Akiko Yamagishi

发表

ERK is an anti-inflammatory signal that suppresses expression of NF-kappaB-dependent inflammatory genes by inhibiting IKK activity in endothelial cells.

N. Mochizuki, J. H. Kim, J. Kwon, 2006, Cellular signalling.

Cyclic AMP Potentiates Vascular Endothelial Cadherin-Mediated Cell-Cell Contact To Enhance Endothelial Barrier Function through an Epac-Rap1 Signaling Pathway

N. Mochizuki, N. Takakura, S. Fukuhara, 2005, Molecular and Cellular Biology.

Vascular endothelial cadherin-mediated cell-cell adhesion regulated by a small GTPase, Rap1.

N. Mochizuki, Keisuke Sako, Akiko Yamagishi, 2006, Journal of biochemistry and molecular biology.

Adaptor protein Crk is required for ephrin-B1-induced membrane ruffling and focal complex assembly of human aortic endothelial cells.

M. Matsuda, N. Mochizuki, H. Ogita, 2002, Molecular biology of the cell.

Identification of Fer tyrosine kinase localized on microtubules as a platelet endothelial cell adhesion molecule-1 phosphorylating kinase in vascular endothelial cells.

N. Mochizuki, M. Okada, Y. Kamioka, 2003, Molecular biology of the cell.

MAGI-1 is required for Rap1 activation upon cell-cell contact and for enhancement of vascular endothelial cadherin-mediated cell adhesion.

N. Mochizuki, S. Fukuhara, Y. Kamioka, 2005, Molecular biology of the cell.

Aryl hydrocarbon receptor is essential for the pathogenesis of pulmonary arterial hypertension

Y. Sakata, H. Ishibashi-Ueda, T. Kishimoto, 2021, Proceedings of the National Academy of Sciences.

Local Activation of Rap1 Contributes to Directional Vascular Endothelial Cell Migration Accompanied by Extension of Microtubules on Which RAPL, a Rap1-associating Molecule, Localizes*

N. Mochizuki, S. Fukuhara, Y. Kamioka, 2005, Journal of Biological Chemistry.

A Novel Actin Bundling/Filopodium-forming Domain Conserved in Insulin Receptor Tyrosine Kinase Substrate p53 and Missing in Metastasis Protein*

Takashi Ohki, N. Mochizuki, Naoki Mochizuki, 2004, Journal of Biological Chemistry.

Revised M 3 : 09408 A novel actin-bundling / filopodium-forming domain conserved in insulin receptor tyrosine kinase substrate p 53 and missing in metastasis protein

N. Mochizuki, M. Masuda, T. Ohki, 2003 .

MAGI-1 is required for Rap1 activation upon cell-cell contact and for enhancement of vascular endothelial cadherin-mediated cell adhesion.

S. Fukuhara, M. Masuda, Keisuke Sako, 2005, Molecular biology of the cell.