M. Steinberg

发表

Retrovirus-mediated transduction of primary ZAP-70-deficient human T cells results in the selective growth advantage of gene-corrected cells: implications for gene therapy

K. Schwarz, N. Taylor, W. Friedrich, 2000, Gene Therapy.

T-cell receptor-induced phosphorylation of the zeta chain is efficiently promoted by ZAP-70 but not Syk.

N. Taylor, N. Noraz, V. Di Bartolo, 2004, Blood.

T Cell Intrinsic Heterodimeric Complexes between HVEM and BTLA Determine Receptivity to the Surrounding Microenvironment1

Matthew G. Macauley, S. Fukuyama, C. Ware, 2009, The Journal of Immunology.

Reconstitution of lymphoid development and function in ZAP-70-deficient mice following gene transfer into bone marrow cells.

N. Taylor, P. Tiberghien, C. Ferrand, 2002, Blood.

BTLA Interaction with HVEM Expressed on CD8+ T Cells Promotes Survival and Memory Generation in Response to a Bacterial Infection

C. Ware, M. Kronenberg, H. Cheroutre, 2013, PloS one.

Alternative Antigen Receptor (TCR) Signaling in T Cells Derived from ZAP-70-deficient Patients Expressing High Levels of Syk*

K. Schwarz, N. Taylor, H. Yssel, 2000, The Journal of Biological Chemistry.

Unconventional ligand activation of herpesvirus entry mediator signals cell survival

Matthew G. Macauley, S. Fukuyama, C. Ware, 2009, Proceedings of the National Academy of Sciences.

Regulation of inflammation, autoimmunity, and infection immunity by HVEM‐BTLA signaling

M. Kronenberg, J. Shui, M. Steinberg, 2011, Journal of leukocyte biology.

Regulating the mucosal immune system: the contrasting roles of LIGHT, HVEM, and their various partners

C. Ware, M. Kronenberg, J. Shui, 2009, Seminars in Immunopathology.

Ex vivo isolation protocols differentially affect the phenotype of human CD4+ T cells.

N. Taylor, H. Yssel, V. Dardalhon, 2002, Journal of immunological methods.

T-cell receptor–induced phosphorylation of the (cid:1) chain is efﬁciently promoted by ZAP-70 but not Syk

N. Noraz, V. Bartolo, M. Steinberg, 2004 .

mice following gene transfer into bone marrow cells deficient - Reconstitution of lymphoid development and function in ZAP-70

N. Taylor, C. Ferrand, F. Candotti, 2013 .

In vivo correction of ZAP-70 immunodeficiency by intrathymic gene transfer.

N. Taylor, D. Klatzmann, V. Thomas-Vaslin, 2005, The Journal of clinical investigation.

Ozanimod (RPC1063), a selective S1PR1 and S1PR5 modulator, reduces chronic inflammation and alleviates kidney pathology in murine systemic lupus erythematosus

Matthew E. Stokes, G. Opiteck, R. Peach, 2018, PloS one.

The tumor necrosis factor family member TNFSF14 (LIGHT) is required for resolution of intestinal inflammation in mice.

Mitchell Kronenberg, P. Krause, Gisen Kim, 2014, Gastroenterology.

The signaling networks of the herpesvirus entry mediator (TNFRSF14) in immune regulation

C. Ware, M. Steinberg, T. Cheung, 2011, Immunological reviews.

A crucial role for HVEM and BTLA in preventing intestinal inflammation

Kenneth M. Murphy, Klaus Pfeffer, Mitchell Kronenberg, 2008, The Journal of experimental medicine.

IL-7 differentially regulates cell cycle progression and HIV-1-based vector infection in neonatal and adult CD4+ T cells

N. Taylor, P. Tiberghien, C. Ferrand, 2001, Proceedings of the National Academy of Sciences of the United States of America.

transendothelial migration Signaling through ZAP-70 is required for CXCL12-mediated T-cell

C. Charvet, M. Deckert, M. Ticchioni, 2013 .

Signaling through ZAP-70 is required for CXCL12-mediated T-cell transendothelial migration.

C. Charvet, M. Deckert, M. Ticchioni, 2002, Blood.

The tumor necrosis factor family member TNFSF14 (LIGHT) is required for resolution of intestinal inflammation in mice.

P. Krause, Gisen Kim, M. Kronenberg, 2014, Gastroenterology.