M. R. Bergman

Cardiac ischemia-reperfusion injury induces matrix metalloproteinase-2 expression through the AP-1 components FosB and JunB.

J. Karliner, D. Lovett, M. Alfonso-Jaume, 2006, American journal of physiology. Heart and circulatory physiology.

Pharmacological modulation of myocardial tumor necrosis factor (alpha) secretion by phosphodiesterase inhibitors in heart failure prone rats

M. R. Bergman, 1996 .

Myocardial tumor necrosis factor-α secretion in hypertensive and heart failure-prone rats.

S. Mccune, M. R. Bergman, B. Holycross, 1999, American journal of physiology. Heart and circulatory physiology.

Cardiac matrix metalloproteinase-2 expression independently induces marked ventricular remodeling and systolic dysfunction.

J. Karliner, D. Lovett, B. Zhu, 2007, American journal of physiology. Heart and circulatory physiology.

Pharmacological modulation of myocardial tumor necrosis factor alpha production by phosphodiesterase inhibitors.

M. R. Bergman, B. Holycross, 1996, The Journal of pharmacology and experimental therapeutics.

Myocardial tumor necrosis factor-alpha secretion in hypertensive and heart failure-prone rats.

S. Mccune, M. R. Bergman, B. Holycross, 1999, The American journal of physiology.

Cardiac transgenic matrix metalloproteinase-2 expression directly induces impaired contractility.

A. Baker, P. Simpson, J. Karliner, 2006, Cardiovascular research.

A functional activating protein 1 (AP-1) site regulates matrix metalloproteinase 2 (MMP-2) transcription by cardiac cells through interactions with JunB-Fra1 and JunB-FosB heterodimers.

Norman Honbo, Joel S Karliner, J. Karliner, 2003, The Biochemical journal.

Endothelin-1 stimulates cardiac fibroblast proliferation through activation of protein kinase C.

J. Karliner, L. Piacentini, N. Honbo, 2000, Journal of molecular and cellular cardiology.