J. E. Sim

发表

Alterations in heat-induced radiosensitization accompanied by nuclear structure alterations in Chinese hamster cells

Amanda Harvey, D. Spitz, J. R. Roti Roti, 2006, International journal of hyperthermia : the official journal of European Society for Hyperthermic Oncology, North American Hyperthermia Group.

Increased lipid peroxidation and impaired antioxidant enzyme function is associated with pathological liver injury in experimental alcoholic liver disease in rats fed diets high in corn oil and fish oil

A. Nanji, L. Oberley, D. Spitz, 1998, Hepatology.

Genomic instability and catalase gene amplification induced by chronic exposure to oxidative stress.

C. Hunt, D. Spitz, T. Featherstone, 1998, Cancer research.

Evaluation of Parameters of Oxidative Stress after In Vitro Exposure to FMCW- and CDMA-Modulated Radiofrequency Radiation Fields

E. Moros, J. Baty, R. Higashikubo, 2004, Radiation research.

Genomic Instability and Catalase Gene Amplification Induced by Chronic Exposure to Oxidative Stress1

Douglas, R. A. Gomez, R. Spitz, 2006 .

Dominant-negative Jun N-terminal protein kinase (JNK-1) inhibits metabolic oxidative stress during glucose deprivation in a human breast carcinoma cell line.

H. Forman, Y. Lee, L. Ridnour, 2000, Free radical biology & medicine.

Differences in basal and hyperoxia-associated HO expression in oxidant-resistant hamster fibroblasts.

D. Spitz, P. Dennery, H. E. Wong, 1996, The American journal of physiology.

A spectrophotometric method for the direct detection and quantitation of nitric oxide, nitrite, and nitrate in cell culture media.

D. Wink, L. Ridnour, D. Spitz, 2000, Analytical biochemistry.

Glucose Deprivation-induced Cytotoxicity and Alterations in Mitogen-activated Protein Kinase Activation Are Mediated by Oxidative Stress in Multidrug-resistant Human Breast Carcinoma Cells*

D. Spitz, B. Davis, P. Corry, 1998, The Journal of Biological Chemistry.

Mitochondrial O2*- and H2O2 mediate glucose deprivation-induced stress in human cancer cells.

S. Flanagan, L. Oberley, M. Mackey, 2005, The Journal of biological chemistry.

Metabolic oxidative stress activates signal transduction and gene expression during glucose deprivation in human tumor cells.

Y. Lee, L. Ridnour, D. Spitz, 1999, Free radical biology & medicine.

Cell cycle-coupled variation in topoisomerase IIalpha mRNA is regulated by the 3'-untranslated region. Possible role of redox-sensitive protein binding in mRNA accumulation.

C. Hunt, L. Albee, R. Higashikubo, 2000, The Journal of biological chemistry.

Glial cell type-specific responses to menadione-induced oxidative stress.

D. Spitz, S. Ledoux, G. Wilson, 2000, Free radical biology & medicine.

Glucose Deprivation‐Induced Oxidative Stress in Human Tumor Cells: A Fundamental Defect in Metabolism?

L. Ridnour, D. Spitz, J. E. Sim, 2000, Annals of the New York Academy of Sciences.

Bcl-2 and Bcl-xL in peroxide-resistant A549 and U87MG cells.

D. Spitz, H. K. Bojes, J. E. Sim, 1998, Toxicological sciences : an official journal of the Society of Toxicology.

Nitric oxide-induced resistance to hydrogen peroxide stress is a glutamate cysteine ligase activity-dependent process.

H. Forman, C. Hunt, L. Ridnour, 2005, Free radical biology & medicine.

Mitochondrial \batchmode \documentclass[fleqn,10pt,legalpaper]{article} \usepackage{amssymb} \usepackage{amsfonts} \usepackage{amsmath} \pagestyle{empty} \begin{document} \(\mathrm{O}_{2}^{{\bar{{\cdot}}}}\) \end{document} and H2O2 Mediate Glucose Deprivation-induced Stress in Human Cancer Cells*

S. Flanagan, L. Oberley, M. Mackey, 2005, Journal of Biological Chemistry.