A. Iida

发表

Carbamazepine-Induced Liver Injury Requires CYP3A-Mediated Metabolism and Glutathione Depletion in Rats

K. Tsuneyama, E. Sasaki, T. Fukami, 2015, Drug Metabolism and Disposition.

K. Tsuneyama, E. Sasaki, T. Fukami, 2015 .

Viability of liver slices exhibiting absorption, metabolism, and elimination of substrates in culture medium.

Y. Momose, A. Iida, N. Higashi, 2000, Experimental and molecular pathology.

Pathogenetic analyses of carbamazepine-induced liver injury in F344 rats focused on immune- and inflammation-related factors.

K. Tsuneyama, E. Sasaki, T. Fukami, 2016, Experimental and toxicologic pathology : official journal of the Gesellschaft fur Toxikologische Pathologie.

A novel mouse model for phenytoin-induced liver injury: involvement of immune-related factors and P450-mediated metabolism.

K. Tsuneyama, E. Sasaki, T. Fukami, 2013, Toxicological sciences : an official journal of the Society of Toxicology.

Human arylacetamide deacetylase hydrolyzes ketoconazole to trigger hepatocellular toxicity.

T. Fukami, M. Nakajima, A. Iida, 2016, Biochemical pharmacology.

Comparison of substrate specificity among human arylacetamide deacetylase and carboxylesterases.

T. Fukami, M. Nakajima, A. Iida, 2015, European journal of pharmaceutical sciences : official journal of the European Federation for Pharmaceutical Sciences.

Human arylacetamide deacetylase hydrolyzes ketoconazole to trigger hepatocellular toxicity.

T. Fukami, M. Nakajima, A. Iida, 2016, Biochemical pharmacology.