Biochemistry.

Effects of Stable Suppression of Group VIA Phospholipase A2 Expression on Phospholipid Content and Composition, Insulin Secretion, and Proliferation of INS-1 Insulinoma Cells*

S. Zhang, J. Turk, A. Bohrer, 2006, Journal of Biological Chemistry.

Phospholipid hydrolysis in pancreatic islet beta cells and the regulation of insulin secretion

R. Gross, J. Turk, S. Ramanadham, 1996 .

Mass spectrometric identification and quantitation of arachidonate-containing phospholipids in pancreatic islets: prominence of plasmenylethanolamine molecular species.

R. Gross, J. Turk, A. Bohrer, 1993, Biochemistry.

Islet complex lipids: involvement in the actions of group VIA calcium-independent phospholipase A(2) in beta-cells.

S. Zhang, Haowei Song, F. Hsu, 2004, Diabetes.

The expression and function of a group VIA calcium-independent phospholipase A2 (iPLA2β) in β-cells

J. Turk, S. Ramanadham, 2004 .

Involvement in the actions of group VIA calcium-independent phospholipase A2 in β-cells

Haowei Song, F. Hsu, J. Turk, 2004 .

Studies of phospholipid metabolism, proliferation, and secretion of stably transfected insulinoma cells that overexpress group VIA phospholipase A2

F. Hsu, J. Turk, Zhongming Ma, 2001, Lipids.

Toxicological aspects of vanadyl sulphate on diabetic rats: effects on vanadium levels and pancreatic B-cell morphology.

J. McNeill, S. Ramanadham, L. Vian, 1990, Pharmacology & toxicology.

Quantification of distinct molecular species of the 2-lyso metabolite of platelet-activating factor by gas chromatography-negative-ion chemical ionization mass spectrometry.

J. Turk, M. Mangino, A. Bohrer, 1992, Journal of chromatography.

Chronic effects of streptozotocin diabetes on myocardial sensitivity in the rat

S. Ramanadham, T. Tenner, 1986, Diabetologia.

ω-Conotoxin inhibits glucose- and arachidonic acid-induced rises in intracellular [Ca2+] in rat pancreatic islet β-cells

J. Turk, S. Ramanadham, 1994 .

Targeting Acid Ceramidase Inhibits Glioblastoma Cell Migration through Decreased AKT Signaling

Emily R. Gordon, S. Cooper, A. Hjelmeland, 2022, Cells.

Dysfunctional mitochondrial bioenergetics and oxidative stress in Akita / Ins 2-derived-cells

S. Barnes, K. Mitra, Jianhua Zhang, 2013 .

Group VIA Ca 2þ -independent phospholipase A2 (iPLA2b) and its role in b-cell programmed cell death

X. Lei, S. Ramanadham, S. Barbour, 2010 .

A link between endoplasmic reticulum stress‐induced β‐cell apoptosis and the group VIA Ca2+‐independent phospholipase A2 (iPLA2β)

S. Zhang, X. Lei, S. Ramanadham, 2010, Diabetes, obesity & metabolism.

A link between endoplasmic reticulum stress-induced β-cell apoptosis and the group VIA Ca 2+ -independent

X. Lei, S. Ramanadham, S. Barbour, 2010 .

Group VIA Ca2+-independent phospholipase A2 (iPLA2beta) and its role in beta-cell programmed cell death.

X. Lei, S. Ramanadham, S. Barbour, 2010, Biochimie.

The group VIA calcium-independent phospholipase A2 participates in ER stress-induced INS-1 insulinoma cell apoptosis by promoting ceramide generation via hydrolysis of sphingomyelins by neutral sphingomyelinase.

Haowei Song, A. Bohrer, X. Lei, 2007, Biochemistry.

Attenuated Free Cholesterol Loading-induced Apoptosis but Preserved Phospholipid Composition of Peritoneal Macrophages from Mice That Do Not Express Group VIA Phospholipase A2*

J. Turk, C. Semenkovich, Yankun Li, 2007, Journal of Biological Chemistry.

Effects of biological oxidants on the catalytic activity and structure of group VIA phospholipase A2.

Haowei Song, J. Turk, S. Ramanadham, 2006, Biochemistry.

Evidence for Proteolytic Processing and Stimulated Organelle Redistribution of iPLA2β

Shenmin Zhang, Haowei Song, J. Turk, 2010 .

Evidence for proteolytic processing and stimulated organelle redistribution of iPLA(2)beta.

S. Zhang, Haowei Song, J. Turk, 2010, Biochimica et biophysica acta.

A bromoenol lactone suicide substrate inactivates group VIA phospholipase A2 by generating a diffusible bromomethyl keto acid that alkylates cysteine thiols.

Haowei Song, F. Hsu, J. Turk, 2006, Biochemistry.

Beta-cell calcium-independent group VIA phospholipase A(2) (iPLA(2)beta): tracking iPLA(2)beta movements in response to stimulation with insulin secretagogues in INS-1 cells.

S. Zhang, J. Turk, Zhongming Ma, 2004, Diabetes.

Pancreatic islets and insulinoma cells express a novel isoform of group VIA phospholipase A2 (iPLA2 beta) that participates in glucose-stimulated insulin secretion and is not produced by alternate splicing of the iPLA2 beta transcript.

S. Zhang, C. Newgard, G. A. Grant, 2003, Biochemistry.

Apoptosis of insulin-secreting cells induced by endoplasmic reticulum stress is amplified by overexpression of group VIA calcium-independent phospholipase A2 (iPLA2 beta) and suppressed by inhibition of iPLA2 beta.

Haowei Song, F. Hsu, J. Turk, 2004, Biochemistry.

Studies of the Role of Group VI Phospholipase A2 in Fatty Acid Incorporation, Phospholipid Remodeling, Lysophosphatidylcholine Generation, and Secretagogue-induced Arachidonic Acid Release in Pancreatic Islets and Insulinoma Cells*

F. Hsu, J. Turk, Zhongming Ma, 1999, The Journal of Biological Chemistry.

Studies of Insulin Secretory Responses and of Arachidonic Acid Incorporation into Phospholipids of Stably Transfected Insulinoma Cells That Overexpress Group VIA Phospholipase A2(iPLA2β) Indicate a Signaling Rather Than a Housekeeping Role for iPLA2β*

F. Hsu, J. Turk, Zhongming Ma, 2001, The Journal of Biological Chemistry.

Human Pancreatic Islets Express mRNA Species Encoding Two Distinct Catalytically Active Isoforms of Group VI Phospholipase A2 (iPLA2) That Arise from an Exon-skipping Mechanism of Alternative Splicing of the Transcript from the iPLA2 Gene on Chromosome 22q13.1*

J. Turk, Zhongming Ma, W. Nowatzke, 1999, The Journal of Biological Chemistry.

Lipid mediators and biomarkers associated with type 1 diabetes development

Alexander J. Nelson, C. Mathews, M. Hessner, 2020, JCI insight.

Matthew J. Silva, K. Yarasheski, B. Christiansen, 2008, The American journal of pathology.

Matthew J. Silva, K. Yarasheski, B. Christiansen, 2008 .

Isolation of human skeletal muscle myosin heavy chain and actin for measurement of fractional synthesis rates.

K. Yarasheski, D. Hasten, G. Morris, 1998, American journal of physiology. Endocrinology and metabolism.

Isolation of human skeletal muscle myosin heavy chain and actin for measurement of fractional synthesis rates.

K. Yarasheski, D. Hasten, G. Morris, 1998, The American journal of physiology.

iPLA2β and its role in male fertility, neurological disorders, metabolic disorders, and inflammation.

Alexander J. Nelson, J. Turk, X. Lei, 2019, Biochimica et biophysica acta. Molecular and cell biology of lipids.

Pancreatic Islet Cells Express a Family of Inwardly Rectifying K+ Channel Subunits Which Interact to Form G-protein-activated Channels (*)

L. Salkoff, C. Nichols, A. Permutt, 1995, The Journal of Biological Chemistry.

Extracellular vesicles in β cell biology: Role of lipids in vesicle biogenesis, cargo, and intercellular signaling

R. Mirmira, E. A. Phelps, E. Nakayasu, 2022, Molecular metabolism.

Role of calcium-independent phospholipase A(2)β in human pancreatic islet β-cell apoptosis.

S. Zhang, A. Bohrer, X. Lei, 2012, American journal of physiology. Endocrinology and metabolism.

Enhanced in vivo sensitivity of vanadyl-treated diabetic rats to insulin.

J. McNeill, S. Ramanadham, J. Serrano, 1990, Canadian journal of physiology and pharmacology.

Sustained prevention of myocardial and metabolic abnormalities in diabetic rats following withdrawal from oral vanadyl treatment.

J. McNeill, R. Brownsey, S. Ramanadham, 1989, Metabolism: clinical and experimental.

Myocardial and metabolic abnormalities in streptozotocin-diabetic Wistar and Wistar-Kyoto rats.

J. McNeill, S. Ramanadham, A. Doroudian, 1990, The Canadian journal of cardiology.

Ca2+-Independent Phospholipase A2β-Derived PGE2 Contributes to Osteogenesis

G. Clines, X. Lei, S. Ramanadham, 2021, Prostaglandins & Other Lipid Mediators.

Stimulation of insulin secretion and associated nuclear accumulation of iPLA(2)beta in INS-1 insulinoma cells.

S. Zhang, J. Turk, Zhongming Ma, 2002, American journal of physiology. Endocrinology and metabolism.

Polarization of Macrophages toward M2 Phenotype Is Favored by Reduction in iPLA2β (Group VIA Phospholipase A2)*

Alexander J. Nelson, J. Turk, Jason W Ashley, 2016, The Journal of Biological Chemistry.

Electrospray ionization mass spectrometric analyses of phospholipids from INS-1 insulinoma cells: comparison to pancreatic islets and effects of fatty acid supplementation on phospholipid composition and insulin secretion.

J. Turk, F. Hsu, A. Bohrer, 2000, Biochimica et biophysica acta.

Rat and human pancreatic islet cells contain a calcium ion independent phospholipase A2 activity selective for hydrolysis of arachidonate which is stimulated by adenosine triphosphate and is specifically localized to islet beta-cells.

R. Gross, J. Turk, X. Han, 1993, Biochemistry.

Alterations in the myocardial β-adrenoceptor system of streptozotocin-diabetic rats

S. Ramanadham, T. Tenner, 1987 .

Δ6-, stearoyl CoA-, and Δ5-desaturase enzymes are expressed in β-cells and are altered by increases in exogenous PUFA concentrations

F. Hsu, J. Turk, Zhongming Ma, 2002 .

Arachidonic acid induces an increase in the cytosolic calcium concentration in single pancreatic islet beta cells.

R. Gross, J. Turk, S. Ramanadham, 1992, Biochemical and biophysical research communications.

HIV-protease inhibitors suppress skeletal muscle fatty acid oxidation by reducing CD36 and CPT1 fatty acid transporters.

K. Yarasheski, S. Zhang, X. Lei, 2010, Biochimica et biophysica acta.

Vitamin D and calcium-sensing receptor polymorphisms differentially associate with resting energy expenditure in peripubertal children

J. Ard, K. Casazza, M. Bray, 2013, Journal of Bone and Mineral Metabolism.

Biochemical evidence for nitric oxide formation from streptozotocin in isolated pancreatic islets.

M. Mcdaniel, J. Corbett, J. Turk, 1993, Biochemical and biophysical research communications.

Alterations in β-Cell Sphingolipid Profile Associated with ER Stress and iPLA2β: Another Contributor to β-Cell Apoptosis in Type 1 Diabetes

C. Chalfant, X. Lei, S. Ramanadham, 2021, Molecules.

Carbonylation Induces Heterogeneity in Cardiac Ryanodine Receptor Function in Diabetes Mellitus

S. Kutty, R. Nagai, Jaipaul Singh, 2012, Molecular Pharmacology.

CARBONYLATION INDUCES HETEROGENEITY IN CARDIAC RYANODINE RECEPTORS (RyR2) FUNCTION DURING DIABETES

Német, S. Kutty, R. Nagai, 2012 .

Spontaneous development of ER stress that can lead to diabetes mellitus is associated with higher calcium-independent phospholipase A2 (iPLA2beta) expression: A role for regulation by SREBP-1

F. R. Papa, S. Zhang, A. Bohrer, 2009 .

Selective expression of an arachidonate 12-lipoxygenase by pancreatic islet beta-cells.

M. Holtzman, V. Shannon, J. Turk, 1992, The American journal of physiology.

Meta-analysis refinement of plasma extracellular vesicle composition identifies proplatelet basic protein as a signaling messenger in type 1 diabetes

Samuel H. Payne, T. Metz, S. Payne, 2022, bioRxiv.

Evidence of contribution of iPLA2β-mediated events during islet β-cell apoptosis due to proinflammatory cytokines suggests a role for iPLA2β in T1D development.

A. Bohrer, K. Bidasee, X. Lei, 2014, Endocrinology.

High glucose exposure promotes activation of protein phosphatase 2A in rodent islets and INS-1 832/13 β-cells by increasing the posttranslational carboxylmethylation of its catalytic subunit.

A. Kowluru, B. Wadzinski, S. Ramanadham, 2014, Endocrinology.

Macrophage polarization is linked to Ca2+-independent phospholipase A2β-derived lipids and cross-cell signaling in mice[S]

Alexander J. Nelson, C. Chalfant, Margaret A. Park, 2019, Journal of Lipid Research.

Cloning and expression of a group IV cytosolic Ca2+-dependent phospholipase A2 from rat pancreatic islets. Comparison of the expressed activity with that of an islet group VI cytosolic Ca2+-independent phospholipase A2.

J. Turk, S. Ramanadham, Z. Hu, 1998, Biochimica et biophysica acta.

Glucose-responsitivity and expression of an ATP-stimulatable, Ca(2+)-independent phospholipase A2 enzyme in clonal insulinoma cell lines.

J. Turk, A. Bohrer, S. Ramanadham, 1997, Biochimica et biophysica acta.

Skeletal muscle group VIA phospholipase A2 (iPLA2beta): expression and role in fatty acid oxidation.

S. Zhang, J. Turk, S. Ramanadham, 2008, Biochemistry.

Type IB secretory phospholipase A2 is contained in insulin secretory granules of pancreatic islet beta-cells and is co-secreted with insulin from glucose-stimulated islets.

J. Turk, S. Zhang, H. Arita, 1998, Biochimica et biophysica acta.

Inhibition of arachidonate release by secretagogue-stimulated pancreatic islets suppresses both insulin secretion and the rise in beta-cell cytosolic calcium ion concentration.

R. Gross, J. Turk, X. Han, 1993, Biochemistry.

Delta6-, Stearoyl CoA-, and Delta5-desaturase enzymes are expressed in beta-cells and are altered by increases in exogenous PUFA concentrations.

S. Zhang, F. Hsu, J. Turk, 2002, Biochimica et Biophysica Acta.

Novel effects of Brefeldin A (BFA) in signaling through the insulin receptor (IR) pathway and regulating FoxO1-mediated transcription

W. Garvey, E. Sztul, Qinglan Liu, 2014, Cellular logistics.

Arachidonic acid metabolism in isolated pancreatic islets. VI. Carbohydrate insulin secretagogues must be metabolized to induce eicosanoid release.

J. Turk, A. Bohrer, S. Ramanadham, 1992, Biochimica et biophysica acta.

The expression and function of a group VIA calcium-independent phospholipase A2 (iPLA2beta) in beta-cells.

J. Turk, S. Ramanadham, 2004, Canadian journal of physiology and pharmacology.

Effects of arachidonyltrifluoromethyl ketone on cytosolic [Ca2+] in HIT insulinoma cells.

J. Turk, S. Ramanadham, D. Stickle, 1997, Journal of lipid mediators and cell signalling.

Inhibition of Ca-Independent Phospholipase A2b (iPLA2b) Ameliorates Islet Infiltration and Incidence of Diabetes in NOD Mice

V. Magrioti, G. Kokotos, X. Lei, 2015 .

Inhibition of Ca2+-Independent Phospholipase A2β (iPLA2β) Ameliorates Islet Infiltration and Incidence of Diabetes in NOD Mice

V. Magrioti, G. Kokotos, X. Lei, 2014, Diabetes.

-Cell Calcium-Independent Group VIA Phospholipase Tracking iPLA 2 (cid:1) Movements in Response to Stimulation With Insulin Secretagogues in INS-1 Cells

J. Turk, Zhongming Ma, S. Ramanadham, 2003 .

Effects of vanadyl derivatives on animal models of diabetes

J. McNeill, S. Ramanadham, J. Serrano, 1992, Molecular and Cellular Biochemistry.

Promiscuity of the catalytic Sec7 domain within the guanine nucleotide exchange factor GBF1 in ARF activation, Golgi homeostasis, and effector recruitment

E. Sztul, G. Belov, E. Viktorova, 2019, Molecular biology of the cell.

Long-Term Effects of Vanadyl Treatment on Streptozocin-Induced Diabetes in Rats

R. Pederson, J. McNeill, A. Buchan, 1989, Diabetes.

β-Lactones: A Novel Class of Ca2+-Independent Phospholipase A2 (Group VIA iPLA2) Inhibitors with the Ability To Inhibit β-Cell Apoptosis.

Varnavas D. Mouchlis, E. Dennis, V. Magrioti, 2019, Journal of medicinal chemistry.

Characterization of an ATP-stimulatable Ca(2+)-independent phospholipase A2 from clonal insulin-secreting HIT cells and rat pancreatic islets: a possible molecular component of the beta-cell fuel sensor.

R. Gross, J. Turk, S. Ramanadham, 1994, Biochemistry.

Group VIA Phospholipase A2 Forms a Signaling Complex with the Calcium/Calmodulin-dependent Protein Kinase IIβ Expressed in Pancreatic Islet β-Cells*

R. Gross, J. Turk, Zhongming Ma, 2005, Journal of Biological Chemistry.

Oral vanadyl sulfate in treatment of diabetes mellitus in rats.

J. McNeill, R. Brownsey, S. Ramanadham, 1989, The American journal of physiology.

A pyrrolidine-based specific inhibitor of cytosolic phospholipase A(2)alpha blocks arachidonic acid release in a variety of mammalian cells.

C. Leslie, M. Gelb, J. Turk, 2001, Biochimica et biophysica acta.

Potentiation of barbiturate- and halothane-induced hypnosis after probenecid or sulfinpyrazone pretreatment

S. Ramanadham, W. Lyness, 1982, Neuropharmacology.

Calcium-independent phospholipases A2 and their roles in biological processes and diseases

X. Lei, Jason W Ashley, S. Ramanadham, 2015, Journal of Lipid Research.

Genetic modulation of islet β-cell iPLA2β expression provides evidence for its impact on β-cell apoptosis and autophagy

J. Turk, A. Bohrer, X. Lei, 2013, Islets.

Sphingolipid Metabolism in Glioblastoma and Metastatic Brain Tumors: A Review of Sphingomyelinases and Sphingosine-1-Phosphate

A. Hjelmeland, S. Ramanadham, Tomader Ali, 2020, Biomolecules.

Chronotropic actions of bPTH-(1-34) in the right atrium of the rat.

S. Ramanadham, P. Pang, T. Tenner, 1983, Canadian journal of physiology and pharmacology.

Arachidonic acid release from aortic smooth muscle cells induced by [Arg8]vasopressin is largely mediated by calcium-independent phospholipase A2.

R. Gross, J. Turk, K. Brown, 1993, The Journal of biological chemistry.

Alterations in aortic and tail artery reactivity to agonists after streptozotocin treatment.

S. Ramanadham, W. Lyness, T. Tenner, 1984, Canadian journal of physiology and pharmacology.

The Impact of the Ca2+-Independent Phospholipase A2β (iPLA2β) on Immune Cells

X. Lei, S. Ramanadham, A. AlMutairi, 2021, Biomolecules.

Reconstitution of membrane fusion between pancreatic islet secretory granules and plasma membranes: catalysis by a protein constituent recognized by monoclonal antibodies directed against glyceraldehyde-3-phosphate dehydrogenase.

R. Gross, J. Turk, X. Han, 1998, Biochimica et biophysica acta.

Evidence for association of an ATP-stimulatable Ca(2+)-independent phospholipase A2 from pancreatic islets and HIT insulinoma cells with a phosphofructokinase-like protein.

R. Gross, J. Turk, J. Wang, 1996, Biochemistry.

Amplification of Insulin Secretion by Lipid Messengers

R. Gross, J. Turk, S. Ramanadham, 1993, Diabetes.

Saturated Hydroxy Fatty Acids Exhibit a Cell Growth Inhibitory Activity and Suppress the Cytokine-Induced β-Cell Apoptosis.

G. Kokotos, A. Kokotos, X. Lei, 2020, Journal of medicinal chemistry.

Characterization of expression of phosphofructokinase isoforms in isolated rat pancreatic islets and purified beta cells and cloning and expression of the rat phosphofructokinase-A isoform.

J. Ladenson, J. Turk, K. Kempe, 1996, Biochimica et biophysica acta.

Characterization of FKGK18 as Inhibitor of Group VIA Ca2+-Independent Phospholipase A2 (iPLA2β): Candidate Drug for Preventing Beta-Cell Apoptosis and Diabetes

V. Magrioti, G. Kokotos, J. Mobley, 2013, PloS one.

Spontaneous Development of Endoplasmic Reticulum Stress That Can Lead to Diabetes Mellitus Is Associated with Higher Calcium-independent Phospholipase A2 Expression

S. Zhang, F. R. Papa, A. Bohrer, 2009, The Journal of Biological Chemistry.

Electrospray Ionization/Mass Spectrometric Analyses of Human Promonocytic U937 Cell Glycerolipids and Evidence That Differentiation Is Associated with Membrane Lipid Composition Changes That Facilitate Phospholipase A2 Activation*

F. Hsu, J. Turk, Zhongming Ma, 2000, The Journal of Biological Chemistry.

Nonspecific supersensitivity induced by reserpine in guinea pig cardiac ventricle tissue.

J. Young, S. Ramanadham, T. Tenner, 1988, The Journal of pharmacology and experimental therapeutics.

Electrospray ionization mass spectrometric analyses of changes in tissue phospholipid molecular species during the evolution of hyperlipidemia and hyperglycemia in Zucker diabetic fatty rats

K. Yarasheski, F. Hsu, J. Turk, 2000, Lipids.

Calcium-independent Phospholipase A2 (iPLA2β)-mediated Ceramide Generation Plays a Key Role in the Cross-talk between the Endoplasmic Reticulum (ER) and Mitochondria during ER Stress-induced Insulin-secreting Cell Apoptosis*

A. Bohrer, X. Lei, S. Ramanadham, 2008, Journal of Biological Chemistry.

Interleukin-1 Reduces the Glycolytic Utilization of Glucose by Pancreatic Islets and Reduces Glucokinase mRNA Content and Protein Synthesis by a Nitric Oxide-dependent Mechanism*

D. Kipnis, J. Turk, Zhongming Ma, 1997, The Journal of Biological Chemistry.

[Vanadium and diabetes].

J. McNeill, S. Ramanadham, J. Serrano, 1998, Journees annuelles de diabetologie de l'Hotel-Dieu.

Protease inhibitors used in the treatment of HIV+ induce beta-cell apoptosis via the mitochondrial pathway and compromise insulin secretion.

K. Yarasheski, S. Zhang, X. Lei, 2009, American journal of physiology. Endocrinology and metabolism.

Interleukin-1 Enhances Pancreatic Islet Arachidonic Acid 12-Lipoxygenase Product Generation by Increasing Substrate Availability through a Nitric Oxide-dependent Mechanism (*)

M. Mcdaniel, J. Corbett, R. Gross, 1996, The Journal of Biological Chemistry.

Effect of insulin replacement on streptozotocin-induced effects in the rat heart.

S. Ramanadham, T. Tenner, P. Decker, 1983, Life sciences.

Erratum to: Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356

Sara W. Bird, Erinna F. Lee, Michael D. Kim, 2016 .

Analyses of Calcium-Independent Phospholipase A2beta (iPLA2β) in Biological Systems.

S. Ramanadham, S. Barbour, 2017, Methods in enzymology.

Editorial :Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition). DOI: 10.1080/15548627.2015.1100356;WOS:000373595400001; 2-s2.0-85013763791&;PMID: 26799652

Lee, Duchen, Izzo, 2016 .

Enhanced in vivo sensitivity of vanadyl-treated diabetic rats to insulin

J. McNeill, S. Ramanadham, J. Serrano, 1990 .

Pancreatic Islets Express a Ca2+-independent Phospholipase A2 Enzyme That Contains a Repeated Structural Motif Homologous to the Integral Membrane Protein Binding Domain of Ankyrin*

J. Ladenson, J. Turk, Zhongming Ma, 1997, The Journal of Biological Chemistry.

Mass Spectrometric Evidence That Agents That Cause Loss of Ca 2؉ from Intracellular Compartments Induce Hydrolysis of Arachidonic Acid from Pancreatic Islet Membrane Phospholipids by a Mechanism That Does Not Require a Rise in Cytosolic Ca 2؉ Concentration*

J. Turk, F. Hsu, A. Bohrer, 2022 .

Electrospray ionization mass spectrometric analyses of phospholipids from rat and human pancreatic islets and subcellular membranes: comparison to other tissues and implications for membrane fusion in insulin exocytosis.

J. Turk, F. Hsu, A. Bohrer, 1998, Biochemistry.

Dysfunctional mitochondrial bioenergetics and oxidative stress in Akita(+/Ins2)-derived β-cells.

S. Barnes, K. Mitra, Jianhua Zhang, 2013, American journal of physiology. Endocrinology and metabolism.

HIV-protease inhibitors induce expression of suppressor of cytokine signaling-1 in insulin-sensitive tissues and promote insulin resistance and type 2 diabetes mellitus.

Sheng Zhang, K. Yarasheski, A. Shalev, 2008, American journal of physiology. Endocrinology and metabolism.

Loss of Brain Angiogenesis Inhibitor-3 (BAI3) G-Protein Coupled Receptor in Mice Regulates Adaptive Thermogenesis by Enhancing Energy Expenditure

M. Latimer, A. Challa, S. Ramanadham, 2023, Metabolites.

Deletion of Gdf15 reduces ER stress-induced beta cell apoptosis and diabetes.

A. Shalev, T. Grayson, Se-Jin Lee, 2022, Endocrinology.

Alterations in the myocardial beta-adrenoceptor system of streptozotocin-diabetic rats.

S. Ramanadham, T. Tenner, 1987, European journal of pharmacology.

Mass spectrometric evidence that agents that cause loss of Ca2+ from intracellular compartments induce hydrolysis of arachidonic acid from pancreatic islet membrane phospholipids by a mechanism that does not require a rise in cytosolic Ca2+ concentration.

A. Bohrer, S. Ramanadham, F. Hsu, 1998, Endocrinology.

HIV-protease inhibitors suppress skeletal muscle fatty acid oxidation by reducing CD36 and CPT1 fatty acid transporters.

K. Yarasheski, S. Zhang, X. Lei, 2010, Biochimica et biophysica acta.