D. Schmitt

发表

Apolipoprotein A-I is a selective target for myeloperoxidase-catalyzed oxidation and functional impairment in subjects with cardiovascular disease.

Michael Kinter, Marlene Goormastic, Lemin Zheng, 2004, The Journal of clinical investigation.

Formation of nitric oxide-derived oxidants by myeloperoxidase in monocytes: pathways for monocyte-mediated protein nitration and lipid peroxidation In vivo.

S. Hazen, E. Podrez, C. Mukhopadhyay, 1999, Circulation research.

Myeloperoxidase-generated reactive nitrogen species convert LDL into an atherogenic form in vitro.

S. Hazen, E. Podrez, H F Hoff, 1999, The Journal of clinical investigation.

Erratum: Macrophage scavenger receptor CD36 is the major receptor for LDL modified by monocyte-generated reactive nitrogen species (Journal of Clinical Investigation (2000) 105 (1095-1108))

S. Hazen, E. Podrez, N. Sheibani, 2000 .

Paradoxical Association of Enhanced Cholesterol Efflux With Increased Incident Cardiovascular Risks

S. Hazen, E. Fisher, Jonathan D. Smith, 2013, Arteriosclerosis, thrombosis, and vascular biology.

Localization of Nitration and Chlorination Sites on Apolipoprotein A-I Catalyzed by Myeloperoxidase in Human Atheroma and Associated Oxidative Impairment in ABCA1-dependent Cholesterol Efflux from Macrophages*

Michael Kinter, Lemin Zheng, Stanley L Hazen, 2005, Journal of Biological Chemistry.

MyD88-dependent interplay between myeloid and endothelial cells in the initiation and progression of obesity-associated inflammatory diseases

S. Hazen, R. Ransohoff, Jonathan D. Smith, 2014, The Journal of experimental medicine.

Cholesteryl Hydroperoxyoctadecadienoate from Oxidized Low Density Lipoprotein Inactivates Platelet-derived Growth Factor*

M. Cathcart, P. Dicorleto, D. Schmitt, 1998, The Journal of Biological Chemistry.

Myeloperoxidase Functions as a Major Enzymatic Catalyst for Initiation of Lipid Peroxidation at Sites of Inflammation*

Marie-Luise Brennan, Stanley L Hazen, S. Hazen, 2002, The Journal of Biological Chemistry.

Relationship of paraoxonase 1 (PON1) gene polymorphisms and functional activity with systemic oxidative stress and cardiovascular risk.

E. Topol, M. Shao, S. Hazen, 2008, JAMA.

Macrophage scavenger receptor CD36 is the major receptor for LDL modified by monocyte-generated reactive nitrogen species.

S. Hazen, E. Podrez, N. Sheibani, 2000, The Journal of clinical investigation.

An abundant dysfunctional apolipoprotein A1 in human atheroma

Valentin Gogonea, Paul L. Fox, Joseph A. DiDonato, 2014, Nature Medicine.

MyD88-dependent interplay between myeloid and endothelial cells in the initiation and progression of obesity-associated inflammatory diseases

S. Hazen, R. Ransohoff, Jonathan D. Smith, 2014, The Journal of experimental medicine.

MyD88-dependent interplay between myeloid and endothelial cells in the initiation and progression of obesity-associated inflammatory diseases

R. Ransohoff, Jonathan D. Smith, A. DeFranco, 2014, The Journal of Experimental Medicine.

Leukocytes utilize myeloperoxidase-generated nitrating intermediates as physiological catalysts for the generation of biologically active oxidized lipids and sterols in serum.

S. Hazen, G. Chisolm, R. G. Salomon, 1999, Biochemistry.

Abstract 53: An Abundant Dysfunctional Apolipoprotein A1 in Human Atheroma

S. Hazen, E. Fisher, Jonathan D. Smith, 2014, Arteriosclerosis, Thrombosis, and Vascular Biology.