N. Safikhan

发表

CD248 enhances tissue factor procoagulant function, promoting arterial and venous thrombosis in mouse models

W. Ruf, E. Conway, A. Mast, 2021, Journal of thrombosis and haemostasis : JTH.

Deletion of Fas protects islet beta cells from cytotoxic effects of human islet amyloid polypeptide

G. Warnock, M. Woo, T. Kieffer, 2012, Diabetologia.

The glucagon-like peptide-1 receptor agonist exenatide restores impaired pro-islet amyloid polypeptide processing in cultured human islets: implications in type 2 diabetes and islet transplantation

G. Warnock, T. Kieffer, D. M. Thompson, 2013, Diabetologia.

Sustained Depletion of FXIII-A by Inducing Acquired FXIII-B Deficiency.

P. Cullis, J. Kulkarni, C. Kastrup, 2020, Blood.

Specific loss of adipocyte CD248 improves metabolic health via reduced white adipose tissue hypoxia, fibrosis and inflammation

James D. Johnson, S. Catrina, A. Carpentier, 2019, EBioMedicine.

Dual role of interleukin‐1β in islet amyloid formation and its β‐cell toxicity: Implications for type 2 diabetes and islet transplantation

G. Warnock, T. Kieffer, Z. Ao, 2017, Diabetes, obesity & metabolism.

The role of caspase-8 in amyloid-induced beta cell death in human and mouse islets

R. Hakem, G. Warnock, M. Woo, 2014, Diabetologia.

Amyloid formation reduces protein kinase B phosphorylation in primary islet β-cells which is improved by blocking IL-1β signaling

G. Warnock, A. Ghahary, Z. Ao, 2018, PloS one.

CD248 promotes insulin resistance by binding to the insulin receptor and dampening its insulin-induced autophosphorylation

B. Craddock, J. D. Botezelli, N. Safikhan, 2023, EBioMedicine.