M. E. Kyle

发表

The killing of cultured hepatocytes by N-acetyl-p-benzoquinone imine (NAPQI) as a model of the cytotoxicity of acetaminophen.

J. Farber, A. Harman, M. E. Kyle, 1991, Biochemical pharmacology.

Superoxide dismutase and catalase protect cultured hepatocytes from the cytotoxicity of acetaminophen.

J. Farber, D. Nakae, M. E. Kyle, 1987, Biochemical and biophysical research communications.

L-carnitine delays the killing of cultured hepatocytes by 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine.

T. Ferraro, M. E. Kyle, J. Snyder, 1990, Archives of biochemistry and biophysics.

Metabolism of acetaminophen by cultured rat hepatocytes. Depletion of protein thiol groups without any loss of viability.

J. Farber, I. Sakaida, M. E. Kyle, 1990, Biochemical pharmacology.

Protein thiol depletion and the killing of cultured hepatocytes by hydrogen peroxide.

J. Farber, D. Nakae, M. E. Kyle, 1989, Biochemical pharmacology.

tert-butyl hydroperoxide kills cultured hepatocytes by peroxidizing membrane lipids.

J. Farber, M. E. Kyle, N. Masaki, 1989, Archives of biochemistry and biophysics.

Endocytosis of superoxide dismutase is required in order for the enzyme to protect hepatocytes from the cytotoxicity of hydrogen peroxide.

J. Farber, D. Nakae, I. Sakaida, 1988, The Journal of biological chemistry.

Mechanisms of cell injury by activated oxygen species.

J. Farber, M. E. Kyle, J. Coleman, 1990, Laboratory investigation; a journal of technical methods and pathology.

Mitochondrial damage as a mechanism of cell injury in the killing of cultured hepatocytes by tert-butyl hydroperoxide.

J. Farber, M. E. Kyle, A. Serroni, 1989, Archives of biochemistry and biophysics.

Metabolism of salicylate by isolated kidney and liver mitochondria.

M. E. Kyle, J. Kocsis, James J. Kocsis, 1986, Chemico-biological interactions.

Piperonyl butoxide reduces salicylate-induced nephrotoxicity and covalent binding in male rats.

M. E. Kyle, J. Kocsis, 1986, Advances in experimental medicine and biology.

Toxic consequence of the abrupt depletion of glutathione in cultured rat hepatocytes.

J. Farber, D. Gilfor, M. E. Kyle, 1988, Archives of biochemistry and biophysics.

The effect of mixed function oxidase induction and inhibition on salicylate-induced nephrotoxicity in male rats.

M. E. Kyle, J. Kocsis, 1986, Toxicology and applied pharmacology.

The effect of age on salicylate-induced nephrotoxicity in male rats.

M. E. Kyle, J. Kocsis, 1985, Toxicology and applied pharmacology.

Oxidative cell injury in the killing of cultured hepatocytes by allyl alcohol.

J. Farber, D. Nakae, D. Gilfor, 1988, Archives of biochemistry and biophysics.

Autophagic degradation of protein generates a pool of ferric iron required for the killing of cultured hepatocytes by an oxidative stress.

J. Farber, I. Sakaida, M. E. Kyle, 1990, Molecular pharmacology.

The inhibition of lipid peroxidation by disulfiram prevents the killing of cultured hepatocytes by allyl alcohol, tert-butyl hydroperoxide, hydrogen peroxide and diethyl maleate.

J. Farber, M. E. Kyle, A. Serroni, 1989, Chemico-biological interactions.